Zobrazeno 1 - 9 of 9 pro vyhledávání: '"E V, Zakliaz'minskaia"'
1
[Clinical polymorphisms and approaches of arrhythmias treatment in a family with δKPQ1505-1507 deletion in SCN5A gene]
Autor: A. V. Gavrilenko, Massoud Houshmand, M Eftekhharzadeh, Nasab Samiei, Amirfarjam Fazelifar, E V Zakliaz'minskaia, Siamak Saber, Majid Haghjoo
Publikováno v: Vestnik Rossiiskoi akademii meditsinskikh nauk. (5-6)
Background : The aim of the study was to analyze spectrum of manifestation and treatment response in large family with rhythm disturbances caused by p.delKPQ1505-1507 mutation in SCN5A gene. Patients and methods : We had under our observation 18 memb
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efd87573c2bef685518a05f3850c075e
https://pubmed.ncbi.nlm.nih.gov/25558681
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2
[Possibilities myocardial biopsy in the diagnosis of myocarditis verification in patients with idiopathic arrhythmias]
Autor: O V, Blagova, A V, Nedostup, E A, Kogan, V A, Sulimov, S A, Abugov, A G, Kupriianov, V A, Zaĭdenov, A E, Donnikov, E V, Zakliaz'minskaia
Publikováno v: Kardiologiia. 53(11)
Aim of the study was to elucidate nosological nature of "idiopathic" arrhythmias by means of right ventricular endomyocardial biopsy (EMB) and to assess effect of etiotropic and pathogenetic treatment.We included into this study 19 patients (mean age
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::6cd9041eee46b08f6f02939596ccca97
https://pubmed.ncbi.nlm.nih.gov/24654431
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3
[Clinic and genetic polymorphism of Brugada syndrome in Russian patients, caused by mutation in SCN5A gene]
Autor: E V, Zakliaz'minskaia, A G, Shestak, A Sh, Revishvili, I V, Pronicheva, D G, Podoliak, M A, Nechaenko, A V, Poliakov, S L, Dzemeshkevich
Publikováno v: Khirurgiia. (2)
Brugada syndrome (BrS) is an inherited cardiac arrhythmic disorder, characterized by ST-segment elevation in right precordial leads V1-V22 mm, pseudo right bundle branch block (RBBB), T-wave inversion and an increased risk of cardiac sudden death (SC
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::9a4f7374889444093e985c8ddc51d2ae
https://pubmed.ncbi.nlm.nih.gov/23503384
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5
[Arrhythmogenic right ventricular dysplasia: polymorphism of clinical manifestations]
Autor: O V, Blagova, A V, Nedostup, N S, Morozova, E A, Kogan, N V, Gagarina, V P, Sedov, E V, Zakliaz'minskaia, Iu V, Frolova, S L, Dzemeshkevich, S A, Aleksandrova
Publikováno v: Kardiologiia. 52(4)
We observed 15 patients with arrhythmogenic right ventricular dysplasia (ARVD): 9 with definite and 5 with probable ARVD (modified European Criteria, 2010). Eight patients had typical ARVD (frequent right ventricular extrasystoles, nonsustained right
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::cbc838e969ae8c4a7ee33e200f52c847
https://pubmed.ncbi.nlm.nih.gov/22839522
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6
[Dilated cardiomyopathy as clinical syndrome: experience with nosological diagnostics with biopsy and treatment approaches]
Autor: O V, Blagova, A V, Nedostup, E A, Kogan, S L, Dzemeshkevich, Iu V, Frolova, V P, Sedov, N V, Gagarina, V A, Sulimov, S A, Abugov, E V, Zakliaz'minskaia, A E, Donnikov, V V, Kadochnikova, A G, Kupriianova, V A, Zaĭdenov, L V, Beletskaia
Publikováno v: Terapevticheskii arkhiv. 83(9)
To study possibility of nosological diagnosis in patients with dilated cardiomyopathy (DCMP) with use of myocardial biopsy.The trial enrolled 62 patients (23 females) with DCMP syndrome (end diastolic left ventricular size5.5 cm, ejection fraction55%
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::22a2fd54a8020b0b80c2b2aa92554dc5
https://pubmed.ncbi.nlm.nih.gov/22145387
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7
[The clinical variability of and approaches to treatment of life-threatening ventricular arrhythmias caused by SCN5A gene mutations]
Autor: L A, Bokeria, A Sh, Revishvili, I V, Pronicheva, E V, Zakliaz'minskaia, A V, Poliakov
Publikováno v: Vestnik Rossiiskoi akademii meditsinskikh nauk. (5)
The reasons for ventricular arrhythmias are variable enough. In many cases the presence and degree of ventricular arrhythmias cannot be explained satisfactorily by the presence of ischemic of inflammatory myocardial lesion. Now that the development o
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::875aae420b11520f8359e3e795abfe83
https://pubmed.ncbi.nlm.nih.gov/17605181
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8
[Clinical, genealogical and molecular genetic study of Emery-Dreifuss muscular dystrophy]
Autor: G E, Rudenskaia, S M, Tverskaia, A L, Chukhrova, E V, Zakliaz'minskaia, Iu V, Kuropatkina, E L, Dadali, V S, Perminov, A V, Poliakov
Publikováno v: Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova. 106(10)
A search for emerin and lamin A/C (LMNA) mutations was performed in a group of 63 unrelated patients with probable Emery-Dreifuss muscular dystrophy (EDMD) and other MD's with concomitant dilated cardiomyopathy (DCMP). Four different emerin mutations
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::26b09cf5f30d255aee855f39e415d720
https://pubmed.ncbi.nlm.nih.gov/17117676
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