Výsledky vyhledávání

Isolation and characterization of a novel, transforming allele of the c-Cbl proto-oncogene from a murine macrophage cell line

Autor: Eva E. Ujack, Sabine A. Bisson, Stephen M. Robbins

Publikováno v: Oncogene. 21:3677-3687

The c-Cbl proto-oncogene acts as an E3 ubiquitin ligase via its RING finger domain to negatively regulate activated cellular signal transduction pathways. We have identified an aberrant Cbl-protein of approximately 95 kDa, which we have called p95Cbl

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4de65ca34cb6ae9d2f250497d29493a1
https://doi.org/10.1038/sj.onc.1205510

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Pseudomonas aeruginosa lasR Transcription Correlates with the Transcription of lasA , lasB , and toxA in Chronic Lung Infections Associated with Cystic Fibrosis

Autor: Eva E. Ujack, Ian Mitchell, Harvey R. Rabin, Douglas G. Storey

Publikováno v: Infection and Immunity. 66:2521-2528

The role of Pseudomonas aeruginosa quorum-sensing systems in the lung infections associated with cystic fibrosis (CF) has not been examined. The purpose of this study was to determine if genes regulated by the LasR-LasI quorum-sensing system were coo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::1c5b99f67f2751585ad68f66c9cfdbbd
https://doi.org/10.1128/iai.66.6.2521-2528.1998

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Positive correlation of algD transcription to lasB and lasA transcription by populations of Pseudomonas aeruginosa in the lungs of patients with cystic fibrosis

Autor: Eva E. Ujack, Douglas G. Storey, Harvey R. Rabin, Ian Mitchell

Publikováno v: Infection and Immunity. 65:4061-4067

Pseudomonas aeruginosa causes a chronic infection in the lungs of individuals with cystic fibrosis. The P. aeruginosa isolates from these infections, when grown under laboratory conditions, characteristically are mucoid and produce low levels of the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db66842252f121e7e7730a309654bab2
https://doi.org/10.1128/iai.65.10.4061-4067.1997

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STEM CELLS

Autor: L. Cheng, Z. Huang, W. Zhou, Q. Wu, J. Rich, S. Bao, P. Baxter, H. Mao, X. Zhao, Z. Liu, Y. Huang, H. Voicu, S. Gurusiddappa, J. M. Su, L. Perlaky, R. Dauser, H.-c. E. Leung, K. M. Muraszko, J. A. Heth, X. Fan, C. C. Lau, T.-K. Man, M. Chintagumpala, X.-N. Li, P. Clark, M. Zorniak, Y. Cho, X. Zhang, D. Walden, E. Shusta, J. Kuo, S. Sengupta, S. Goel-Bhattacharya, S. Kulkarni, B. Cochran, C. Cusulin, A. Luchman, S. Weiss, M. Wu, N. Fernandez, S. Agnihotri, R. Diaz, J. Rutka, M. Bredel, J. Karamchandani, S. Das, B. Day, B. Stringer, F. Al-Ejeh, M. Ting, J. Wilson, K. Ensbey, P. Jamieson, Z. Bruce, Y. C. Lim, C. Offenhauser, S. Charmsaz, L. Cooper, J. Ellacott, A. Harding, J. Lickliter, P. Inglis, B. Reynolds, D. Walker, M. Lackmann, A. Boyd, A. Berezovsky, L. Poisson, L. Hasselbach, S. Irtenkauf, A. Transou, T. Mikkelsen, A. C. deCarvalho, D. Emlet, C. Del Vecchio, P. Gupta, G. Li, S. Skirboll, A. Wong, J. Figueroa, T. Shahar, A. Hossain, F. Lang, S. Fouse, J. Nakamura, C. D. James, S. Chang, J. Costello, J. M. Frerich, S. Rahimpour, Z. Zhuang, J. D. Heiss, A. Golebiewska, D. Stieber, L. Evers, E. Lenkiewicz, N. H. C. Brons, N. Nicot, A. Oudin, S. Bougnaud, F. Hertel, R. Bjerkvig, M. Barrett, L. Vallar, S. P. Niclou, X. Hao, J. Rahn, E. Ujack, X. Lun, G. Cairncross, D. Senger, S. Robbins, J. Harness, R. Lerner, Y. Ihara, R. Santos, J. D. L. Torre, A. Lu, T. Ozawa, T. Nicolaides, D. James, C. Petritsch, D. Higgins, M. Schroeder, B. Ball, B. Milligan, F. Meyer, J. Sarkaria, J. Henley, W. Flavahan, M. Hitomi, N. Rahim, Y. Kim, A. Sloan, R. Weil, I. Nakano, M. Li, J. Lathia, A. Hjelmeland, M. Kaluzova, S. Platt, M. Kent, A. Bouras, R. Machaidze, C. Hadjipanayis, S.-G. Kang, S.-H. Kim, Y.-M. Huh, E.-H. Kim, E.-K. Park, J. H. Chang, S. H. Kim, Y. K. Hong, D. S. Kim, S.-J. Lee, E. H. Kim, S. G. Kang, L. Deleyrolle, M. Sinyuk, W. Goan, B. Otvos, M. Rohaus, M. Oli, V. Vedam-Mai, D. Schonberg, S.-T. Lee, K. Chu, S. K. Lee, M. Kim, J.-K. Roh, A. Griveau, B. Reichholf, M. McMahon, D. Rowitch, R. Nitta, S. Mitra, M. Agarwal, T. Bui, J. Lin, C. Adamson, J. Martinez-Quintanilla, S.-H. Choi, D. Bhere, P. Heidari, D. He, U. Mahmood, K. Shah, S. Gholamin, A. Feroze, A. Achrol, S. Kahn, I. Weissman, S. Cheshier, E. P. Sulman, Q. Wang, E. Mostovenko, H. Liu, C. F. Lichti, A. Shavkunov, R. A. Kroes, J. R. Moskal, C. A. Conrad, F. F. Lang, M. R. Emmett, C. L. Nilsson, S. Osuka, O. Sampetrean, T. Shimizu, I. Saga, N. Onishi, E. Sugihara, J. Okubo, S. Fujita, S. Takano, A. Matsumura, H. Saya, N. Saito, J. Fu, S. Wang, W. K. A. Yung, D. Koul, R. S. Schmid, D. M. Irvin, M. Vitucci, R. E. Bash, A. M. Werneke, C. R. Miller, N. Shinojima, T. Takezaki, J. Fueyo, J. Gumin, F. Gao, F. Nwajei, F. C. Marini, M. Andreeff, J.-I. Kuratsu, S. Singh, K. Burrell, E. Koch, S. Jalali, A. Vartanian, E. Sulman, B. Wouters, G. Zadeh, R. Spelat, E. Singer, L. Matlaf, S. McAllister, L. Soroceanu, S. Spiegl-Kreinecker, D. Loetsch, M. Laaber, C. Schrangl, A. Wohrer, J. Hainfellner, C. Marosi, J. Pichler, S. Weis, G. Wurm, G. Widhalm, E. Knosp, W. Berger, J.-i. Kuratsu, Q. Tam, S. Tanaka, M. Nakada, D. Yamada, T. Todo, Y. Hayashi, J.-i. Hamada, A. Hirao, J. Tilghman, M. Ying, J. Laterra, M. Venere, C. Chang, M. Summers, S. Rosenfeld, S. Luk, J. Iafrate, D. Cahill, R. Martuza, S. Rabkin, A. Chi, H. Wakimoto, H.-G. Wirsching, S. Krishnan, K. Frei, N. Krayenbuhl, G. Reifenberger, M. Weller, G. Tabatabai, J. Man, J. Shoemake, J. Yu

