Zobrazeno 1 - 10
of 46
pro vyhledávání: '"E. Yu. Moskaleva"'
Autor:
A. M. Golubev, E. Yu. Moskaleva, S. E. Severin, T. P. Vesnyanko, A. N. Kouzovlev, A. S. Alkadarsky, G. G. Poroshenko
Publikováno v:
Общая реаниматология, Vol 2, Iss 6, Pp 184-190 (2006)
Apoptosis is a variant of programmed cell death. This term was introduced by Kerr et al. in 1972, but information on the important role of apoptosis of some cells in critical conditions has recently appeared. The review of literature considers the ba
Externí odkaz:
https://doaj.org/article/32908bc0aa6a429d998ae78cd725db3a
Autor:
M. G. Ratushnyak, A. S. Zhirnik, O. D. Smirnova, Yu.P. Semochkina, A. A. Parfenova, K. V. Goryunov, D. N. Silachev, E. Yu. Moskaleva
Publikováno v:
Bulletin of Experimental Biology and Medicine. 174:571-577
Publikováno v:
Biology Bulletin. 49:2314-2321
Publikováno v:
Biology Bulletin. 49:2467-2474
Publikováno v:
Biology Bulletin. 48:2060-2070
Autor:
E. S. Zhorova, E. Yu. Moskaleva, Yu. P. Semochkina, V. P. Saprykin, A. V. Rodina, V G Shuvatova
Publikováno v:
Bulletin of Experimental Biology and Medicine. 171:141-149
Mesenchymal stem cells from the adipose tissue (AT MSC) and the bone marrow (BM MSC) stimulated migration of melanoma B16 cells, while mammary adenocarcinoma Ca755 cells stimulated migration of mesenchymal stem cells. Mesenchymal stem cells retained
Autor:
I K Malashenkova, S A Krynskiy, E. Yu. Moskaleva, Gavrilova Si, Yu. P. Semochkina, D P Ogurtsov, N A Hailov, E. V. Ponomareva
Publikováno v:
Biochemistry (Moscow), Supplement Series B: Biomedical Chemistry. 15:139-146
The individual differences in the efficiency of DNA DSB repair were estimated by the level of residual γH2AX foci after γ-irradiation at a dose of 2 Gy, in lymphocytes of patients with amnestic mild cognitive impairment (AMCI) and Alzheimer's disea
Autor:
A. V. Rodina, E. Yu. Moskaleva, Yu. P. Semochkina, E. S. Zhorova, V. P. Saprykin, Russia Moscow, V G Shuvatova
Publikováno v:
Cell Technologies in Biology and Medicine. :38-46
Publikováno v:
Biochemistry (Moscow), Supplement Series B: Biomedical Chemistry. 15:80-88
Publikováno v:
Biomeditsinskaya Khimiya. 66:265-273
In proliferating normal and tumor cells, the telomere length (TL) is maintained by high telomerase activity (TA). In the absence of TA the TL maintenance involves a mechanism of alternative lengthening of telomeres (ALT). The aim of this study was to