Contractile Dysfunction Irrespective of the Mutant Protein in Human Hypertrophic Cardiomyopathy With Normal Systolic Function

Autor: Folkert J. ten Cate, Jolanda van der Velden, Lucie Carrier, Marjon van Slegtenhorst, Cris dos Remedios, Dennis Dooijes, Sakthivel Sadayappan, Michelle Michels, E. Rosalie Paalberends, Aref Najafi, Nicky M. Boontje, Ger J.M. Stienen, Diederik W. D. Kuster, Sabine J. van Dijk

Publikováno v: Circulation: Heart Failure
Circulation-Heart Failure, 5(1), 36-U126. Lippincott Williams & Wilkins
Circulation. Heart failure, 5(1), 36-U126. Lippincott Williams and Wilkins
van Dijk, S J, Paalberends, E R, Najafi, A, Michels, M, Sadayappan, S, Carrier, L, Boontje, N M, Kuster, D W D, van Slegtenhorst, M, Dooijes, D, dos Remedios, C, ten Cate, F J, Stienen, G J M & van der Velden, J 2012, ' Contractile Dysfunction Irrespective of the Mutant Protein in Human Hypertrophic Cardiomyopathy With Normal Systolic Function ', Circulation. Heart failure, vol. 5, no. 1, pp. 36-U126 . https://doi.org/10.1161/CIRCHEARTFAILURE.111.963702
Circulation: Heart Failure; Vol 5

Background— Hypertrophic cardiomyopathy (HCM), typically characterized by asymmetrical left ventricular hypertrophy, frequently is caused by mutations in sarcomeric proteins. We studied if changes in sarcomeric properties in HCM depend on the under

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9a84e6e3f64373a1080cbfa3122d15e
https://doi.org/10.1161/CIRCHEARTFAILURE.111.963702