Contractile force is enhanced in Aortas from pendrin null mice due to stimulation of angiotensin II-dependent signaling

Autor: Wall, Susan, Sutliff, RL, Walp, ER, Kim, YH, Walker, LA, El-Ali, AM, Ma, J, Bonsall, R, Ramosevac, S, Eaton, DC, Verlander, JW

Publikováno v: Wall, Susan; Sutliff, RL; Walp, ER; Kim, YH; Walker, LA; El-Ali, AM; et al.(2014). Contractile force is enhanced in Aortas from pendrin null mice due to stimulation of angiotensin II-dependent signaling.. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/4ww9068h

Pendrin is a Cl-/HCO3- exchanger expressed in the apical regions of renal intercalated cells. Following pendrin gene ablation, blood pressure falls, in part, from reduced renal NaCl absorption. We asked if pendrin is expressed in vascular tissue and

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______325::3e8835d739e7313453b185c158bd660a
http://www.escholarship.org/uc/item/4ww9068h

S-adenosyl-L-homocysteine hydrolase is necessary for aldosterone-induced activity of epithelial Na+ channels

Autor: Stockand, JD, Zeltwanger, S, Bao,H F, Worrell, RT, Eaton, DC, BECCHETTI, ANDREA

The A6 cell line was used to study the role of S-adenosyl-L-homocysteine hydrolase (SAHHase) in the aldosterone-induced activation of the epithelial Na+ channel (ENaC). Because aldosterone increases methylation of several different molecules, and bec

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______1299::740e81096a13500f8e811ee1b1da36da
http://hdl.handle.net/10281/526

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