Zobrazeno 1 - 10
of 299
pro vyhledávání: '"EHRLICH HP"'
Autor:
Dallon JC (AUTHOR), Ehrlich HP (AUTHOR)
Publikováno v:
Wound Repair & Regeneration. Jul/Aug2008, Vol. 16 Issue 4, p472-479. 8p.
Publikováno v:
The Journal of Trauma: Injury, Infection, and Critical Care. 47:533-537
BACKGROUND Tumor necrosis factor (TNF) is an important mediator of impaired wound healing during sepsis. To determine whether the inhibitory effects of systemic TNF on wound healing are mediated directly by TNF or by means of the induction of interle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d1152d4f395f53277f7f63d64dc4c028
https://doi.org/10.1097/00005373-199909000-00018
https://doi.org/10.1097/00005373-199909000-00018
Publikováno v:
Plastic and Reconstructive Surgery. 101:6-11
Human amniotic fluid inhibits the contraction of fibroblast-populated collagen lattices. Amniotic fluid of 21 weeks' gestation was previously reported to have peak inhibiting activity. An attempt to isolate the inhibitory factor(s) from human amnioti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::609efa3a665de9cc7da6bb145e448e55
https://doi.org/10.1097/00006534-199801000-00002
https://doi.org/10.1097/00006534-199801000-00002
Publikováno v:
Plastic and Reconstructive Surgery. 91:1287-1293
The effect of human amniotic fluid on fetal wound healing remains to be fully elucidated and may lead to the isolation of factors that could modulate adult wound healing. This study uses an in vitro model of wound contraction, the fibroblast-populate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d82d97bf58d9b33bd59c87be77384eff
https://doi.org/10.1097/00006534-199306000-00015
https://doi.org/10.1097/00006534-199306000-00015
Autor:
Rockwell Wb, Ehrlich Hp
Publikováno v:
Journal of Burn Care & Rehabilitation. 13:403-406
Treatment of the patient with burns has advanced dramatically during the last several decades. Patients with burns that cover a greater percentage of the body surface area are now surviving. Cardiac, pulmonary, and vascular insults are better underst
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15d3a8a1c89455e6178a289539856cdd
https://doi.org/10.1097/00004630-199207000-00003
https://doi.org/10.1097/00004630-199207000-00003
Publikováno v:
Experimental Biology and Medicine. 194:320-326
Daily therapeutic injections of cortisone to rats will cause weight loss and impaired wound healing. Weight loss is attributed to the catabolic effect of steroid, whereas impaired healing is associated with reductions in fibroplasia and connective ti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8c5a031703c56ab42601955e9fa04f2
https://doi.org/10.3181/00379727-194-43098
https://doi.org/10.3181/00379727-194-43098
Publikováno v:
Plastic and reconstructive surgery. 107(2)
Repair of incision wounds closed by suturing is evaluated by the progressive gain in wound breaking strength. Previously the closure of open wounds in rats ingesting vanadate, an inhibitor of tyrosine phosphate phosphatases, was shown to occur with d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d584d30fbac5832609fb88ecbd0fcdce
https://pubmed.ncbi.nlm.nih.gov/11214063
https://pubmed.ncbi.nlm.nih.gov/11214063
Publikováno v:
Archives of surgery (Chicago, Ill. : 1960). 134(5)
Hypothesis Fibroblasts, not myofibroblasts, are responsible for wound contraction. Only myofibroblasts express a smooth muscle actin for which vanadate blocks its expression. Wound contraction in vanadate-treated rats will proceed normally in the abs
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a6b70fc4bfe087712240afb3a89e415c
https://pubmed.ncbi.nlm.nih.gov/10323421
https://pubmed.ncbi.nlm.nih.gov/10323421
Publikováno v:
The Journal of burn carerehabilitation. 12(2)
A model of full-thickness dermal necrosis was produced in rats by the application of liquid nitrogen to a 20% total body surface area of the dorsal skin surface. In this model there was an alteration of reticuloendothelial system phagocytic activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ce967ae0741b80e6d838dc23fd24024
https://pubmed.ncbi.nlm.nih.gov/2050735
https://pubmed.ncbi.nlm.nih.gov/2050735