Zobrazeno 1 - 3
of 3
pro vyhledávání: '"EDUARDO B.B. CUNHA"'
Autor:
EDUARDO B.B. CUNHA, NATÁLIA F. DA SILVA, JEAN DE LIMA, JULIA A. SERRATO, CARLOS A.M. AITA, ROBERTO H. HERAI
Publikováno v:
Anais da Academia Brasileira de Ciências, Vol 92, Iss 4 (2020)
Abstract Atherosclerosis is caused by a monocyte-mediated inflammatory process that, in turn, is stimulated by cytokines and adhesion molecules. Monocytes are then differentiated into macrophages, leading to the formation of arterial atherosclerotic
Externí odkaz:
https://doaj.org/article/680abd4072a24eb987f3c15bbd0a830d
Autor:
FERNANDA D.M. CAMARA, GIULIA S. PEDROSO, SILVANE S. ROMAN, ROGÉRIO M. DALLAGO, ALICE T. VALDUGA, BRUNA B. FERNANDES, EDUARDO B.B. CUNHA, PAULO CESAR L. SILVEIRA, RENATA T. NESI, RICARDO A. PINHO
Publikováno v:
Anais da Academia Brasileira de Ciências, Vol 92, Iss 2 (2020)
Abstract The aim of this study was to investigate the effects of yerba mate (Ilex paraguariensis St. Hil.) extract (YME) on oxidative stress parameters and pathological changes in the lungs of mice chronically exposed to hand-rolled cornhusk cigarett
Externí odkaz:
https://doaj.org/article/844d0f31ff23469dab78af59fa8dd908
Autor:
EDUARDO B.B. CUNHA, NATÁLIA F. DA SILVA, JEAN DE LIMA, JULIA A. SERRATO, CARLOS A.M. AITA, ROBERTO H. HERAI
Publikováno v:
Anais da Academia Brasileira de Ciências v.92 n.4 2020
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Volume: 92, Issue: 4, Article number: e20191486, Published: 20 NOV 2020
Anais da Academia Brasileira de Ciências, Vol 92, Iss 4 (2020)
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Volume: 92, Issue: 4, Article number: e20191486, Published: 20 NOV 2020
Anais da Academia Brasileira de Ciências, Vol 92, Iss 4 (2020)
Atherosclerosis is caused by a monocyte-mediated inflammatory process that, in turn, is stimulated by cytokines and adhesion molecules. Monocytes are then differentiated into macrophages, leading to the formation of arterial atherosclerotic plaques.