Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Elżbieta Salińska"'
Autor:
Jakub Godlewski, Ruben Ferrer-Luna, Arun K. Rooj, Marco Mineo, Franz Ricklefs, Yuji S. Takeda, M. Oskar Nowicki, Elżbieta Salińska, Ichiro Nakano, Hakho Lee, Ralph Weissleder, Rameen Beroukhim, E. Antonio Chiocca, Agnieszka Bronisz
Publikováno v:
Stem Cell Reports, Vol 8, Iss 6, Pp 1497-1505 (2017)
Despite the importance of molecular subtype classification of glioblastoma (GBM), the extent of extracellular vesicle (EV)-driven molecular and phenotypic reprogramming remains poorly understood. To reveal complex subpopulation dynamics within the he
Externí odkaz:
https://doaj.org/article/6a981b2fa5054d1bbf381dcaaae8eaed
Autor:
Daisuke Ogawa, Khairul Ansari, Michal O. Nowicki, Elżbieta Salińska, Agnieszka Bronisz, Jakub Godlewski
Publikováno v:
Non-Coding RNA, Vol 5, Iss 1, p 25 (2019)
Malignant glioblastoma (GBM, glioma) is the most common and aggressive primary adult brain tumor. The prognosis of GBM patients remains poor, despite surgery, radiation and chemotherapy. The major obstacles for successful remedy are invasiveness and
Externí odkaz:
https://doaj.org/article/8214984655a749fb9d0527337e2de589
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e113954 (2014)
The etiology of multiple sclerosis (MS) is currently unknown. However, one potential mechanism involved in the disease may be excitotoxicity. The elevation of glutamate in cerebrospinal fluid, as well as changes in the expression of glutamate recepto
Externí odkaz:
https://doaj.org/article/91916a3145d84f9e9c0752dc4b6ebcd8
Autor:
Elzbieta Salinska, Creed Stary
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 18 (2024)
Externí odkaz:
https://doaj.org/article/ff626952b6654cae8cd4ff86ce85bbe5
Publikováno v:
Frontiers in Neurology, Vol 15 (2024)
Birth asphyxia and its main sequel, hypoxic-ischemic encephalopathy, are one of the leading causes of children’s deaths worldwide and can potentially worsen the quality of life in subsequent years. Despite extensive research efforts, efficient ther
Externí odkaz:
https://doaj.org/article/9af50b3c0df44ddf94db5fee61a044fe
Autor:
Małgorzata, Duszczyk, Marcin, Gamdzyk, Apolonia, Ziembowicz, Paweł M, Boguszewski, Jerzy W, Łazarewicz, Elżbieta, Salińska
Publikováno v:
Acta neurobiologiae experimentalis. 75(4)
Previous studies have demonstrated that repeated submission of rats to mild hypobaric hypoxia reduces the persistent behavioral and hormonal depressive symptoms induced by exposure to footshock in the learned helplessness paradigm. The aim of this st
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::04bf31f464fed3ae7763cbb06f453967
https://pubmed.ncbi.nlm.nih.gov/26994415
https://pubmed.ncbi.nlm.nih.gov/26994415
Publikováno v:
Antioxidants, Vol 10, Iss 11, p 1775 (2021)
The over-activation of NMDA receptors and oxidative stress are important components of neonatal hypoxia–ischemia (HI). Kynurenic acid (KYNA) acts as an NMDA receptor antagonist and is known as a reactive oxygen species (ROS) scavenger, which makes
Externí odkaz:
https://doaj.org/article/93a58beeddcc4ebeb74d1e4501a38547
Publikováno v:
Antioxidants, Vol 9, Iss 9, p 877 (2020)
N-acetylaspartylglutamate (NAAG), the most abundant peptide transmitter in the mammalian nervous system, activates mGluR3 at presynaptic sites, inhibiting the release of glutamate, and acts on mGluR3 on astrocytes, stimulating the release of neuropro
Externí odkaz:
https://doaj.org/article/1c5a70f7e1e34f8f9439f91d90a809d5
Publikováno v:
PLoS ONE, Vol 13, Iss 7, p e0200933 (2018)
Birth asphyxia resulting in brain hypoxia-ischemia (H-I) can cause neonatal death or lead to persistent brain damage. Recent investigations have shown that group II metabotropic glutamate receptor (mGluR2/3) activation can provide neuroprotection aga
Externí odkaz:
https://doaj.org/article/6756e8578e6b4f32925b2b55c65e373c
Publikováno v:
Non-Coding RNA, Vol 5, Iss 1, p 20 (2019)
The mammalian brain is made up of billions of neurons and supporting cells (glial cells), intricately connected. Molecular perturbations often lead to neurodegeneration by progressive loss of structure and malfunction of neurons, including their deat
Externí odkaz:
https://doaj.org/article/198a818187bc4b20a53b54fd6f1f92e8