Zobrazeno 1 - 5 of 5 pro vyhledávání: '"E. H. Olesen"'
1
The requirement of localized, CR2-mediated, alternative pathway activation of complement for covalent deposition of C3 fragments on normal B cells
Autor: A. A. Johnson, R. G. Q. Leslie, E. H. Olesen, G. Damgaard
Publikováno v: Immunology. 93:177-183
We have shown previously that normal B cells share, with Epstein-Barr virus-transformed and malignant B cells, the ability to activate the alternative pathway (AP) of complement in vitro, resulting in the deposition of C3 fragments on the cell surfac
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::438fca3456cf7268b2ff1428d3f03c80
https://doi.org/10.1046/j.1365-2567.1998.00429.x
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2
The requirement of localized, CR2-mediated, alternative pathway activation of complement for covalent deposition of C3 fragments on normal B cells
Autor: E H, Olesen, A A, Johnson, G, Damgaard, R G, Leslie
Publikováno v: Immunology. 93(2)
We have shown previously that normal B cells share, with Epstein-Barr virus-transformed and malignant B cells, the ability to activate the alternative pathway (AP) of complement in vitro, resulting in the deposition of C3 fragments on the cell surfac
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::665db1411218e39681870cf06c1392e6
https://pubmed.ncbi.nlm.nih.gov/9616366
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3
A comparative study of normal B cells and the EBV-positive Burkitt's lymphoma cell line, Raji, as activators of the complement system
Autor: R. G. Q. Leslie, H. V. Marquart, E. H. Olesen, G. Damgaard, A. A. Johnson
Publikováno v: Scandinavian journal of immunology. 46(3)
Contradictory reports regarding the ability of complement receptor type 2 (CR2,CD21) on normal B cells to activate complement (C') via the alternative pathway (AP), prompted us to compare the performance of human peripheral blood B cells and the Epst
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42702de9863abeb582f7433ee11cd259
https://pubmed.ncbi.nlm.nih.gov/9315112
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