Zobrazeno 1 - 10
of 75
pro vyhledávání: '"Eirikur, Benedikz"'
Publikováno v:
Journal of Migration and Health, Vol 8, Iss , Pp 100200- (2023)
Aims: The aim was to evaluate self-reported health status and wellbeing in a well-defined group of refugee families from Syria 2–4 years after resettlement in Denmark, and, where possible, compare it with a Danish reference population. The purpose
Externí odkaz:
https://doaj.org/article/f1002bb621c840b892e9f52255e7561d
Autor:
Nellie A. Martin, Kirsten H. Hyrlov, Maria L. Elkjaer, Eva K. Thygesen, Agnieszka Wlodarczyk, Kirstine J. Elbaek, Christopher Aboo, Justyna Okarmus, Eirikur Benedikz, Richard Reynolds, Zoltan Hegedus, Allan Stensballe, Åsa Fex Svenningsen, Trevor Owens, Zsolt Illes
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Background: MiR-146a is an important regulator of innate inflammatory responses and is also implicated in cell death and survival.Methods: By sorting CNS resident cells, microglia were the main cellular source of miR-146a. Therefore, we investigated
Externí odkaz:
https://doaj.org/article/07b96028e2194af8b68ac37fea6e76a2
Autor:
Nellie A. Martin, Viktor Molnar, Gabor T. Szilagyi, Maria L. Elkjaer, Arkadiusz Nawrocki, Justyna Okarmus, Agnieszka Wlodarczyk, Eva K. Thygesen, Miklos Palkovits, Ferenc Gallyas, Martin R. Larsen, Hans Lassmann, Eirikur Benedikz, Trevor Owens, Asa F. Svenningsen, Zsolt Illes
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
BackgroundThe cuprizone (CPZ) model of multiple sclerosis (MS) was used to identify microRNAs (miRNAs) related to in vivo de- and remyelination. We further investigated the role of miR-146a in miR-146a-deficient (KO) mice: this miRNA is differentiall
Externí odkaz:
https://doaj.org/article/a2c04274028e4e74a0394ae4a70c3f2a
Autor:
Simone Hjæresen, Tobias Sejbaek, Markus Axelsson, Sif Kløvedal Mortensen, Helle Vinsløv-Jensen, Gorm Pihl-Jensen, Lenka Novakova, Christian Bonde Pedersen, Bo Halle, Frantz Rom Poulsen, Mengliang Zhang, Eirikur Benedikz, Jette Lautrup Frederiksen, Jan Lycke, Zsolt Illes, Åsa Fex-Svenningsen
Publikováno v:
Journal of the neurological sciences. 439
Macrophage migration inhibitory factor (MIF) is involved in the function of both the innate and adaptive immune systems and in neuroprotection and has recently been implicated in multiple sclerosis (MS).Determination of MIF levels in the cerebrospina
Autor:
Eirikur Benedikz, Bo Halle, Jette L. Frederiksen, Lenka Novakova, Tobias Sejbaek, Julie Damgaard Rosgaard Christensen, Gorm Pihl-Jensen, Helle Vinsløv-Jensen, Mengliang Zhang, Jan Lycke, Sif Kløvedal Mortensen, Zsolt Illes, Åsa Fex Svenningsen, Christian Bonde Pedersen, Marcus Axelsson, Frantz Rom Poulsen, Simone Hjæresen
Publikováno v:
Hjæresen, S, Sejbaek, T, Axelsson, M, Vinsløv-Jensen, H, Mortensen, S K, Pihl-Jensen, G, Novakova, L, Christensen, J D R, Pedersen, C B, Halle, B, Poulsen, F R, Lautrup Frederiksen, J, Zhang, M, Benedikz, E, Lycke, J, Illes, Z & Fex Svenningsen, Å 2021, ' The levels of the serine protease HTRA1 in cerebrospinal fluid correlate with progression and disability in multiple sclerosis ', Journal of Neurology, vol. 268, no. 9, pp. 3316-3324 . https://doi.org/10.1007/s00415-021-10489-7
BACKGROUND: High Temperature Requirement Serine Protease A1 (HTRA1) degrades extracellular matrix molecules (ECMs) and growth factors. It interacts with several proteins implicated in multiple sclerosis (MS), but has not previously been linked to the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be2dec05a95f76f4ed589eb67889a195
https://findresearcher.sdu.dk:8443/ws/files/180313994/The_levels_of_the_serine_protease_HTRA1_in_cerebrospinal_fluid_correlate_with_progression_and_disability_in_multiple_sclerosis.pdf
https://findresearcher.sdu.dk:8443/ws/files/180313994/The_levels_of_the_serine_protease_HTRA1_in_cerebrospinal_fluid_correlate_with_progression_and_disability_in_multiple_sclerosis.pdf
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0132456 (2015)
A single nucleotide polymorphism in the ZNF804A gene, rs1344706, is associated with schizophrenia. The polymorphism has been suggested to alter fetal expression of ZNF804A. It has also been reported to be associated with altered cortical functioning
