Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Elisabeth K, Malle"'
Autor:
Robert Brink, David Zahra, Jeanette E. Villanueva, Benjamin T. Porebski, Garry P. Nolan, Murray P. Cox, Carla M. Roots, Claudia Loetsch, Cecile King, Paul Z. Benitez-Aguirre, Jia Tang, Belinda Whittle, Juliana Teo, Joanna Warren, Wendy Sandoval, Marcel E. Dinger, Elisabeth K. Malle, Christopher C. Goodnow, Geeta Chaudhri, Velimir Gayevskiy, Ingrid E. Wertz, Jin Yan Yap, John B. Ziegler, Yogesh Jeelall, Keisuke Horikawa, Colin J. Jackson, Stacey N. Walters, Daniele Cultrone, Daniel Christ, Frank Schmitz, Nathan W. Zammit, Shane T. Grey, Melanie Wong, David B. Langley, Craig N. Jenne, Owen M. Siggs, Tim Wiltshire, Anselm Enders, Lewis L. Lanier, Mark J. Cowley, Matthew H. Spitzer, Wilson Phung, Stuart G. Tangye, Peter D. Mabbitt, Derek W. Abbott, Susan R. Watson, Benjamin E. Clifton, Stephen R. Daley, Alan Aderem, Paul Gray, Ashley M. Buckle, Gunasegaran Karupiah, Michiko Yamada, Edward M. Bertram, Amanda J. Russell, Maria E. Craig
Publikováno v:
Nature Immunology. 20:1299-1310
Resisting and tolerating microbes are alternative strategies to survive infection, but little is known about the evolutionary mechanisms controlling this balance. Here genomic analyses of anatomically modern humans, extinct Denisovan hominins and mic
Autor:
Nathan W. Zammit, Bernice M. Tan, Stacey N. Walters, David Liuwantara, Jeanette E. Villanueva, Elisabeth K. Malle, Shane T. Grey
Publikováno v:
Cell Transplantation, Vol 22 (2013)
Islet grafts can contribute to their own destruction via the elaboration of proinflammatory genes, many of which are transcriptionally regulated by nuclear factor κ-light-chain-enhancer of activated B-cells (NF-κB). Thus, NF-κB constitutes an enti
Externí odkaz:
https://doaj.org/article/8c19b9f1c7b644a6baaafca645a8c4d4
Autor:
Elisabeth K. Malle, Katherine A. Watson, Stacey N. Walters, Nicole L. La Gruta, Nathan W. Zammit, M. Saito, Robert Brink, Stephen I. Alexander, Shane T. Grey, Jeanette E. Villanueva
Publikováno v:
Diabetologia. 60:679-689
Administration of anti-CD40 ligand (CD40L) antibodies has been reported to allow long-term islet allograft survival in non-human primates without the need for exogenous immunosuppression. However, the use of anti-CD40L antibodies was associated with
Autor:
Susan R. Watson, Stephen R. Daley, Edward M. Bertram, David Zahra, Joanna Warren, Stacey N. Walters, Ashley M. Buckle, Claudia Loetsch, Cecile King, Yogesh Jeelall, Keisuke Horikawa, Colin J. Jackson, Shane T. Grey, Anselm Enders, Paul Gray, Benjamin T. Porebski, Craig N. Jenne, Marcel E. Dinger, Jeanette E. Villanueva, Amanda J. Russell, Owen M. Siggs, Michiko Yamada, Derek W. Abbott, David B. Langley, Maria E. Craig, Matthew H. Spitzer, Melanie Wong, Ingrid E. Wertz, Mark J. Cowley, John B. Ziegler, Tim Wiltshire, Paul Z. Benitez-Aguirre, Christopher C. Goodnow, Daniel Christ, Garry P. Nolan, Murray P. Cox, Benjamin E. Clifton, Daniele Cultrone, Gunasegaran Karupiah, Alan Aderem, Robert Brink, Nathan W. Zammit, Carla M. Roots, Lewis L. Lanier, Juliana Teo, Elisabeth K. Malle, Geeta Chaudhri, Peter D. Mabbitt, Belinda Whittle, Frank Schmitz, Velimir Gayevskiy, Wilson Phung
