Zobrazeno 1 - 10
of 83
pro vyhledávání: '"Earl E. Henderson"'
Autor:
Oleg Chertov, Ning Zhang, Xin Chen, Joost J Oppenheim, Jacek Lubkowski, Connor McGrath, Raymond C Sowder, Bruce J Crise, Anatoli Malyguine, Michele A Kutzler, Amber D Steele, Earl E Henderson, Thomas J Rogers
Publikováno v:
PLoS ONE, Vol 6, Iss 1, p e14474 (2011)
The sequential interaction of the envelope glycoprotein of the human immunodeficiency virus type 1 (HIV-1) with CD4 and certain chemokine coreceptors initiates host cell entry of the virus. The appropriate chemokines have been shown to inhibit viral
Externí odkaz:
https://doaj.org/article/c33e88f88d76462f85eb5258f040392a
Publikováno v:
Antiviral Research. 72:233-241
The ability of HIV-1 to form latent reservoirs presents a major obstacle to eradication. One approach to elimination of the latent reservoir is induction therapy, whereby cells harboring latent virus are activated and therefore initiate virus replica
Publikováno v:
Virology. 352(2):253-267
Herpesvirus saimiri (HVS)-transformed human T cells become permissive for X4 and R5 strains of human immunodeficiency virus type 1 (HIV-1), evidence that HVS-encoded proteins associated with T cell transformation enhance HIV-1 replication. Analyzing
Publikováno v:
Virology. 324:60-66
Herpesvirus saimiri (HVS)-transformed T-lymphocytes are permissive for both X4 and R5 strains of human immunodeficiency virus type 1 (HIV-1). HVS-encoded proteins tyrosine-kinase interacting protein (Tip) and saimiri transformation-associated protein
Autor:
O. M. Zack Howard, Filip Bednar, Earl E. Henderson, Amber D. Steele, Imre Szabo, David E. Kaminsky, Michele A. Wetzel, Chongguang Chen, Joost J. Oppenheim, Lee-Yuan Liu-Chen, Thomas J. Rogers, Ning Zhang
Publikováno v:
Journal of Leukocyte Biology. 74:1074-1082
The opiates are well-established immunomodulatory factors, and recent evidence suggests that μ- and δ-opioid receptor ligands alter chemokine-driven chemotactic responses through the process of heterologous desensitization. In the present report, w
Publikováno v:
Virology. 309:99-107
A substantial proportion of HIV-1-infected individuals are intravenous drug users (i.v.DUs) who abuse opiates. Opioids induce a number of immunomodulatory effects that may directly influence HIV-1 disease progression. In the present report, we have i
Autor:
Joseph W. Homan, Kathryn T. Iacono, Robert J. Suhadolnik, Ramamurthy Charubala, Camille Martinand-Mari, Susan E. Horvath, Wolfgang Pfleiderer, Amber D. Steele, Earl E. Henderson, Nicholas F. Muto, Thomas J. Rogers
Publikováno v:
Helvetica Chimica Acta. 85:2284-2299
A new labelling technique attaching fluorescein via a carbamoyl linker directly to the amino groups of the nucleobases was developed. The amino groups were first converted to the phenoxycarbonyl derivatives (10, 15, 19, 58), which reacted under mild
Autor:
Ramamurthy Charubala, Thomas J. Rogers, Nancy L. Reichenbach, Amber D. Steele, Earl E. Henderson, Camille Martinand-Mari, Wolfgang Pfleiderer, Robert J. Suhadolnik, Joseph W. Homan
Publikováno v:
Journal of Acquired Immune Deficiency Syndromes
Journal of Acquired Immune Deficiency Syndromes, Lippincott, Williams & Wilkins, 2002, 30 (1), pp.9-20. ⟨10.1097/00042560-200205010-00002⟩
Journal of Acquired Immune Deficiency Syndromes, Lippincott, Williams & Wilkins, 2002, 30 (1), pp.9-20. ⟨10.1097/00042560-200205010-00002⟩
Opioids potentiate HIV-1 infection in vitro at least partly by suppressing immunoresponsive processes in human lymphocytes and monocytes. For example, it appears that morphine inhibits the interferon (IFN)-alpha, -beta, and -gamma-mediated natural an
Publikováno v:
Virology. 292:6-15
To better define a mechanism underlying the increase in expression of certain proinflammatory chemokines during HIV-1 infection, we analyzed the effect of X4 HIV-1 infection on C, C-C, and C-X-C chemokine mRNA levels. We demonstrate that X4 HIV-1 inf
Autor:
Wanghua Gong, Earl E. Henderson, Thomas J. Rogers, Michele A. Wetzel, Weiping Shen, Ji Ming Wang, Yingying Le
Publikováno v:
Clinical Immunology. 99:365-372
Chemokine receptors are subjected to heterologous desensitization by activation of formyl peptide receptors. We investigated the cross-talk between formyl peptide receptors and the chemokine receptor CCR5 in human monocyte-differentiated immature den