Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Edith Sturm"'
Autor:
Violetta Refolo, Francesco Bez, Alexia Polissidis, Daniela Kuzdas-Wood, Edith Sturm, Martina Kamaratou, Werner Poewe, Leonidas Stefanis, M. Angela Cenci, Marina Romero-Ramos, Gregor K. Wenning, Nadia Stefanova
Publikováno v:
Acta Neuropathologica Communications, Vol 6, Iss 1, Pp 1-24 (2018)
Abstract Multiple system atrophy (MSA) is a rapidly progressive neurodegenerative disorder characterized by widespread oligodendroglial cytoplasmic inclusions of filamentous α-synuclein, and neuronal loss in autonomic centres, basal ganglia and cere
Externí odkaz:
https://doaj.org/article/16be6e442f47485baeb4dc246067118b
Autor:
Simon Schafferer, Rimpi Khurana, Violetta Refolo, Serena Venezia, Edith Sturm, Paolo Piatti, Clara Hechenberger, Hubert Hackl, Roman Kessler, Michaela Willi, Ronald Gstir, Anne Krogsdam, Alexandra Lusser, Werner Poewe, Gregor K Wenning, Alexander Hüttenhofer, Nadia Stefanova
Publikováno v:
PLoS ONE, Vol 11, Iss 3, p e0150705 (2016)
Multiple system atrophy (MSA) is a fatal rapidly progressive α-synucleinopathy, characterized by α-synuclein accumulation in oligodendrocytes. It is accepted that the pathological α-synuclein accumulation in the brain of MSA patients plays a leadi
Externí odkaz:
https://doaj.org/article/40afa1d6303546e095d2e4162e6a0a06
Autor:
Nadia Stefanova, Edith Sturm
Publikováno v:
Experimental Neurobiology
Multiple system atrophy (MSA) is a rare, late-onset and fatal neurodegenerative disease including multisystem neurodegeneration and the formation of α-synuclein containing oligodendroglial cytoplasmic inclusions (GCIs), which present the hallmark of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbba39b61c0c48712fb2248de67966ef
https://doi.org/10.5607/en.2014.23.4.277
https://doi.org/10.5607/en.2014.23.4.277
Publikováno v:
Neurotherapeutics
Similar to Parkinson disease, multiple system atrophy (MSA) presents neuropathologically with nigral neuronal loss; however, the hallmark intracellular α-synuclein (αSyn) accumulation in MSA affects typically oligodendrocytes to form glial cytoplas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::546237c7bfb62c06515897b3a4a09cc9