Externí odkaz:
https://doaj.org/article/0cacad59e6d440eda3e7317a2ff1ec6a
Autor:
Åke Seiger, Anna Falk, Scott Falci, Emilia Rotstein, Eirikur Benedikz, Ning Xu, Xiaofei Li, Elisabet Åkesson, Erik Sundström, Tingting Xu, Raymond V. Mirasol, Lena Holmberg, Per Henrik Vincent
Publikováno v:
Xu, N, Xu, T, Mirasol, R, Holmberg, L, Vincent, P H, Li, X, Falk, A, Benedikz, E, Rotstein, E, Seiger, Å, Åkesson, E, Falci, S & Sundström, E 2021, ' Transplantation of Human Neural Precursor Cells Reverses Syrinx Growth in a Rat Model of Post-Traumatic Syringomyelia ', Neurotherapeutics, vol. 18, no. 2, pp. 1257-1272 . https://doi.org/10.1007/s13311-020-00987-3
Neurotherapeutics
Neurotherapeutics
Posttraumatic syringomyelia (PTS) is a serious condition of progressive expansion of spinal cord cysts, affecting patients with spinal cord injury years after injury. To evaluate neural cell therapy to prevent cyst expansion and potentially replace l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::677eac4dcfc2f3191a431bf95f61e26a
https://findresearcher.sdu.dk:8443/ws/files/199463983/Xu2021_Article_TransplantationOfHumanNeuralPr.pdf
https://findresearcher.sdu.dk:8443/ws/files/199463983/Xu2021_Article_TransplantationOfHumanNeuralPr.pdf
Autor:
Marianne Pia Lindahl, Signe Andersen, Liselotte Jensen, Annette Joergensen, Christian Frandsen, Eirikur Benedikz
Publikováno v:
Quality of Life Research. 27:267-271
The aim of this study was to translate and culturally adapt the Short Musculoskeletal Function Assessment (SMFA) into Danish (SMFA-DK) and assess the psychometric properties. SMFA was translated and cross-culturally adapted according to a standardize
Publikováno v:
Vincent, P, Benedikz, E, Uhlén, P, Hovatta, O & Sundström, E 2017, ' Expression of Pluripotency Markers in Nonpluripotent Human Neural Stem and Progenitor Cells ', Stem Cells and Development, vol. 26, no. 12, pp. 876-887 . https://doi.org/10.1089/scd.2016.0346
Nonpluripotent neural progenitor cells (NPCs) derived from the human fetal central nervous system were found to express a number of messenger RNA (mRNA) species associated with pluripotency, such as NANOG, REX1, and OCT4. The expression was restricte
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed6f32f9357cc01f476a669c2f93600f
https://portal.findresearcher.sdu.dk/da/publications/896c442d-a360-4aba-af33-ed1b2fff4335
https://portal.findresearcher.sdu.dk/da/publications/896c442d-a360-4aba-af33-ed1b2fff4335
Autor:
Solveig B. Nielsen, Svenja Löring, Maria L. Elkjaer, Eirikur Benedikz, Malin Andersson, Nellie Anne Martin, Uffe Holmskov, Anna Lahn Sørensen, Jesper B. Moeller, Zsolt Illes, Åsa Fex Svenningsen, Simone Hjæresen, Ha Uyen Buu Huynh
Publikováno v:
Cellular and molecular life sciences, 74(24), 4561-4572. Birkhauser Verlag Basel
Cellular and Molecular Life Sciences
Fex Svenningsen, Å, Loering, S, Sørensen, A L, Uyen Buu Huynh, H, Hjæresen, S, Martin, N A, Møller, J B, Elkjær, M L, Holmskov, U, Illés, Z, Andersson, M, Nielsen, S B & Benedikz, E 2017, ' Macrophage migration inhibitory factor (MIF) modulates trophic signaling through interaction with serine protease HTRA1 ', Cellular and Molecular Life Sciences, vol. 74, no. 24, pp. 4561–4572 . https://doi.org/10.1007/s00018-017-2592-z
Cellular and Molecular Life Sciences
Fex Svenningsen, Å, Loering, S, Sørensen, A L, Uyen Buu Huynh, H, Hjæresen, S, Martin, N A, Møller, J B, Elkjær, M L, Holmskov, U, Illés, Z, Andersson, M, Nielsen, S B & Benedikz, E 2017, ' Macrophage migration inhibitory factor (MIF) modulates trophic signaling through interaction with serine protease HTRA1 ', Cellular and Molecular Life Sciences, vol. 74, no. 24, pp. 4561–4572 . https://doi.org/10.1007/s00018-017-2592-z
Macrophage migration inhibitory factor (MIF), a small conserved protein, is abundant in the immune- and central nervous system (CNS). MIF has several receptors and binding partners that can modulate its action on a cellular level. It is upregulated i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f6b87c5bec4a5fc198aaa307735a546e
http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-330365
http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-330365