Zobrazeno 1 - 10
of 186
pro vyhledávání: '"E. Mirkes"'
Autor:
A.J. Bell, S. Ram, W.W. Labaki, S. Murray, E. Kazerooni, S. Galban, F.J. Martinez, C. Hatt, J.M. Wang, E. Mirkes, A. Zinovyev, A. Gorban, M.K. Han, C.J. Galban
Publikováno v:
D97. IMPACT OF COPD ACROSS THE LIFESPAN.
Akademický článek
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Publikováno v:
Birth Defects Research Part B: Developmental and Reproductive Toxicology. 86:40-47
BACKGROUND: Hyperthermia (HS) is a well-studied teratogen that induces serious malformations, including neural tube defects. Our previous studies have shown that HS induces apoptosis by activating the mitochondrial apoptotic pathway. Prior to activat
Akademický článek
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Autor:
Philip E. Mirkes
Publikováno v:
Congenital Anomalies. 48:7-17
Research over the past 50 years has consistently documented that cell death is an integral part of both normal development and the etiology of birth defects; however, the significance of this cell death has been, until recently, unclear. Research pub
Publikováno v:
Toxicological Sciences. 95:257-269
Hyperthermia (HS) and 4-hydroperoxycyclophosphamide (4CP) activate the mitochondrial apoptotic pathway in day 9 mouse embryos. Previous microarray analyses Microarray analyses revealed that several p53 target genes are upregulated after exposure to H
Autor:
Philip E. Mirkes, Marianne Barrier
Publikováno v:
Reproductive Toxicology. 19:291-304
The objective of this presentation is to review the major proteomic technologies available to developmental toxicologists and, when possible, to provide examples of how various proteomic technologies have been used in developmental toxicology or toxi
Publikováno v:
Birth Defects Research Part A: Clinical and Molecular Teratology. 67:98-107
BACKGROUND Using vital dyes, we have previously shown that while hyperthermia (HS), 4-hydroperoxycyclophosphamide (4CP), and staurosporine (ST) induce cell death within specific tissues (e.g., neuroepithelium) of day 9 mouse embryos, cells of the hea
Publikováno v:
Birth Defects Research Part A: Clinical and Molecular Teratology. 67:21-34
Keywords: developmental toxicology; developmental biology; signal transduction pathways; genomics; proteomics; model organisms