Zobrazeno 1 - 9 of 9 pro vyhledávání: '"Elias, Sevdalis"'
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Clinical and Genetic Characterization of Families With Arrhythmogenic Right Ventricular Dysplasia/Cardiomyopathy Provides Novel Insights Into Patterns of Disease Expression
Autor: Elias Sevdalis, William J. McKenna, Angeliki Asimaki, Petros Syrris, Srijita Sen-Chowdhry, Deirdre Ward
Publikováno v: Circulation. 115:1710-1720
Background— According to clinical-pathological correlation studies, the natural history of arrhythmogenic right ventricular dysplasia/cardiomyopathy is purported to progress from localized to global right ventricular dysfunction, followed by left v
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ba9a8b4f58380f216e390486b43b834
https://doi.org/10.1161/circulationaha.106.660241
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3
Natural history and familial characteristics of isolated left ventricular non-compaction
Autor: Ross T, Murphy, Rajesh, Thaman, Juan Gimeno, Blanes, Deirdre, Ward, Elias, Sevdalis, Efi, Papra, Anatoli, Kiotsekoglou, Anatoli, Kiotsekolglou, Maria T, Tome, Denis, Pellerin, William J, McKenna, Perry M, Elliott
Publikováno v: European Heart Journal. 26:187-192
Aims Non-compaction of the left ventricle (LVNC) is a disorder of endomyocardial morphogenesis that results in multiple trabeculations in the left ventricular myocardium. The current literature suggests that LVNC in adults is rare and associated with
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::abf4d27da8fed255ba4ba214e038833a
https://doi.org/10.1093/eurheartj/ehi025
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4
Genotype-phenotype assessment in autosomal recessive arrhythmogenic right ventricular cardiomyopathy (Naxos disease) caused by a deletion in plakoglobin
Autor: Kostas Gatzoulis, Godfrina McKoy, Kostas Stratos, Aris Anastasakis, K. Tentolouris, William J. McKenna, Chara Spiliopoulou, Demos Panagiotakos, Elias Sevdalis, P. Toutouzas, Nikos Protonotarios, Adalena Tsatsopoulou
Publikováno v: Journal of the American College of Cardiology. 38:1477-1484
OBJECTIVES The purpose of this study was to examine the genotype-phenotype relation with respect to penetrance, age and severity of expression, disease progression and prognosis in a recessively inherited arrhythmogenic right ventricular cardiomyopat
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dbe619610aeb57467a83d0fa5c5b2b3e
https://doi.org/10.1016/s0735-1097(01)01568-6
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5
Sudden cardiac death: investigation of the classical risk factors in a community-based hypertrophic cardiomyopathy cohort
Autor: Aris, Anastasakis, Artemisia, Theopistou, Angelos, Rigopoulos, Christina, Kotsiopoulou, Stavros, Georgopoulos, Konstantinos, Fragakis, Elias, Sevdalis, Christodoulos, Stefanadis
Publikováno v: Hellenic journal of cardiology : HJC = Hellenike kardiologike epitheorese. 54(4)
The identification of high-risk patients in hypertrophic cardiomyopathy (HCM) is still a challenge. The classical clinical risk factors for sudden death have been reported by studies coming from referral HCM cohorts. So far, other studies of communit
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::5a5f15a1e7080e0ba0fff8a0530b44d0
https://pubmed.ncbi.nlm.nih.gov/23912920
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7
Prevalence of exercise-induced left ventricular outflow tract obstruction in symptomatic patients with non-obstructive hypertrophic cardiomyopathy
Autor: Elias Sevdalis, Bryan Mist, Rajesh Thaman, JS Shah, Deirdre Ward, Maria Teresa Tome Esteban, Andre Keren, Perry M. Elliott, Rajan Sharma, Camelia Demetrescu, Sanjay K Kohli, William J. McKenna, Antonis Pantazis, Denis Pellerin, Steve P. Page
Publikováno v: Heart (British Cardiac Society). 94(10)
Resting left ventricular outflow tract obstruction (LVOTO) occurs in 25% of patients with hypertrophic cardiomyopathy (HCM) and is an important cause of symptoms and disease progression. The prevalence and clinical significance of exercise induced LV
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffcd05c9b7e78ed35fd88506632eec8b
https://pubmed.ncbi.nlm.nih.gov/18801784
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