Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Elif Demirdogen Sevinc"'
Autor:
Tugba H. Solakoglu, Sabire Peker, Saime Güzel, Elif Demirdogen Sevinc, Basak D. Balaban, Yunus Gurpinar, Abdullah Yalcin, Jason Chesney, Serap Celikler, Selahattin C. Ozcan
Transforming growth factor [31 (TGF beta 1) is a well -established inducer of the epithelial-mesenchymal transition (EMT) that is essential for the acquisition of malignant properties, such as invasion, in tumor cells. Although recent studies suggest
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e6ae6d11d3534992ede96afd53fa4a1
http://hdl.handle.net/11452/30221
http://hdl.handle.net/11452/30221
Autor:
Ismet Tasdelen, Gulsah Cecener, Sahsine Tolunay, Unal Egeli, Berrin Tunca, Sehsuvar Gokgoz, Secil Ak, Elif Demirdogen Sevinc
Publikováno v:
Gene. 590(2)
The dysregulation of miRNA expression has frequently been observed in breast cancer. Therefore, we investigated the expression profile of miRNAs that may be associated with expression of the FHIT gene in breast cancer and assessed their clinicopathol
Autor:
Turkkan Evrensel, Gulsah Cecener, Pinar Sarkut, Yilmaz Ozen, Secil Ak, Elif Demirdogen Sevinc, Gulcin Tezcan, Berrin Tunca, Omer Yerci, Nesrin Ugras, Unal Egeli, Ekrem Kaya, Halit Ziya Dündar
Objectives: The success of gemcitabine plus radiotherapy is dependent on the mutation status of pancreatic ductal adenocarcinoma (PDAC) tumors in the EGFR and KRAS genes; however, radiotherapy resistance may also be modulated epigenetically by microR
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67fe6f61506b9a48651125e18944a702
http://hdl.handle.net/11452/29517
http://hdl.handle.net/11452/29517
Autor:
Turkkan Evrensel, Pinar Sarkut, Gulsah Cecener, Gulcin Tezcan, Nesrin Ugras, Secil Ak, Unal Egeli, Omer Yerci, Ozkan Balcin, Ekrem Kaya, Halit Ziya Dündar, Berrin Tunca, Yilmaz Ozen, Elif Demirdogen Sevinc
Publikováno v:
Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]. 16(4)
Objective Several studies have demonstrated the importance of mutations in codons 12, 13 and 61 and variations in the 3′ untranslated region (3′UTR) of the KRAS gene, frequently observed genetic events in the progression of pancreatobiliary tumor