Zobrazeno 1 - 10
of 345
pro vyhledávání: '"E, Baraona"'
Publikováno v:
Journal of Lipid Research, Vol 34, Iss 7, Pp 1237-1244 (1993)
Alcohol consumption markedly increases the hepatic output of very low density lipoprotein (VLDL), whereas it decreases the resulting low density lipoprotein (LDL) levels and apolipoprotein B. As ethylation of apoB-lysine renders LDL immunogenic and a
Externí odkaz:
https://doaj.org/article/1275fb08106147c4b8eb6b156b4cf096
Publikováno v:
Journal of Lipid Research, Vol 27, Iss 10, Pp 1073-1083 (1990)
To study the mechanism of alcoholic hypertriglyceridemia, baboons were pair-fed liquid diets containing 50% of energy as ethanol or as additional carbohydrate for 5-16 months. Alcohol-fed animals developed hypertriglyceridemia and early stages of alc
Externí odkaz:
https://doaj.org/article/d38c588e8550482da931afe5a0d51864
Autor:
E Baraona, C S Lieber
Publikováno v:
Journal of Lipid Research, Vol 20, Iss 3, Pp 289-315 (1979)
Alcohol promotes accumulation of fat in the liver mainly by substitution of ethanol for fatty acids as the major hepatic fuel. The degree of lipid accumulation depends on the supply of dietary fat. Progressive alteration of the mitochondria, which oc
Externí odkaz:
https://doaj.org/article/4a314abde2b14a0eb1e99cac882dab24
Publikováno v:
Journal of Lipid Research, Vol 25, Iss 8, Pp 813-820 (1984)
To study the effects of alcoholic liver injury on the ability of ethanol to promote hepatic fat accumulation and hyperlipemia, baboons were pair-fed liquid diets containing 50% of energy either as ethanol or as additional carbohydrate (controls) for
Externí odkaz:
https://doaj.org/article/d23552b74a4c42f2a474f76f5373106d
Publikováno v:
Alcoholism, clinical and experimental research. 25(5 Suppl)
This article represents the proceedings of a symposium at the 2000 ISBRA Meeting in Yokohama, Japan. The chairs were Nobuhiro Sato and Kai O. Lindros. The presentations were (1) Sex differences in ethanol pharmacokinetics, by E. Baraona; (2) Estrogen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c4cfd501749a2f73b8ec65059baa6865
https://pubmed.ncbi.nlm.nih.gov/11391047
https://pubmed.ncbi.nlm.nih.gov/11391047
Publikováno v:
Alcoholism, clinical and experimental research. 24(7)
A portion of ingested alcohol does not reach the systemic blood, undergoing a first-pass metabolism (FPM) during gastric and hepatic circulation.To determine whether the stomach can metabolize sufficient ethanol to account for the FPM, and to what ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::327abca1855de943e481a6a5be87f280
https://pubmed.ncbi.nlm.nih.gov/10923995
https://pubmed.ncbi.nlm.nih.gov/10923995
Publikováno v:
Journal of lipid research. 40(6)
Alcohol taken in moderation may prevent atherosclerosis, whereas heavy drinking has the opposite effect, in part by promoting oxidation of low density lipoproteins (LDL), a pathogenetic factor in atherogenesis. We assess here: 1 ) whether similar alt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::526b620f2ff23cc6201428324ca67620
https://pubmed.ncbi.nlm.nih.gov/10357829
https://pubmed.ncbi.nlm.nih.gov/10357829
Autor:
E, Baraona, C S, Lieber
Publikováno v:
Recent developments in alcoholism : an official publication of the American Medical Society on Alcoholism, the Research Society on Alcoholism, and the National Council on Alcoholism. 14
Alcoholic fatty liver and hyperlipemia result from the interaction of ethanol and its oxidation products with hepatic lipid metabolism. An early target of ethanol toxicity is mitochondrial fatty acid oxidation. Acetaldehyde and reactive oxygen specie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2aad587b7d6146d73fac08539e783c47
https://pubmed.ncbi.nlm.nih.gov/9751944
https://pubmed.ncbi.nlm.nih.gov/9751944