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pro vyhledávání: '"E, Helset"'
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Publikováno v:
Mediators of Inflammation, Vol 3, Iss 2, Pp 155-160 (1994)
In the present study we examined whether endothelin-1 stimulation of human monocytes causes release of chemotactic factors. It was found that monocytes released neutrophil- and monocyte-chemotactic activity in a dose- and time-dependent manner in res
Externí odkaz:
https://doaj.org/article/c4d753b39e6f45dfa49e15e75f8ca727
Publikováno v:
Mediators of Inflammation, Vol 2, Iss 6, Pp 417-422 (1993)
Increased plasma- and tissue levels of endothelin-1 (ET-1) during inflammatory diseases, have suggested a role of ET-1 in the pathophysiology of inflammatory reactions. The authors have studied the effect of ET-1 on cytokine release from monocytes an
Externí odkaz:
https://doaj.org/article/37095e7d732e43658db17d8e7756969c
Publikováno v:
Scandinavian Journal of Clinical and Laboratory Investigation. 55:369-376
The release and vascular effects of calcitonin gene-related peptide (CGRP) and endothelin-1 (ET-1) during acute alveolar hypoxia (O2 2%) were examined in isolated blood-perfused rat lungs. In 10 lungs, repeatedly ventilated with hypoxic gas for 5 min
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::523286d2557af69fac9e16c64627514d
https://doi.org/10.3109/00365519509104975
https://doi.org/10.3109/00365519509104975
Publikováno v:
Mediators of Inflammation
Mediators of Inflammation, Vol 2, Iss 6, Pp 417-422 (1993)
Mediators of Inflammation, Vol 2, Iss 6, Pp 417-422 (1993)
Increased plasma- and tissue levels of endothelin-1 (ET-1) during inflammatory diseases, have suggested a role of ET-1 in the pathophysiology of inflammatory reactions. The authors have studied the effect of ET-1 on cytokine release from monocytes an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d34c19e9430920bf083c1f244e1f8662
https://doi.org/10.1155/s0962935193000596
https://doi.org/10.1155/s0962935193000596
Publikováno v:
Acta Anaesthesiologica Scandinavica. 36:449-453
Unlike other vascular beds, lung vessels constrict when exposed to hypoxia. However, a marked difference has been noticed as regards the elicitability of hypoxic pulmonary vasoconstriction (HPV) in vivo as compared to in vitro models, like a preparat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ea44ce756c69890958d367c49fc2f66
https://doi.org/10.1111/j.1399-6576.1992.tb03495.x
https://doi.org/10.1111/j.1399-6576.1992.tb03495.x
Publikováno v:
The American journal of physiology. 271(4 Pt 1)
We previously showed that endothelin-1 (ET-1) causes accumulation of leukocytes in the pulmonary microvasculature and increases vascular permeability in isolated rat lungs provided the presence of leukocytes in the perfusate. In the present study, we
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3688abb6e00c09d5fdf83a703478d35
https://pubmed.ncbi.nlm.nih.gov/8897900
https://pubmed.ncbi.nlm.nih.gov/8897900
Publikováno v:
Canadian journal of physiology and pharmacology. 74(4)
The present experiments were carried out in the rat to investigate the peripheral vascular function prior to the development of posthypothermic circulatory collapse. In the first study, mean arterial blood pressure, heart rate, cardiac output, region
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::03a242c93e65a6861ce46dd0303f40fe
https://pubmed.ncbi.nlm.nih.gov/8828884
https://pubmed.ncbi.nlm.nih.gov/8828884
Publikováno v:
Circulatory shock. 44(4)
We previously reported that the endothelin-1 (ET-1)-induced increase in microvascular permeability in isolated rat lungs required leukocytes in the perfusate. The present study examines whether intravenous administration of ET-1 in rats causes an inf
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b8063742e8eaccd4861159d7e2f08f05
https://pubmed.ncbi.nlm.nih.gov/7628062
https://pubmed.ncbi.nlm.nih.gov/7628062
Autor:
John F. Stover, E Helset, Julia Hasslacher, Harm Peters, Reto Stocker, T Henneberg, Stanislao Morgera, Michael Joannidis, H.-H. Neumayer, M Wehner, K Andersen, Torsten Slowinski, Detlef Kindgen-Milles, Justyna Kozik-Jaromin, S Brett
Publikováno v:
Critical Care
Regional citrate anticoagulation in continuous venovenous hemodialysis (citrate-CVVHD) has become a widely used technique in the ICU, which decreases risk of bleeding. However, concern exists about safety of citrate in liver failure patients. The aim
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc80c484974b5cbf080a38b94985ed6a
http://europepmc.org/articles/PMC3061757
http://europepmc.org/articles/PMC3061757