Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Egle Katkeviciutė"'
Autor:
Gerhard Rogler, Arndt Hartmann, Marianne Rebecca Spalinger, Michael Scharl, Elisabeth Naschberger, Jesus Francisco Glaus Garzon, Silvia Lang, Kirstin Atrott, Philipp Busenhart, Ana Montalban-Arques, Egle Katkeviciute, Yasser Morsy, Chiara Van Passen, Larissa Hering, Michael Stürzl
Publikováno v:
Journal for ImmunoTherapy of Cancer, Vol 10, Iss 2 (2022)
Externí odkaz:
https://doaj.org/article/19040f65b7694fdf88048f127a56fc97
Autor:
Larissa Hering, Egle Katkeviciute, Marlene Schwarzfischer, Philipp Busenhart, Claudia Gottier, Dunja Mrdjen, Juliana Komuczki, Marcin Wawrzyniak, Silvia Lang, Kirstin Atrott, Burkhard Becher, Gerhard Rogler, Michael Scharl, Marianne R. Spalinger
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Protein tyrosine phosphatase non-receptor type 2 (PTPN2) plays a pivotal role in immune homeostasis and has been associated with human autoimmune and chronic inflammatory diseases. Though PTPN2 is well-characterized in lymphocytes, little is known ab
Externí odkaz:
https://doaj.org/article/cbebd70e616b461caf11840570e37a9a
Autor:
Larissa Hering, Egle Katkeviciute, Marlene Schwarzfischer, Anna Niechcial, Julianne B. Riggs, Marcin Wawrzyniak, Kirstin Atrott, Marnix van de Sande, Silvia Lang, Burkhard Becher, Gerhard Rogler, Michael Scharl, Marianne R. Spalinger
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 13, p 6820 (2021)
Protein tyrosine phosphatase nonreceptor type 2 (PTPN2) plays a critical role in the pathogenesis of inflammatory bowel diseases (IBD). Mice lacking PTPN2 in dendritic cells (DCs) develop skin and liver inflammation by the age of 22 weeks due to a ge
Externí odkaz:
https://doaj.org/article/87684b289e354c539e20a9e5c94c2d05
Autor:
Javier Conde, Marlene Schwarzfischer, Egle Katkeviciute, Janine Häfliger, Anna Niechcial, Nathalie Brillant, Roberto Manzini, Katharina Bäbler, Kirstin Atrott, Silvia Lang, Michael Scharl
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 2, p 772 (2021)
Environmental and genetic factors have been demonstrated to contribute to the development of inflammatory bowel disease (IBD). Recent studies suggested that the food additive; titanium dioxide (TiO2) might play a causative role in the disease. Theref
Externí odkaz:
https://doaj.org/article/0f3ef984e76e49c3b4fe9d2d73ab5b86
Autor:
Marlene Schwarzfischer, Anna Niechcial, Kristina Handler, Yasser Morsy, Marcin Wawrzyniak, Andrea S Laimbacher, Kirstin Atrott, Roberto Manzini, Katharina Baebler, Larissa Hering, Egle Katkeviciutė, Janine Häfliger, Silvia Lang, Maja E Keller, Jérôme Woodtli, Lisa Eisenbeiss, Thomas Kraemer, Elisabeth M Schraner, Mahesa Wiesendanger, Sebastian Zeissig, Gerhard Rogler, Andreas E Moor, Michael Scharl, Marianne R Spalinger
Publikováno v:
Gut. 72:1101-1114
ObjectiveInflammatory bowel disease (IBD) is a multifactorial condition driven by genetic and environmental risk factors. A genetic variation in the protein tyrosine phosphatase non-receptor type 22 (PTPN22) gene has been associated with autoimmune d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b82a3a498d670663c1e786d0ad3ad136
https://doi.org/10.1136/gutjnl-2021-325911
https://doi.org/10.1136/gutjnl-2021-325911
Autor:
Marlene, Schwarzfischer, Anna, Niechcial, Kristina, Handler, Yasser, Morsy, Marcin, Wawrzyniak, Andrea S, Laimbacher, Kirstin, Atrott, Roberto, Manzini, Katharina, Baebler, Larissa, Hering, Egle, Katkeviciutė, Janine, Häfliger, Silvia, Lang, Maja E, Keller, Jérôme, Woodtli, Lisa, Eisenbeiss, Thomas, Kraemer, Elisabeth M, Schraner, Mahesa, Wiesendanger, Sebastian, Zeissig, Gerhard, Rogler, Andreas E, Moor, Michael, Scharl, Marianne R, Spalinger
Publikováno v:
Gut.
Inflammatory bowel disease (IBD) is a multifactorial condition driven by genetic and environmental risk factors. A genetic variation in the protein tyrosine phosphatase non-receptor type 22 (PTPN22) gene has been associated with autoimmune disorders
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::262654e5ca5d21c4972015ff59a71543
https://pubmed.ncbi.nlm.nih.gov/36191962
https://pubmed.ncbi.nlm.nih.gov/36191962