Zobrazeno 1 - 10
of 172
pro vyhledávání: '"Eisaku, Kondo"'
Autor:
Fan Jiang, Youyi Chen, Nahoko Tomonobu, Rie Kinoshita, Ni Luh Gede Yoni Komalasari, Carlos Ichiro Kasano-Camones, Kazumi Ninomiya, Hitoshi Murata, Ken-ichi Yamamoto, Yuma Gohara, Toshiki Ochi, I Made Winarsa Ruma, I Wayan Sumardika, Jin Zhou, Tomoko Honjo, Yoshihiko Sakaguchi, Akira Yamauchi, Futoshi Kuribayashi, Junichiro Futami, Eisaku Kondo, Yusuke Inoue, Shinichi Toyooka, Masakiyo Sakaguchi
Publikováno v:
Frontiers in Oncology, Vol 14 (2024)
BackgroundTriple-negative breast cancer (TNBC) cells are a highly formidable cancer to treat. Nonetheless, by continued investigation into the molecular biology underlying the complex regulation of TNBC cell activity, vulnerabilities can be exposed a
Externí odkaz:
https://doaj.org/article/d72357d8c23541b5bcda63ea74cc18de
Autor:
Tetta Takahashi, Nahoko Tomonobu, Rie Kinoshita, Ken-ichi Yamamoto, Hitoshi Murata, Ni Luh Gede Yoni Komalasari, Youyi Chen, Fan Jiang, Yuma Gohara, Toshiki Ochi, I Made Winarsa Ruma, I Wayan Sumardika, Jin Zhou, Tomoko Honjo, Yoshihiko Sakaguchi, Akira Yamauchi, Futoshi Kuribayashi, Eisaku Kondo, Yusuke Inoue, Junichiro Futami, Shinichi Toyooka, Yoshito Zamami, Masakiyo Sakaguchi
Publikováno v:
Frontiers in Oncology, Vol 14 (2024)
BackgroundOur earlier research revealed that the secreted lysyl oxidase-like 4 (LOXL4) that is highly elevated in triple-negative breast cancer (TNBC) acts as a catalyst to lock annexin A2 on the cell membrane surface, which accelerates invasive outg
Externí odkaz:
https://doaj.org/article/4ed9034638704304a664180937894550
Autor:
Fan Jiang, Youyi Chen, Nahoko Tomonobu, Rie Kinoshita, Gede Yoni Komalasari, Ni Luh, Ichiro Kasano-Camones, Carlos, Kazumi Ninomiya, Hitoshi Murata, Ken-ichi Yamamoto, Yuma Gohara, Toshiki Ochi, Winarsa Ruma, I Made, Sumardika, I Wayan, Jin Zhou, Tomoko Honjo, Yoshihiko Sakaguchi, Akira Yamauchi, Futoshi Kuribayashi, Junichiro Futami, Eisaku Kondo
Publikováno v:
Frontiers in Oncology; 2024, p1-13, 13p
Autor:
Ken Saito, Hidekazu Iioka, Satoshi Maruyama, I. Wayan Sumardika, Masakiyo Sakaguchi, Eisaku Kondo
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 21, Iss 12, Pp 1121-1132 (2019)
Pancreatic invasive ductal adenocarcinoma (PDAC) is a representative intractable malignancy under the current cancer therapies, and is considered a scirrhous carcinoma because it develops dense stroma. Both PODXL1, a member of CD34 family molecules,
Externí odkaz:
https://doaj.org/article/2687d921d335400bb9ad64352e884503
Autor:
Youyi Chen, I Wayan Sumardika, Nahoko Tomonobu, Rie Kinoshita, Yusuke Inoue, Hidekazu Iioka, Yosuke Mitsui, Ken Saito, I Made Winarsa Ruma, Hiroki Sato, Akira Yamauchi, Hitoshi Murata, Ken-ichi Yamamoto, Shuta Tomida, Kazuhiko Shien, Hiromasa Yamamoto, Junichi Soh, Junichiro Futami, Miyoko Kubo, Endy Widya Putranto, Takashi Murakami, Ming Liu, Toshihiko Hibino, Masahiro Nishibori, Eisaku Kondo, Shinichi Toyooka, Masakiyo Sakaguchi
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 21, Iss 7, Pp 627-640 (2019)
