Stabilization of atherosclerotic plaques: an update

Autor: Yla-Herttuala, S., Bentzon, J.F., Daemen, M., Falk, E., Garcia-Garcia, H.M., Herrmann, J., Hoefer, I., Jauhiainen, S., Jukema, J.W., Krams, R., Kwak, B.R., Marx, N., Naruszewicz, M., Newby, A., Pasterkamp, G., Serruys, P.W.J.C., Waltenberger, J., Weber, C., Tokgozoglu, L., ESC Working Grp Atherosclerosis Va

Publikováno v: European Heart Journal, 34(42), 3251-3258
EUROPEAN HEART JOURNAL
European heart journal, 34(42), 3251-3258. Oxford University Press
Ylä-Herttuala, S, Bentzon, J F, Daemen, M, Falk, E, Garcia-Garcia, H M, Herrmann, J, Hoefer, I, Jauhiainen, S, Jukema, J W, Krams, R, Kwak, B R, Marx, N, Naruszewicz, M, Newby, A, Pasterkamp, G, Serruys, P W J C, Waltenberger, J, Weber, C, Tokgözoglu, L & ESC Working Group of Atherosclerosis and Vascular Biology 2013, ' Stabilization of atherosclerotic plaques : an update ', European Heart Journal . https://doi.org/10.1093/eurheartj/eht301
European Heart Journal, 34(42), 3251-3258. Oxford University Press
Europe PubMed Central
European heart journal

The majority of coronary thrombi (∼75%) is caused by plaque rupture .1,2 Prototype of the rupture-prone plaque contains a large, soft, lipid-rich necrotic core with a thin and inflamed fibrous cap, so-called thin-cap fibroatheroma (TCFA) ( Figure 1

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https://hdl.handle.net/1765/52935