Výsledky vyhledávání

Potential Association Between Calcified Thoracic Lymphadenopathy Due to Previous Histoplasma capsulatum Infection and Pulmonary Mycobacterium avium Complex Disease

Autor: E D Chan, M D Iseman

Publikováno v: Southern Medical Journal. 92:572-576

Background Among patients with pulmonary disease due to Mycobacterium avium complex (MAC) seen recently at our center, a substantial number have had extensive calcified mediastinal, hilar, and peribronchial lymphadenopathy, a finding historically inc

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29c82c2b6a34ba9f1daf3f978bb3e757
https://doi.org/10.1097/00007611-199906000-00005

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Activation of p38mapk, MKK3, and MKK4 by TNF-alpha in mouse bone marrow-derived macrophages

Autor: B W Winston, E D Chan, G L Johnson, D W Riches

Publikováno v: The Journal of Immunology. 159:4491-4497

TNF-alpha regulates the expression of many proinflammatory and profibrogenic gene products in macrophages, and hence plays a vital role in controlling the inflammatory response. We have shown previously that exposure of macrophages to TNF-alpha stimu

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::dd49a925bdf184d3de3b41aa93c0cf1d
https://doi.org/10.4049/jimmunol.159.9.4491

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Fc gamma receptor cross-linking activates p42, p38, and JNK/SAPK mitogen-activated protein kinases in murine macrophages: role for p42MAPK in Fc gamma receptor-stimulated TNF-alpha synthesis

Autor: D M Rose, B W Winston, E D Chan, D W Riches, P Gerwins, G L Johnson, P M Henson

Publikováno v: The Journal of Immunology. 158:3433-3438

Fc gamma R cross-linking on murine macrophages resulted in the activation of mitogen-activated protein kinase (MAPK) family members p42MAPK, p38, and c-Jun NH2-terminal kinase (JNK)/stress-activated protein kinase (SAPK). The temporal pattern of acti

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::027b13dbd9ad85b1a98c85c8bbcf5e76
https://doi.org/10.4049/jimmunol.158.7.3433

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Does Reduced 1-Antitrypsin Activity Explain the Link Between Cigarette Smoking and Idiopathic Pulmonary Fibrosis?

Autor: E D, Chan, A H, Ralston, L, Shapiro

Publikováno v: Chest. 120:72S-74

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a5fd73ba1e98ec65e213fa3d77ce89a8
https://doi.org/10.1378/chest.120.1_suppl.s72-a

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Mycoplasma pneumoniae-associated bronchiolitis causing severe restrictive lung disease in adults: report of three cases and literature review

Autor: E D, Chan, T, Kalayanamit, D A, Lynch, R, Tuder, P, Arndt, R, Winn, M I, Schwarz

Publikováno v: Chest

Study objectives To characterize adult Mycoplasma pneumoniae-induced bronchiolitis requiring hospitalization. Design We encountered an adult patient with severe bronchiolitis in the absence of pneumonia due to M pneumoniae. To determine the relative

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::a3f49ab6e7d5baa080e9b90ba993f29c
https://pubmed.ncbi.nlm.nih.gov/10208228

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Evaluation of the role of mitogen-activated protein kinases in the expression of inducible nitric oxide synthase by IFN-gamma and TNF-alpha in mouse macrophages

Autor: E D, Chan, B W, Winston, S T, Uh, M W, Wynes, D M, Rose, D W, Riches

Publikováno v: Journal of immunology (Baltimore, Md. : 1950). 162(1)

The expression of inducible nitric oxide synthase (iNOS) by macrophages is stimulated by coexposure to IFN-gamma and a number of stimuli, including TNF-alpha. Recent work has shown that TNF-alpha activates members of the mitogen-activated protein kin

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::4ab3a22cc4903e8a031721f85c7805b8
https://pubmed.ncbi.nlm.nih.gov/9886415

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Morphometric analysis of insulin-like growth factor-I localization in lung tissues of patients with idiopathic pulmonary fibrosis

Autor: E. D. Chan, S.-T. Uh, Y. Inoue, L. S. Newman, D. W. H. Riches, Talmadge E. King

Publikováno v: American journal of respiratory and critical care medicine. 158(5 Pt 1)

Insulin-like growth factor-I (IGF-I) has been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF) through its ability to stimulate fibroblast proliferation and collagen synthesis. However, although alveolar macrophages (AM) have bee

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e928306ce4aa6ea32a28ed7cd2460fb4
https://pubmed.ncbi.nlm.nih.gov/9817718

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