Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Efrat Cohen-Hillel"'
Autor:
Christina Katanov, Ben Zion Garty, Adit Ben-Baruch, Efrat Cohen-Hillel, Roni Mintz, Nufar Marcus, Tsipi Meshel
Publikováno v:
Immunology Letters. 130:97-106
Hyper IgE Syndrome (HIES) is a rare genetic disorder, characterized by elevated serum IgE levels and reduced inflammatory responses to bacterial infections. This leads to dermatitis, recurrent lung infections and "cold abscesses". Recently, progress
Publikováno v:
Cellular and Molecular Life Sciences. 66:884-899
The chemokine CXCL8 is a powerful inducer of directional cell motility, primarily during inflammation. In this study, we found that CXCL8 stimulation led to paxillin phosphorylation in normal neutrophils, and that both CXCL8 receptors (CXCR1 and CXCR
Autor:
Marcelo Ehrlich, Efrat Cohen-Hillel, Neora Yaal-Hahoshen, Elina Azenshtein, Larisa Ryvo, Leonor Leider-Trejo, Alex Shtabsky, Iafa Keydar, Gali Soria, Tsipi Meshel, Sima Shina, Adit Ben-Baruch
Publikováno v:
Cytokine. 44:191-200
The chemokines RANTES (CCL5) and MCP-1 (CCL2) were suggested to contribute, independently, to breast malignancy. In the present study, we asked if the two chemokines are jointly expressed in clinical samples of breast cancer patients, and do they int
Publikováno v:
Experimental Cell Research. 314:352-365
In the present study, we used the human chemokine receptors CXCR1 and CXCR2 as a model system for the study of intracellular cross-talk between two closely related G protein-coupled receptors (GPCR). In cells expressing either CXCR1 or CXCR2, exposur
Publikováno v:
Cytokine. 33:1-16
CXCL8 is a potent chemokine, inducing focal adhesion kinase (FAK) phosphorylation, and migration via a FAK-mediated pathway. Since, unlike growth factors, chemokines directly control integrins and cytoskeleton rearrangements, we determined whether th
Publikováno v:
The Israel Medical Association journal : IMAJ. 9(8)
Interleukin 8 is a prototypical inflammatory chemokine that induces leukocyte migration to inflammatory sites. Leukocyte recruitment in response to gradients of this chemokine is attenuated at advanced stages of inflammation to prevent damage to surr