Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Eleftheria Barlaka"'
Publikováno v:
Cardiovascular Therapeutics. 34:37-48
Peroxisome proliferator-activated receptors, PPARα, PPARβ/δ, and PPARγ, are a group of nuclear receptors that function as transcriptional regulators of lipid metabolism, energy homeostasis, and inflammation. Given the role of metabolism imbalance
Autor:
Antigone Lazou, Eleftheria Barlaka
Publikováno v:
Encyclopedia of Signaling Molecules ISBN: 9781461464389
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::930d5e26811ee25bb3228706fd016c08
https://doi.org/10.1007/978-1-4614-6438-9_101829-1
https://doi.org/10.1007/978-1-4614-6438-9_101829-1
Publikováno v:
Cell Biochemistry and Function. 29:442-451
In response to pathophysiological stresses, cardiac myocytes undergo hypertrophic growth or apoptosis. Multiple signalling pathways have been implicated in these responses and among them, kinases such as mitogen-activated protein kinases (MAPKs) and
Autor:
Péter Ferdinandy, Antigone Lazou, Anikó Görbe, János Pálóczi, Eleftheria Barlaka, Renáta Gáspár
Publikováno v:
Pharmacological research.
Heart failure still remains one of the leading causes of morbidity and mortality worldwide. A major contributing factor is reactive oxygen/nitrogen species (RONS) overproduction which is associated with cardiac remodeling partly through cardiomyocyte
Autor:
František Kolář, P. Mandíková, Táňa Ravingerová, Nemčeková M, Antigone Lazou, Adriana Adameova, Eleftheria Galatou, A. Chytilová, Eleftheria Barlaka, V. Ledvényiová, S Carnicka
Publikováno v:
Scopus-Elsevier
UNLABELLED Genes encoding enzymes involved in fatty acids (FA) and glucose oxidation are transcriptionally regulated by peroxisome proliferator-activated receptors (PPAR), members of the nuclear receptor superfamily. Under conditions associated with
Autor:
Frank Lezoualc'h, Satoko Ishikawa, Pierre Sicard, Keitaro Nishizawa, Bijan Ghaleh, Tetsuji Miura, Wataru Ohwada, Kyriakos Mellidis, A Moraiti, Loubina Fazal, M Laudette, Masashi Mizuno, Atsushi Kuno, Yuki Tatekoshi, Eleftheria Barlaka, Florence Tortosa, Takayuki Miki, Antigone Lazou, Toshiyuki Yano, Jeanne Mialet-Perez, Masaya Tanno, Sandrine Pons, A Apostolopoulos
Publikováno v:
Cardiovascular Research. 111:S1-S1
5 Epac1 deletion prevents cardiomyocyte apoptosis during ischemia/reperfusion {#article-title-2} Introduction: Early coronary reperfusion has been established as the best therapeutic strategy to limit infarct size and improve prognosis. Therefore, el
Autor:
Táňa Ravingerová, S Carnicka, Tara Kelly, Eleftheria Galatou, Adriana Adameova, Nemčeková M, Antigone Lazou, Eleftheria Barlaka, V. Ledvényiová, Vinoth Kumar Megraj Khandelwal
Publikováno v:
Canadian journal of physiology and pharmacology. 90(8)
Peroxisome proliferator-activated receptors (PPAR) regulate the expression of genes involved in lipid metabolism, energy production, and inflammation. Their role in ischaemia–reperfusion (I/R) is less clear, although research indicates involvement
Publikováno v:
Cell biochemistry and function. 29(6)
In response to pathophysiological stresses, cardiac myocytes undergo hypertrophic growth or apoptosis. Multiple signalling pathways have been implicated in these responses and among them, kinases such as mitogen-activated protein kinases (MAPKs) and
Autor:
A. Chytilová, V. Ledvényiová, Nemčeková M, Antigone Lazou, S Carnicka, Eleftheria Barlaka, Eleftheria Galatou, Petra Mandikova, Frantisek Kolar, Táňa Ravingerová
Publikováno v:
Cardiovascular Research. 103:S78.3-S78
Ischemia alters the balance between fatty acids (FA) and glucose utilization as the main ATP-producing pathways in the heart that may dramatically affect myocardial salvage upon ischemia/reperfusion (I/R). Genes encoding enzymes involved in metabolic
Publikováno v:
Cardiovascular Research. 103:S14.4-S14
Background and purpose: Oxidative stress promotes cardiac myocyte death and it is a key factor in the development of various cardiovascular diseases. Several signaling pathways have been implicated in the response of cardiac myocytes to oxidative str