Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Edyta Działo"'
Autor:
Marzena Lenart, Edyta Działo, Anna Kluczewska, Kazimierz Węglarczyk, Anna Szaflarska, Magdalena Rutkowska-Zapała, Marcin Surmiak, Marek Sanak, Anna Pituch-Noworolska, Maciej Siedlar
Publikováno v:
Frontiers in Immunology, Vol 11 (2021)
Severe and/or recurrent infection with Herpes simplex virus (HSV) is observed in a large group of patients treated in clinical immunology facilities. Atypical and prolonged HSV infection is the most common clinical manifestation of disturbed NK cell
Externí odkaz:
https://doaj.org/article/22e013c540ba4195beb82567ab138b4a
Autor:
Karolina Tkacz, Filip Rolski, Marcin Czepiel, Edyta Działo, Maciej Siedlar, Urs Eriksson, Gabriela Kania, Przemysław Błyszczuk
Publikováno v:
Cells, Vol 9, Iss 3, p 700 (2020)
Progressive cardiac fibrosis is a common cause of heart failure. Rho-associated, coiled-coil-containing protein kinases (ROCKs) have been shown to enhance fibrotic processes in the heart and in other organs. In this study, using wild-type, Rock1+/−
Externí odkaz:
https://doaj.org/article/c695e361ea414d19852478cdbfb8c4e7
Autor:
Edyta Działo, Przemyslaw Blyszczuk, Filip Rolski, Marcin Czepiel, Gabriela Kania, Maciej Siedlar, Kazimierz Weglarczyk, Agnieszka Jazwa-Kusior, Karolina Tkacz
Publikováno v:
European Heart Journal. 42
Background/Introduction Heart-specific inflammation – myocarditis is a common cause dilated cardiomyopathy which is characterized by pathological tissue remodeling, ventricular stiffening, cardiomyopathy and heart failure. In experimental autoimmun
Autor:
Gabriela Kania, Maciej Siedlar, Przemyslaw Blyszczuk, Karolina Tkacz, Marcin Czepiel, Edyta Działo
Publikováno v:
International Journal of Molecular Sciences
Volume 22
Issue 18
International Journal of Molecular Sciences, Vol 22, Iss 10072, p 10072 (2021)
Volume 22
Issue 18
International Journal of Molecular Sciences, Vol 22, Iss 10072, p 10072 (2021)
Cardiac fibrosis is a pathological process associated with the development of heart failure. TGF-β and WNT signaling have been implicated in pathogenesis of cardiac fibrosis, however, little is known about molecular cross-talk between these two path
Autor:
Marcin Czepiel, Gabriela Kania, Przemyslaw Blyszczuk, Kazimierz Weglarczyk, Karolina Tkacz, Edyta Działo, Maciej Siedlar
Publikováno v:
European Heart Journal. 41
Background/Introduction Myocarditis, a heart-specific inflammation, is a common cause of pathological tissue remodeling and cardiac fibrosis resulting in stiffening of ventricles, functional impairment and heart failure. Immunization of susceptible m
Autor:
Urs Eriksson, Przemyslaw Blyszczuk, Karolina Tkacz, Filip Rolski, Maciej Siedlar, Edyta Działo, Marcin Czepiel, Gabriela Kania
Publikováno v:
Cells
Volume 9
Issue 3
Cells, Vol 9, Iss 3, p 700 (2020)
Volume 9
Issue 3
Cells, Vol 9, Iss 3, p 700 (2020)
Progressive cardiac fibrosis is a common cause of heart failure. Rho-associated, coiled-coil-containing protein kinases (ROCKs) have been shown to enhance fibrotic processes in the heart and in other organs. In this study, using wild-type, Rock1+/&mi
Cardiac fibrosis is referred to as an excessive accumulation of stromal cells and extracellular matrix proteins in the myocardium. Progressive fibrosis causes stiffening of the cardiac tissue and affects conduction of electrical impulses, leading to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b4ff8a25e55f43ca7a4c75659358a5e1
https://ruj.uj.edu.pl/xmlui/handle/item/93214
https://ruj.uj.edu.pl/xmlui/handle/item/93214