Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Eduardo E. Cervera-Ceballos"'
Autor:
Yael Cazares-Ordonez, Elizabeth Guadarrama-Beltran, Erick Crespo-Solís, Alvaro Aguayo, Xavier López-Karpovitch, Eduardo E. Cervera-Ceballos, Eunice Garcia-Alvarez, Deborah Martinez-Baños
Publikováno v:
Clinical Leukemia. 2:261-266
Background: Chronic myeloid leukemia (CML) is a malignant disease caused by the protein coded by the Philadelphia chromosome. Imatinib mesylate (IM) is now the treatment of choice for all stages of CML. Clinicoepidemiologic information on CML from de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ad39a1aa3426cc0b789659d8ed7e7ad8
https://doi.org/10.3816/clk.2008.n.036
https://doi.org/10.3816/clk.2008.n.036
Autor:
Myrna Candelaria-Hernández, Judith Cruz-Velazquez, Pablo Vargas-Viveros, Rafael Hurtado-Monroy, Eduardo E Cervera-Ceballos, Silvia Carrillo-Muñoz
Publikováno v:
Blood. 120:1689-1689
Abstract 1689 The increase dose of Imatinib, the shift to a Tyrosine Kinase (TKI) inhibitor of second generation or hematopoietic stem cell transplantation are treatment options when there is loss or lack of response to IM in CML; However, in patient
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::547e24ceedd5629c01988ca6b414debd
https://doi.org/10.1182/blood.v120.21.1689.1689
https://doi.org/10.1182/blood.v120.21.1689.1689
Autor:
Deborah Martínez-Baños, Alvaro Aguayo, Erick Crespo-Solís, Guadarrama-Beltran S.W. Elizabeth, Yael Cazares-Ordonez, Eduardo E. Cervera-Ceballos, Eunice Garcia-Alvarez, Xavier López-Karpovitch
Publikováno v:
Blood. 110:4557-4557
Chronic myeloid leukemia (CML) is a malignant disease of the hematopoietic stem cell caused by the t(9;22)(q13;q11). The disfunctional hybrid-protein (PBCR-ABL) produced has a tyrosin kinase activity leading to a factor-independent myeloid proliferat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f71eaa37456291a1a1941b1bfdf7c32f
https://doi.org/10.1182/blood.v110.11.4557.4557
https://doi.org/10.1182/blood.v110.11.4557.4557