Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Elfi Quente"'
Publikováno v:
Genetics and Molecular Biology, Vol 35, Iss 3, Pp 567-574 (2012)
An important area of genetic research is the identification of functional mechanisms in polymorphisms associated with diseases. A highly relevant functional mechanism is the influence of polymorphisms on gene expression levels (differential allelic e
Externí odkaz:
https://doaj.org/article/9641204a94b74493a3626109b888a531
Autor:
Jana Burkhardt, Mechthild Blume, Elisabeth Petit-Teixeira, Vitor Hugo Teixeira, Anke Steiner, Elfi Quente, Grit Wolfram, Markus Scholz, Céline Pierlot, Paola Migliorini, Stefano Bombardieri, Alejandro Balsa, René Westhovens, Pilar Barrera, Timothy R D J Radstake, Helena Alves, Thomas Bardin, Bernard Prum, Frank Emmrich, François Cornelis, Peter Ahnert, Holger Kirsten
Publikováno v:
PLoS ONE, Vol 9, Iss 8, p e103872 (2014)
In rheumatoid arthritis (RA), a key event is infiltration of inflammatory immune cells into the synovial lining, possibly aggravated by dysregulation of cellular adhesion molecules. Therefore, single nucleotide polymorphisms of 14 genes involved in c
Externí odkaz:
https://doaj.org/article/bdc89cf155be4f199c0a904ba3741878
Autor:
Jens Minnerup, Alexander Kranz, Wenke Fröhlich, Johanna Scheibe, Johannes Boltze, Viktoria Bothe, Daniel-Christoph Wagner, Franziska Lange, Wolf-Rüdiger Schäbitz, Elfi Quente, Claudia Pösel
Publikováno v:
Stroke. 45:2431-2437
Background and Purpose— We aimed to determine a possible synergistic effect of granulocyte colony-stimulating factor (G-CSF) and bone marrow–derived mononuclear cells (BM MNC) after stroke in spontaneously hypertensive rats. Methods— Male spont
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c7f518c7767576a08529dba397847e2
https://doi.org/10.1161/strokeaha.113.004460
https://doi.org/10.1161/strokeaha.113.004460
Autor:
Jana Burkhardt, Ivonne Czepezauer, Jens Brauer, Holger Kirsten, B. Mueller, Elfi Quente, Johannes Boltze, Peter Ahnert, Arndt Wilcke
Publikováno v:
Genes, Brain and Behavior. 13:350-356
Dyslexia is characterized by impaired reading and spelling. The disorder has a prevalence of about 5% in Germany, and a strong hereditary component. Several loci are thought to be involved in the development of dyslexia. Scerri et al. identified eigh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::68e45be885f37d82874c9e34c67e15b7
https://doi.org/10.1111/gbb.12118
https://doi.org/10.1111/gbb.12118
Autor:
Sven Cichon, Bertram Müller-Myhsok, Holger Kirsten, Michael Alexander, Jana Burkhardt, Johannes Boltze, Albert J. Becker, Peter Ahnert, Christiane Wolf, Carolin Ligges, Arndt Wilcke, Elfi Quente, Per Hoffmann
Publikováno v:
European Journal of Human Genetics. 20:224-229
Dyslexia is a developmental disorder characterised by extensive difficulties in the acquisition of reading or spelling. Genetic influence is estimated at 50-70%. However, the link between genetic variants and phenotypic deficits is largely unknown. O
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb6475c6773cb1af1b48809cc7d6e8cb
https://doi.org/10.1038/ejhg.2011.160
https://doi.org/10.1038/ejhg.2011.160
Autor:
Jana Burkhardt, Carolin Ligges, Heidrun Holland, Wolfgang Krupp, Johannes Boltze, Peter Ahnert, Elfi Quente, Holger Kirsten, Arndt Wilcke
Publikováno v:
Psychiatric Genetics. 22:307-308
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca5723ff9aff917488c1419a1257cc1a
https://doi.org/10.1097/ypg.0b013e328353aeae
https://doi.org/10.1097/ypg.0b013e328353aeae
Publikováno v:
New Biotechnology. 27:S69-S70
An important question in ongoing research of genetic traits in complex diseases is functional relevance of identified disease associating genetic factors. previous termDifferential allelic expressionnext term (DAE) has been estimated to affect 20-50%
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91a909af871bad3af6ceac354e486013
https://doi.org/10.1016/j.nbt.2010.01.198
https://doi.org/10.1016/j.nbt.2010.01.198
Autor:
Carolin Ligges Jana Burkhardt, Michael Alexander, Johannes Boltze, Peter Ahnert, Albert J. Becker, Sven Cichon, Bertram Müller-Myhsok, Elfi Quente, Arndt Wilcke, Christiane Wolf, Per Hoffmann, Holger Kirsten
Publikováno v:
European Journal of Human Genetics. 20:714-714
Correction to: European Journal of Human Genetics (2012) 20, 224–229; doi:10.1038/ejhg.2011.160; published online 7 September 2011 The authors would like to make the following amendments: The following sentence in the Materials and Methods section:
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::303034765ee6b177bdd682f1fe8ba434
https://doi.org/10.1038/ejhg.2012.31
https://doi.org/10.1038/ejhg.2012.31
Publikováno v:
ResearcherID
Genetics and Molecular Biology, Vol 35, Iss 3, Pp 567-574 (2012)
Genetics and Molecular Biology
Genetics and Molecular Biology, Volume: 35, Issue: 3, Pages: 567-574, Published: 02 AUG 2012
Genetics and Molecular Biology v.35 n.3 2012
Sociedade Brasileira de Genética (SBG)
instacron:SBG
Genetics and Molecular Biology, Vol 35, Iss 3, Pp 567-574 (2012)
Genetics and Molecular Biology
Genetics and Molecular Biology, Volume: 35, Issue: 3, Pages: 567-574, Published: 02 AUG 2012
Genetics and Molecular Biology v.35 n.3 2012
Sociedade Brasileira de Genética (SBG)
instacron:SBG
An important area of genetic research is the identification of functional mechanisms in polymorphisms associated with diseases. A highly relevant functional mechanism is the influence of polymorphisms on gene expression levels (differential allelic e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40f838596f5cc75bfcda17b44635f825
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000307726400004&KeyUID=WOS:000307726400004
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000307726400004&KeyUID=WOS:000307726400004
