Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Edward T. Kim"'
Publikováno v:
Cells, Vol 12, Iss 11, p 1528 (2023)
Purpose: Inducible Slc4a11 KO leads to corneal edema by disruption of the pump and barrier functions of the corneal endothelium (CE). The loss of Slc4a11 NH3-activated mitochondrial uncoupling leads to mitochondrial membrane potential hyperpolarizati
Externí odkaz:
https://doaj.org/article/a410014bf61d4a0d88a2bfeefddd1547
Autor:
Rajalekshmy Shyam, PhD, Diego G. Ogando, PhD, Edward T. Kim, BS, Subashree Murugan, BOptom, Moonjung Choi, PhD, Joseph A. Bonanno, OD, PhD
Publikováno v:
Ophthalmology Science, Vol 2, Iss 1, Pp 100084- (2022)
Purpose: Congenital hereditary endothelial dystrophy (CHED) is a rare condition that manifests at an early age showing corneal edema, increased oxidative stress, mitochondrial dysfunction, and eventually apoptosis of the endothelium due to loss of fu
Externí odkaz:
https://doaj.org/article/1bdfc09091034259b4b4ac32e785d473
Autor:
Edward T Kim, Marcelo Rezende
Publikováno v:
The Review of Corporate Finance Studies. 12:291-325
Deposit insurance premiums impose costs on banks’ balance sheets, narrowing profit margins and inducing banks to “search for yield.” This paper estimates the effects of deposit insurance premiums on bank portfolio rebalancing using supervisory
Publikováno v:
Cells; Volume 12; Issue 11; Pages: 1528
Purpose: Inducible Slc4a11 KO leads to corneal edema by disruption of the pump and barrier functions of the corneal endothelium (CE). The loss of Slc4a11 NH3-activated mitochondrial uncoupling leads to mitochondrial membrane potential hyperpolarizati
Purpose: Inducible Slc4a11 KO leads to corneal edema by disruption of the pump and barrier functions of the corneal endothelium (CE). The loss of Slc4a11 NH3-activated mitochondrial uncoupling leads to mitochondrial membrane potential hyperpolarizati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fd714d0cf384d90de5270d383ec04f50
https://doi.org/10.20944/preprints202305.1059.v1
https://doi.org/10.20944/preprints202305.1059.v1
Autor:
Rajalekshmy Shyam, Diego G. Ogando, Joseph A. Bonanno, Chia-Yang Liu, Yen-Chiao Wang, Edward T. Kim
Publikováno v:
Investigative ophthalmologyvisual science. 62(7)
Purpose The conventional Slc4a11 knockout (KO) shows significant corneal edema at eye opening, a fact that complicates the study of the initial events leading to edema. An inducible KO would provide opportunities to examine early events following los
Publikováno v:
Investigative Ophthalmology & Visual Science
Purpose Confirm that the corneal endothelial pump uses a lactate-coupled water efflux mechanism. Methods Corneal thickness, lactate efflux, and stromal [lactate] were measured in de-epithelialized swollen and nonswollen ex vivo-mounted rabbit corneas
Autor:
Diego G. Ogando, Moonjung Choi, Rajalekshmy Shyam, Subashree Murugan, Joseph A. Bonanno, Edward T. Kim
Publikováno v:
Ophthalmology Science, Vol 2, Iss 1, Pp 100084-(2022)
Purpose Congenital Hereditary Endothelial Dystrophy (CHED) is a rare condition that manifests at early age showing corneal edema, increased oxidative stress, mitochondrial dysfunction, and eventually apoptosis of the endothelium due to loss of functi
Autor:
Hongde Li, Diego G. Ogando, Joseph A. Bonanno, Wenlin Zhang, Moonjung Choi, Edward T. Kim, Jason M. Tenessen
Publikováno v:
Investigative Ophthalmology & Visual Science
Purpose To establish conditionally immortal mouse corneal endothelial cell lines with genetically matched Slc4a11+/+ and Slc4a11-/- mice as a model for investigating pathology and therapies for SLC4A11 associated congenital hereditary endothelial dys
Publikováno v:
American Journal of Physiology-Cell Physiology. 311:C116-C126
Maintenance of corneal hydration is dependent on the active transport properties of the corneal endothelium. We tested the hypothesis that lactic acid efflux, facilitated by buffering, is a component of the endothelial fluid pump. Rabbit corneas were