Metastatic breast cancer is the leading cause of cancer-associated death in women. The progression of this fatal disease is associated with inflammatory responses that promote cancer cell growth and dissemination, eventually leading to a reduction of
Externí odkaz:
https://doaj.org/article/242b6d1fb82f4f15bdf82fd9c9d95e87
Autor:
Yushi Ueki, Ken Saito, Hidekazu Iioka, Izumi Sakamoto, Yasuhiro Kanda, Masakiyo Sakaguchi, Arata Horii, Eisaku Kondo
Publikováno v:
iScience, Vol 23, Iss 2, Pp - (2020)
Summary: Identifying the specific functional regulator of integrin family molecules in cancer cells is critical because they are directly involved in tumor invasion and metastasis. Here we report high expression of PLOD2 in oropharyngeal squamous cel
Externí odkaz:
https://doaj.org/article/e2f3e938a5784324b00746dbef6ff44f
Autor:
Eisaku Kondo, Katsuyuki Kiura, Eiki Ichihara, Kazuhiko Kuwahara, Junya Fukuoka, Ryuzo Ueda, Yuki Tomita, Hidekazu Iioka, Naoko Ohtani, Nagio Takigawa, Ken Saito
Activation of the epidermal growth factor receptor (EGFR) has been observed in many malignant tumors and its constitutive signal transduction facilitates the proliferation of tumors. EGFR-tyrosine kinase inhibitors, such as gefitinib, are widely used
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::154cb3c25fc91b1b7a22bc1d64bc8fb9
https://doi.org/10.1158/1535-7163.c.6536568.v1
https://doi.org/10.1158/1535-7163.c.6536568.v1
Autor:
Tadashi Yoshino, Ryuzo Ueda, Isao Date, Akiyoshi Moriwaki, Nobuya Ohara, Koichi Ichimura, Kazuhiro Yoshikawa, Masaki Adachi, Masahiko Hirai, Tomotsugu Ichikawa, Takayoshi Miyake, Takehiro Tanaka, Eisaku Kondo
Molecular targeting agents have become formidable anticancer weapons, which show much promise against the refractory tumors. Functional peptides are among the more desirable of these nanobio-tools. Intracellular delivery of multiple functional peptid
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a7e6f5ef0cb3b1c03b2a042771abfa5
https://doi.org/10.1158/1535-7163.c.6531570.v1
https://doi.org/10.1158/1535-7163.c.6531570.v1
Autor:
Tadashi Yoshino, Ryuzo Ueda, Isao Date, Akiyoshi Moriwaki, Nobuya Ohara, Koichi Ichimura, Kazuhiro Yoshikawa, Masaki Adachi, Masahiko Hirai, Tomotsugu Ichikawa, Takayoshi Miyake, Takehiro Tanaka, Eisaku Kondo
Supplementary Fig. from Potent synergy of dual antitumor peptides for growth suppression of human glioblastoma cell lines
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b344dc43fdaf17fd9abbf13fab08bc48
https://doi.org/10.1158/1535-7163.22484760
https://doi.org/10.1158/1535-7163.22484760
Publikováno v:
FEBS lettersReferences.
OGFOD1, a prolyl-hydroxylase, has been reported to translocate from the nucleus to the cytoplasm in response to cellular stress. Here, we demonstrate that OGFOD1 regulates the transcription and post-transcriptional stabilization of cell cycle-related
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::af436fc5353a2de8c0e9f63aa5e9432e
https://pubmed.ncbi.nlm.nih.gov/36464654
https://pubmed.ncbi.nlm.nih.gov/36464654