Publikováno v:
Nature Immunology
bioRxiv
bioRxiv
Resisting or tolerating microbes are alternative strategies to survive infection, but little is known about the evolutionary mechanisms controlling this balance. Here, genomic analyses of anatomically modern humans, extinct Denisovan hominins, and mi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2f197ac2554c943341a13dcc627644f
https://doi.org/10.1101/589507
https://doi.org/10.1101/589507
Autor:
Nathan W, Zammit, Owen M, Siggs, Paul E, Gray, Keisuke, Horikawa, David B, Langley, Stacey N, Walters, Stephen R, Daley, Claudia, Loetsch, Joanna, Warren, Jin Yan, Yap, Daniele, Cultrone, Amanda, Russell, Elisabeth K, Malle, Jeanette E, Villanueva, Mark J, Cowley, Velimir, Gayevskiy, Marcel E, Dinger, Robert, Brink, David, Zahra, Geeta, Chaudhri, Gunasegaran, Karupiah, Belinda, Whittle, Carla, Roots, Edward, Bertram, Michiko, Yamada, Yogesh, Jeelall, Anselm, Enders, Benjamin E, Clifton, Peter D, Mabbitt, Colin J, Jackson, Susan R, Watson, Craig N, Jenne, Lewis L, Lanier, Tim, Wiltshire, Matthew H, Spitzer, Garry P, Nolan, Frank, Schmitz, Alan, Aderem, Benjamin T, Porebski, Ashley M, Buckle, Derek W, Abbott, John B, Ziegler, Maria E, Craig, Paul, Benitez-Aguirre, Juliana, Teo, Stuart G, Tangye, Cecile, King, Melanie, Wong, Murray P, Cox, Wilson, Phung, Jia, Tang, Wendy, Sandoval, Ingrid E, Wertz, Daniel, Christ, Christopher C, Goodnow, Shane T, Grey
Publikováno v:
Nature immunology. 20(10)
Resisting and tolerating microbes are alternative strategies to survive infection, but little is known about the evolutionary mechanisms controlling this balance. Here genomic analyses of anatomically modern humans, extinct Denisovan hominins and mic
Autor:
Stacey N. Walters, Robert Brink, Elisabeth K. Malle, Bernice M. Tan, Shelli R. McAlpine, Yen Chin Koay, Thomas Loudovaris, Daniel Hesselson, James Cantley, Nathan W. Zammit, Jeanette E. Villanueva, Jianmin Wu, Shane T. Grey
Publikováno v:
The Journal of Experimental Medicine
Malle et al. identify a role for nuclear factor inducing κB (NIK) in pancreatic β cell failure. NIK activation disrupts glucose homeostasis in zebrafish in vivo and impairs glucose-stimulated insulin secretion in mouse and human islets in vitro. NI
Autor:
Jeanette E. Villanueva, Elisabeth K. Malle, Pablo A. Silveira, Nathan W. Zammit, Stacey N. Walters, Robert Brink, Sandra Gardam, Shane T. Grey
Publikováno v:
European Journal of Immunology. 45:1820-1831
In this study, a critical and novel role for TNF receptor (TNFR) associated factor 2 (TRAF2) is elucidated for peripheral CD8(+) T-cell and NKT-cell homeostasis. Mice deficient in TRAF2 only in their T cells (TRAF2TKO) show ∼40% reduction in effect
Autor:
Elisabeth K. Malle, Shane T. Grey, Stacey N. Walters, David Liuwantara, Nathan W. Zammit, Jeanette E. Villanueva, Bernice M. Tan
Publikováno v:
Cell Transplantation, Vol 22 (2013)
Islet grafts can contribute to their own destruction via the elaboration of proinflammatory genes, many of which are transcriptionally regulated by nuclear factor κ-light-chain-enhancer of activated B-cells (NF-κB). Thus, NF-κB constitutes an enti