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f94f7a03a17079a76eb686dc4535928
https://europepmc.org/articles/PMC3222962/

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Analysis of multiple growth regulatory proteins using dissociable staining antibody arrays on solid tumor biopsy specimens

Autor: Eva E. Ujack, Aru Narendran, Christopher Blackmore, Aarthi Jayanthan, Tony Magliocco

Publikováno v: Fetal and pediatric pathology. 30(3)

Growth of tumor cells is often a function of deregulated growth factor receptors and their corresponding intracellular signalling molecules. The dissociable antibody staining arrays have the versatility to rapidly identify the expression, activation,

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26271a5a74ec9ef4cc10b9d54471a610
https://pubmed.ncbi.nlm.nih.gov/21355677

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Decreased expression of the INK4 family of cyclin-dependent kinase inhibitors in Wilms tumor

Autor: M Y, Arcellana-Panlilio, R M, Egeler, E, Ujack, A, Pinto, D J, Demetrick, S M, Robbins, M J, Coppes

Publikováno v: Genes, chromosomescancer. 29(1)

Cyclin-dependent kinase (CDK) inhibitors represented by the INK4 family (including p16(INK4a, CDKN2A), p15(INK4b, CDKN2B), p18(INK4c, CDKN2C), and p19(INK4d, CDKN2D)) are regulators of the cell cycle shown to be aberrant in many types of human cancer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::38d053ef4adc237dde03edf907dd9ef6
https://pubmed.ncbi.nlm.nih.gov/10918395

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Mesna inactivates platinum agents in vitro

Autor: J E, Wolff, R M, Egeler, R, Anderson, E, Ujack, S, Iceton, M J, Coppes

Publikováno v: Anticancer research. 18(6A)

Platinum agents are frequently combined with ifosfamide. Mesna, originally coadministered to protect from ifosfamide side effects, might also react with the platinum agents in these combinations.Malignant glioma cells were incubated with cisplatin, c

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::cf76982386f8b48acaf4a8191d5bf05f
https://pubmed.ncbi.nlm.nih.gov/9891448

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