Zobrazeno 1 - 8
of 8
pro vyhledávání: '"E C, Castelli"'
Autor:
A S, Moreno, L S M, Maia, P B, Palhas, M M, Dias, V F, Muglia, E C, Castelli, K, Bork, L K, Arruda
Publikováno v:
Journal of investigational allergologyclinical immunology. 26(1)
Publikováno v:
HLA. 87(2)
Human leukocyte antigen G (HLA-G) is an immunomodulatory molecule with important roles both physiologically as well as an escape mechanism of cancer cells. In this study, we evaluated the impact of eight polymorphisms at the 3' untranslated region (3
Autor:
L P, Felício, I O P, Porto, C T, Mendes-Junior, L C, Veiga-Castelli, K E, Santos, R P, Vianello-Brondani, A, Sabbagh, P, Moreau, E A, Donadi, E C, Castelli
Publikováno v:
Tissue antigens. 83(2)
The human leukocyte antigen-E (HLA-E) locus is a human major histocompatibility complex (MHC) gene associated with immune-modulation and suppression of the immune response by the interaction with specific natural killer (NK) and T cell receptors (TCR
Publikováno v:
Tissue antigens. 82(3)
We report a novel nonclassical class I HLA-E*01:06 allele observed in Brazilian individuals.
Autor:
L C, Veiga-Castelli, A S, de Paula Cruz, M M, Inácio, C T, Mendes-Junior, R, Vianello-Brondani, P, Moreau, E C, Castelli, E A, Donadi
Publikováno v:
Tissue antigens. 82(3)
Autor:
A, Sabbagh, D, Courtin, J, Milet, J D, Massaro, E C, Castelli, F, Migot-Nabias, B, Favier, N, Rouas-Freiss, P, Moreau, A, Garcia, E A, Donadi
Publikováno v:
Tissue antigens. 82(1)
Host and Plasmodium interactions result in highly variable clinical phenotypes, partly explained by the nature and level of anti-malarial antibody response. Human leukocyte antigen (HLA)-G can create a tolerogenic environment, allowing parasites to e
Autor:
M M, Silva, R T, Simões, E C, Castelli, C T, Mendes-Junior, N H S, Deghaide, L C, Tsuda, A A, Machado, E A, Donadi, A P M, Fernandes
Publikováno v:
International journal of immunogenetics. 37(5)
The aim of this study was to evaluate the frequency of TNFa-e microsatellites and the promoter region (TNF-308 and TNF-238) in HIV/AIDS-infected patients presenting or not lipodystrophy syndrome (LS). The design is the genetic case-control associatio
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