Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Elena S. Klushevskaya"'
Autor:
Nickolai A. Tchurikov, Elena S. Klushevskaya, Daria M. Fedoseeva, Ildar R. Alembekov, Galina I. Kravatskaya, Vladimir R. Chechetkin, Yuri V. Kravatsky, Olga V. Kretova
Publikováno v:
Cells, Vol 9, Iss 12, p 2587 (2020)
Chromosomes are organized into 3D structures that are important for the regulation of gene expression and differentiation. Important role in formation of inter-chromosome contacts play rDNA clusters that make up nucleoli. In the course of differentia
Externí odkaz:
https://doaj.org/article/4bb5ee7e87724caf8bc55aab7bfddccf
Autor:
Nickolai A. Tchurikov, Daria M. Fedoseeva, Elena S. Klushevskaya, Ivan Y. Slovohotov, Vladimir R. Chechetkin, Yuri V. Kravatsky, Olga V. Kretova
Publikováno v:
Cells, Vol 8, Iss 11, p 1393 (2019)
Human rDNA clusters form numerous contacts with different chromosomal regions as evidenced by chromosome conformation capture data. Heterochromatization of rDNA genes leads to heterochromatization in different chromosomal regions coupled with the act
Externí odkaz:
https://doaj.org/article/2f9ac5da5ff24e1bacf891ed8f937844
Autor:
Kravatsky, Nickolai A. Tchurikov, Elena S. Klushevskaya, Ildar R. Alembekov, Antonina N. Kretova, Vladimir R. Chechetkin, Galina I. Kravatskaya, Yuri V.
Publikováno v:
International Journal of Molecular Sciences; Volume 24; Issue 12; Pages: 9842
The expression of clusters of rDNA genes influences pluripotency; however, the underlying mechanisms are not yet known. These clusters shape inter-chromosomal contacts with numerous genes controlling differentiation in human and Drosophila cells. Thi
Autor:
Nickolai A. Tchurikov, Ildar R. Alembekov, Elena S. Klushevskaya, Antonina N. Kretova, Ann M. Keremet, Anastasia E. Sidorova, Polina B. Meilakh, Vladimir R. Chechetkin, Galina I. Kravatskaya, Yuri V. Kravatsky
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 13; Pages: 7201
Double-strand DNA breakes (DSBs) are the most deleterious and widespread examples of DNA damage. They inevitably originate from endogenous mechanisms in the course of transcription, replication, and recombination, as well as from different exogenous