Zobrazeno 1 - 10
of 180
pro vyhledávání: '"Edward S Chen"'
Autor:
Roman E Magallon, Laura D Harmacek, Nicholas K Arger, Pineet Grewal, Linda Powers, Brenda R Werner, Briana Q Barkes, Li Li, Kristyn MacPhail, May Gillespie, Elizabeth K White, Sarah E Collins, Talyor Brown, Jessica Cardenas, Edward S Chen, Lisa A Maier, Sonia M Leach, Nabeel Y Hamzeh, Laura L Koth, Brian P O'Connor
Publikováno v:
PLoS ONE, Vol 18, Iss 3, p e0281210 (2023)
The contribution and regulation of various CD4+ T cell lineages that occur with remitting vs progressive courses in sarcoidosis are poorly understood. We developed a multiparameter flow cytometry panel to sort these CD4+ T cell lineages followed by m
Externí odkaz:
https://doaj.org/article/78fc6a48ef654c3da5acbddbd7d70f96
Autor:
Li Li, Edward S Chen, Linda Powers, Sonia M Leach, Laura L Koth, Lisa A Maier, Laura D Harmacek, Elizabeth K White, Nicholas Kostandinos Arger, Brenda R Werner, Roman E Magallon, Pineet Grewal, Briana Q Barkes, May Gillespie, Sarah E Collins, Jessica Cardenas, Brian P O’Connor, Nabeel Y Hamzeh
Publikováno v:
BMJ Open, Vol 11, Iss 11 (2021)
Introduction Sarcoidosis is a multiorgan granulomatous disorder thought to be triggered and influenced by gene–environment interactions. Sarcoidosis affects 45–300/100 000 individuals in the USA and has an increasing mortality rate. The greatest
Externí odkaz:
https://doaj.org/article/8897a02a81d842018cb40e5c8b2a7444
Publikováno v:
PLoS Genetics, Vol 15, Iss 7, p e1008287 (2019)
CD36 is a platelet membrane glycoprotein whose engagement with oxidized low-density lipoprotein (oxLDL) results in platelet activation. The CD36 gene has been associated with platelet count, platelet volume, as well as lipid levels and CVD risk by ge
Externí odkaz:
https://doaj.org/article/69c6fd46da1d4e02802974eb75c2b360
Autor:
Tong Zhou, Wei Zhang, Nadera J Sweiss, Edward S Chen, David R Moller, Kenneth S Knox, Shwu-Fan Ma, Michael S Wade, Imre Noth, Roberto F Machado, Joe G N Garcia
Publikováno v:
PLoS ONE, Vol 7, Iss 9, p e44818 (2012)
Sarcoidosis, a systemic granulomatous syndrome invariably affecting the lung, typically spontaneously remits but in ~20% of cases progresses with severe lung dysfunction or cardiac and neurologic involvement (complicated sarcoidosis). Unfortunately,
Externí odkaz:
https://doaj.org/article/2657016fb3344bc88ece2409bdeca797
Autor:
Ye Cao, Mari J. Tokita, Edward S. Chen, Rajarshi Ghosh, Tiansheng Chen, Yanming Feng, Elizabeth Gorman, Federica Gibellini, Patricia A. Ward, Alicia Braxton, Xia Wang, Linyan Meng, Rui Xiao, Weimin Bi, Fan Xia, Christine M. Eng, Yaping Yang, Tomasz Gambin, Chad Shaw, Pengfei Liu, Pawel Stankiewicz
Publikováno v:
Genome Medicine, Vol 11, Iss 1, Pp 1-11 (2019)
Abstract Background Although mosaic variation has been known to cause disease for decades, high-throughput sequencing technologies with the analytical sensitivity to consistently detect variants at reduced allelic fractions have only recently emerged
Externí odkaz:
https://doaj.org/article/86ee908a71bb4a7ab56d4cd217c72cb4
Autor:
Alison L. Wand, Noelle Pavlovic, Chloe Duvall, Natalie S. Rosen, Jessica Chasler, Jan M. Griffin, David R. Okada, Artrish Jefferson, Jonathan Chrispin, Harikrishna Tandri, Stephen C. Mathai, Michelle Sharp, Edward S. Chen, Edward K. Kasper, Allison G. Hays, Nisha A. Gilotra
Publikováno v:
The American Journal of Cardiology. 177:108-115
Cardiac sarcoidosis (CS) is an important cause of cardiomyopathy. The trajectory of left ventricular ejection fraction (LVEF) in patients with CS undergoing treatment remains unclear. Patients with CS who were treated with corticosteroids and who und
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ec12c2b3586d6941c4b797706ca94e5f
https://doi.org/10.1016/j.amjcard.2022.04.051
https://doi.org/10.1016/j.amjcard.2022.04.051
Autor:
Katharine E. Henry, Roy Adams, Cassandra Parent, Hossein Soleimani, Anirudh Sridharan, Lauren Johnson, David N. Hager, Sara E. Cosgrove, Andrew Markowski, Eili Y. Klein, Edward S. Chen, Mustapha O. Saheed, Maureen Henley, Sheila Miranda, Katrina Houston, Robert C. Linton, Anushree R. Ahluwalia, Albert W. Wu, Suchi Saria
Publikováno v:
Nature Medicine. 28:1447-1454
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::24c46d056668d7642055935851a19857
https://doi.org/10.1038/s41591-022-01895-z
https://doi.org/10.1038/s41591-022-01895-z
Autor:
Roy Adams, Katharine E. Henry, Anirudh Sridharan, Hossein Soleimani, Andong Zhan, Nishi Rawat, Lauren Johnson, David N. Hager, Sara E. Cosgrove, Andrew Markowski, Eili Y. Klein, Edward S. Chen, Mustapha O. Saheed, Maureen Henley, Sheila Miranda, Katrina Houston, Robert C. Linton, Anushree R. Ahluwalia, Albert W. Wu, Suchi Saria
Publikováno v:
Nature Medicine. 28:1455-1460
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f6a1248e61d81d8b76978eb37afaf8c6
https://doi.org/10.1038/s41591-022-01894-0
https://doi.org/10.1038/s41591-022-01894-0
Autor:
CHLOE DUVALL, NOELLE PAVLOVIC, NATALIE S. ROSEN, ALISON L. WAND, JAN M. GRIFFIN, DAVID R. OKADA, HARIKRISHNA TANDRI, EDWARD K. KASPER, MICHELLE SHARP, EDWARD S. CHEN, JONATHAN CHRISPIN, NISHA A. GILOTRA
Publikováno v:
Journal of Cardiac Failure.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::340f3344d23d5a0b8674584115ed299e
https://doi.org/10.1016/j.cardfail.2023.03.022
https://doi.org/10.1016/j.cardfail.2023.03.022
Autor:
Rachel Schiff, C. Kent Osborne, Jorge S. Reis-Filho, Britta Weigelt, Joe W. Gray, Mothaffar F. Rimawi, Susan G. Hilsenbeck, Chad A. Shaw, Ben H. Park, Gary C. Chamness, Felipe C. Geyer, Chandandeep Nagi, Carolina Gutierrez, Kenneth L. Scott, Daniel J. Zabransky, Ian Waters, Mahitha Rajendran, Tamika Mitchell, Martin Shea, Sarmistha Nanda, Tao Wang, Alexander Renwick, Edward S. Chen, Charlotte K.Y. Ng, Nicholas Wang, Laura M. Heiser, Vidyalakshmi Sethunath, Jamunarani Veeraraghavan, Lanfang Qin, Huizhong Hu, Agostina Nardone, Kathleen A. Burke, Carmine De Angelis, Xiaowei Xu
Purpose: Resistance to anti-HER2 therapies in HER2+ breast cancer can occur through activation of alternative survival pathways or reactivation of the HER signaling network. Here we employed BT474 parental and treatment-resistant cell line models to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e201b57c505d629e55aa1001b1f5bfb
https://doi.org/10.1158/1078-0432.c.6526164.v1
https://doi.org/10.1158/1078-0432.c.6526164.v1
