Zobrazeno 1 - 8
of 8
pro vyhledávání: '"E. I. Talalaeva"'
Publikováno v:
Biophysics. 55:760-764
Experimental evidence has been obtained that mutations in the presenilin 1 (PS1) gene in familial Alzheimer’s disease can lead to the disturbance of cell adhesion in model cell cultures. It was shown that, in L fibroblasts of mice with stable expre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::96c0308a8ecb3d796cef21188e26a430
https://doi.org/10.1134/s0006350910050131
https://doi.org/10.1134/s0006350910050131
Autor:
O. L. Runova, E. I. Talalaeva, S. V. Sarantseva, A. L. Shwartsman, K. V. Solovyov, Michael P. Vitek
Publikováno v:
Biophysics. 53:550-554
A comparative study of growth cone morphology in cultured embryonic neurons derived from wild type PS 1(+/+) and knockout PS 1(-/-) mice has been performed. Growth cones from wild type PS 1(+/+) mice were well spread and usually formed radially conti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::abda6192fe5fbe0f42441a5b7bf671ff
https://doi.org/10.1134/s0006350908060158
https://doi.org/10.1134/s0006350908060158
Autor:
S. V. Sarantseva, Michael P. Vitek, O. L. Runova, Yu. A. Tatishcheva, E. I. Talalaeva, A. L. Schwartsman
Publikováno v:
Biophysics. 51:740-743
Most cases of early-onset familial Alzheimer’s disease are caused by mutations in the presenilin 1 gene. Nonetheless, the function of presenilin 1 are not yet completely understood. It was shown that endogenous presenilin 1, as well as the adhesion
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3608b8399fa461873c1e9cfc5e41447c
https://doi.org/10.1134/s0006350906050125
https://doi.org/10.1134/s0006350906050125
Publikováno v:
Biofizika. 55(5)
Experimental evidence has been obtained that mutations in the presenilin 1 (PS1) gene in familial Alzheimer's disease can lead to the disturbance of cell adhesion in model cell cultures. It was shown that, in L fibroblasts of mice with stable express
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::fae26973a97a638539ec6743c990f896
https://pubmed.ncbi.nlm.nih.gov/21033353
https://pubmed.ncbi.nlm.nih.gov/21033353
Publikováno v:
Biofizika. 53(6)
A comparative study of growth cone morphology in cultured embryonic neurons derived from wild type PS 1(+/+) and knockout PS 1(-/-) mice has been performed. Growth cones from wild type PS 1(+/+) mice were well spread and usually formed radially conti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::3bcce909fddb23f1ae02d879b10756c3
https://pubmed.ncbi.nlm.nih.gov/19137685
https://pubmed.ncbi.nlm.nih.gov/19137685
Publikováno v:
Biofizika. 51(5)
Most cases of familial early-onset Alzheimer's disease are caused by mutations in the presenilin 1 (PS1) gene. However, the cellular functions of PS1 are not yet completely understood. We showed that endogenous PS1 and the adhesion protein CD44 are r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::75255c3008ed9e05429fb17539442f54
https://pubmed.ncbi.nlm.nih.gov/17131822
https://pubmed.ncbi.nlm.nih.gov/17131822
Autor:
N A, Skobeleva, V I, Vasina, M V, Volkova, A M, Sverdlova, E V, Fomicheva, G I, Obraztsova, E I, Talalaeva, Kh, Shakir, M, Laasri, I M, Vorontsov, Iu P, Kovalev, E I, Shvarts
Publikováno v:
Molekuliarnaia genetika, mikrobiologiia i virusologiia. (4)
Polymorphisms of 3 apolipoprotein genes Xba I apoB, Sstl apoCIII, and apoE and the insertion-deletion polymorphism of the angiotensin-converting enzyme gene (I/D ACE) and lipid levels were studied in a random sample of 403 children and adolescents ag
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d1d65dbc8013d43f8236b0e40060aea0
https://pubmed.ncbi.nlm.nih.gov/9411220
https://pubmed.ncbi.nlm.nih.gov/9411220
Publikováno v:
Gigiena i sanitariia. (7)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::69db6879fc3b61e0065a89b6e026eaa5
https://pubmed.ncbi.nlm.nih.gov/2227522
https://pubmed.ncbi.nlm.nih.gov/2227522