Zobrazeno 1 - 10
of 92
pro vyhledávání: '"E N, Moore"'
Autor:
Juma S. Mbwana, S. Shamim, Jessica Mayo, L. G. Vezina, Steven L. Weinstein, Phillip L. Pearl, S. Sato, W. H. Theodore, William D. Gaillard, E. N. Moore, Lisa R. Rosenberger, Joan A. Conry, Madison M. Berl, E. K. Ritzl
Publikováno v:
Brain. 132:347-356
Neural networks for processing language often are reorganized in patients with epilepsy. However, the extent and location of within and between hemisphere re-organization are not established. We studied 45 patients, all with a left hemisphere seizure
Autor:
E N, MOORE
Publikováno v:
A listing of research in the cardiovascular field. 10
Publikováno v:
Journal of Cardiovascular Electrophysiology. 8:199-214
Cellular Uncoupling and Procainamide's Effect. Introduction: How cell-to-cell electrical coupling influences an antiarrhythmic agent's effect on conduction is largely unknown. To investigate this, we evaluated the effects of procainamide on myocardia
Publikováno v:
Journal of Cardiovascular Electrophysiology. 4:320-337
Electrocardiographic manifestation of "supernormal" conduction is defined as conduction that is more rapid than expected or presence of conduction when block is anticipated. It is not supernormal in the sense or being more rapid than normal. Therefor
Autor:
E N, MOORE
Publikováno v:
Journal of the American Veterinary Medical Association. 108
Autor:
E N, MOORE
Publikováno v:
The Cornell veterinarian. 37(2)
Autor:
E N, MOORE
Publikováno v:
Journal of the American Veterinary Medical Association. 111(845)
Autor:
E N, MOORE
Publikováno v:
The Cornell veterinarian. 37(1)
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 263:H1382-H1389
Abnormalities in cellular coupling, modulated in part by intracellular gap junctions, have an important role in the genesis of reentrant arrhythmias in the setting of chronic myocardial infarction. The effects of heptanol, which has a relatively sele
Gap junctional conductance in ventricular myocyte pairs isolated from postischemic rabbit myocardium
Publikováno v:
Circulation Research. 71:127-136
Abnormalities of myocardial gap junction-mediated cell coupling have been implicated in cardiac arrhythmogenesis. The potential role of gap junctional dysfunction in the generation of reperfusion-induced arrhythmias is uncertain. The purpose of this