Zobrazeno 1 - 10
of 42
pro vyhledávání: '"E. Imesch"'
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1270(1):95-99
Genetically hypertensive rats are excellent animal models for investigating putative Na+/K(+)-ATPase alterations associated with the disease. Highly purified Na+/K(+)-ATPase preparations from these animals have not yet been examined. Na+/K(+)-ATPases
Publikováno v:
Molecular Membrane Biology. 11:237-245
The Na,K-ATPase (NKA) system is the receptor for the cardioactive steroids of plant or animal origin. It is not yet known whether passive ion fluxes traverse the inactivated receptor and thereby contribute to the hormonal, pharmacological or toxic ac
Publikováno v:
Biochemical and Biophysical Research Communications. 167:1115-1121
The SH-reactive HgCl2 inhibits the Na,K-ATPase activity potently in a manner antagonized only partially by EDTA or cysteine; solely dimercaprol, a dithiol antidote for mercury, blocks the HgCl2 effects entirely as confirmed also by 203Hg-binding expe
Publikováno v:
The American journal of physiology. 262(5 Pt 2)
An inhibitory receptor for cardioactive steroids such as digoxin and ouabain is located at the extracellular surface of the Na-K-adenosinetriphosphatase (ATPase) molecule. Besides cardioactive steroids, mercury is a potent inhibitor of the Na-K-ATPas
Publikováno v:
American Journal of Physiology-Renal Physiology. 263:F984-F984
The presence of circulating inhibitors able to decrease the renal Na-K-adenosinetriphosphatase (ATPase) activity (natriuretic hormones) was postulated some 30 years ago. In the present work, the natriuretic inhibitor HgCl2 was selected as a model com
Autor:
A. Chinet, F.Assimacopoulos Jeannet, Lucien Girardier, S. Bas, Josiane Seydoux, E. Imesch, G. Schneider-Picard, Jean-Paul Giacobino
Publikováno v:
Endocrinology. 113:604-610
Defects of both diet-induced thermogenesis and cold tolerance have been reported for streptozotocin-diabetic rats. Since brown adipose tissue (BAT) is a major effector of both diet- and cold-induced thermogenesis in the rat, the possible cause of the
Publikováno v:
Experientia. 32(3)
In perfused livers of 24 hour-fasted rats, PGE1 (prostaglandin E1) infused continuously into the perfusate, was found to cause a 45% increase in the incorporation of 1-14C acetate into liver fatty acids. PGE1 was found to have no effect, however, on
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