Zobrazeno 1 - 10
of 55
pro vyhledávání: '"Eduard, Berenshtein"'
Publikováno v:
Diabetes & Metabolism Journal, Vol 48, Iss 1, Pp 59-71 (2024)
Background The inflammatory process is known to be an integral part of the pathophysiology of type 2 diabetes mellitus (T2DM). The “labile,” redox-active iron, serving as a catalyst in Fenton reaction, producing the deleterious reactive oxygen sp
Externí odkaz:
https://doaj.org/article/d7717becb79d4ce297b91404ed96c9b9
Autor:
Haim Bibi, Vladimir Vinokur, Dan Waisman, Yigal Elenberg, Amir Landesberg, Anna Faingersh, Moran Yadid, Vera Brod, Jimy Pesin, Eduard Berenshtein, Ron Eliashar, Mordechai Chevion
Publikováno v:
Redox Biology, Vol 2, Iss C, Pp 814-819 (2014)
Background: Redox-active iron, a catalyst in the production of hydroxyl radicals via the Fenton reaction, is one of the key participants in ROS-induced tissue injury and general inflammation. According to our recent findings, an excess of tissue iron
Externí odkaz:
https://doaj.org/article/23f60f1f06184bffb32118f056689b2e
Autor:
Sharon Karniely, Adi Faigenboim, Salsabeel Watted, Katia Lapin, Eduard Berenshtein, Avshalom Hurvitz, Arieli Bouznach, Ezra Rozenblut, Massimo Orioles, Marco Galeotti, Irene Salinas, Asaf Berkowitz, Eran Bacharach, Avi Eldar
Publikováno v:
iScience
Rainbow trout (Oncorhynchus mykiss) is the principal species of inland-farmed fish in the Western hemisphere. Recently, we diagnosed in farmed rainbow trout a disease in which the hallmark is granulomatous-like hepatitis. No biotic agents could be is
Publikováno v:
PLoS ONE, Vol 11, Iss 7, p e0159951 (2016)
Preconditioning (PC) procedures (ischemic or pharmacological) are powerful procedures used for attaining protection against prolonged ischemia and reperfusion (I/R) injury, in a variety of organs, including the heart. The detailed molecular mechanism
Externí odkaz:
https://doaj.org/article/dfbc53d4891a4d129a1b01d06be1207a
Publikováno v:
PLoS ONE, Vol 11, Iss 7, p e0159908 (2016)
Whether the diabetic heart benefits from ischemic preconditioning (IPC), similar to the non-diabetic heart, is a subject of controversy. We recently proposed new roles for iron and ferritin in IPC-protection in Type 1-like streptozotocin-induced diab
Externí odkaz:
https://doaj.org/article/2322660d167847a6b1ddd5d11c7f34ef
Autor:
Michal Lederman, Shira Hagbi-Levi, Michelle Grunin, Alexey Obolensky, Eduard Berenshtein, Eyal Banin, Mordechai Chevion, Itay Chowers
Publikováno v:
PLoS ONE, Vol 9, Iss 2, p e87751 (2014)
Oxidative injury is involved in retinal and macular degeneration. We aim to assess if retinal degeneration associated with genetic defect modulates the retinal threshold for encountering additional oxidative challenges.Retinal oxidative injury was in
Externí odkaz:
https://doaj.org/article/34264103996e4f829ff4402d9ae43170
Autor:
Vladimir Vinokur, Eduard Berenshtein, Baruch Bulvik, Leonid Grinberg, Ron Eliashar, Mordechai Chevion
Publikováno v:
PLoS ONE, Vol 8, Iss 5, p e62948 (2013)
Cardiovascular dysfunction is a major complication of diabetes. Examining mechanistic aspects underlying the incapacity of the diabetic heart to respond to ischemic preconditioning (IPC), we could show that the alterations in iron homeostasis can exp
Externí odkaz:
https://doaj.org/article/6dde990495b24f25a4f17cd677e4dbe9
Autor:
Baruch E Bulvik, Eduard Berenshtein, Esther G Meyron-Holtz, Abraham M Konijn, Mordechai Chevion
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e48947 (2012)
Ischemia associated injury of the myocardium is caused by oxidative damage during reperfusion. Myocardial protection by ischemic preconditioning (IPC) was shown to be mediated by a transient 'iron-signal' that leads to the accumulation of apoferritin
Externí odkaz:
https://doaj.org/article/3b8ef50245984185810951eb638c1f4d
Autor:
Mordechai Chevion, Vera Brod, Dan Waisman, Haim Bibi, Eduard Berenshtein, Ron Eliashar, Jimy Pesin, Yigal Elenberg, Anna Faingersh, Moran Yadid, Amir Landesberg, Vladimir Vinokur
Publikováno v:
Redox Biology, Vol 2, Iss C, Pp 814-819 (2014)
Redox Biology
Redox Biology
Background Redox-active iron, a catalyst in the production of hydroxyl radicals via the Fenton reaction, is one of the key participants in ROS-induced tissue injury and general inflammation. According to our recent findings, an excess of tissue iron
Publikováno v:
PLoS ONE, Vol 11, Iss 7, p e0159908 (2016)
PLoS ONE
PLoS ONE
Whether the diabetic heart benefits from ischemic preconditioning (IPC), similar to the non-diabetic heart, is a subject of controversy. We recently proposed new roles for iron and ferritin in IPC-protection in Type 1-like streptozotocin-induced diab