Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Dustin P. Martin"'
Publikováno v:
NeoBiota, Vol 60, Iss , Pp 97-115 (2020)
Prevention of aquatic invasive species is a fundamental management challenge. With hundreds of millions of people participating in fishing trips each year, understanding angler movements that transmit invasive species can provide critical insight int
Externí odkaz:
https://doaj.org/article/50580a00c7f3405bb86aba9334e855a5
Publikováno v:
PLoS ONE, Vol 13, Iss 1, p e0190745 (2018)
We investigated the spatial and temporal relationship of catch rates and angler party location for two days following a publicly announced put-and-take stocking of rainbow trout (Oncorhynchus mykiss). Catch rates declined with time since stocking and
Externí odkaz:
https://doaj.org/article/9ef892f9582745a592e4839d297fcc24
Autor:
Xigang Jing, Dustin P. Martin, Ru-Jeng Teng, Stephen Naylor, Girija G. Konduri, Neil Hogg, Billy W. Day, Kirkwood A. Pritchard, Aaron Haefke
Publikováno v:
Free Radic Biol Med
Bronchopulmonary dysplasia (BPD) is caused primarily by oxidative stress and inflammation. To induce BPD, neonatal rat pups were raised in hyperoxic (>90% O2) environments from day one (P1) until day ten (P10) and treated with N-acetyl-lysyltyrosylcy
Autor:
Dustin P. Martin, Anna Strzępa, Kirkwood A. Pritchard, Mary G. Sorci-Thomas, Savannah D. Neu, Bonnie N. Dittel
Publikováno v:
Antioxidants
Volume 10
Issue 9
Antioxidants, Vol 10, Iss 1338, p 1338 (2021)
Volume 10
Issue 9
Antioxidants, Vol 10, Iss 1338, p 1338 (2021)
Plaque psoriasis is a common inflammatory condition of the skin characterized by red, flaking lesions. Current therapies for plaque psoriasis target many facets of the autoimmune response, but there is an incomplete understanding of how oxidative dam
Autor:
John G. Krolikowski, Dustin P. Martin, Deron W. Jones, Kirkwood A. Pritchard, Stephen Naylor, Dorothee Weihrauch, Janine A. Struve
Publikováno v:
Diabetes & Vascular Disease Research
Objective: Diabetes mellitus is a significant risk factor for peripheral artery disease. Diabetes mellitus induces chronic states of oxidative stress and vascular inflammation that increase neutrophil activation and release of myeloperoxidase. The go
Autor:
Lili You, Pin Gao, Di Wu, Ujala Rana, Ye Du, Aiping Shi, Bing Han, Zhimin Fan, Qing (Robert) Miao, Chao Zheng, Gang Zhao, Dustin P. Martin
Publikováno v:
Patient preference and adherence
Pin Gao,1 Lili You,2 Di Wu,1 Aiping Shi,1 Qing Miao,3 Ujala Rana,4 Dustin Paul Martin,3 Ye Du,1 Gang Zhao,1 Bing Han,1 Chao Zheng,5 Zhimin Fan1 1Department of Breast Surgery, First Hospital of Jilin University, Changchun, Jilin, China; 2Department of
Autor:
Dilshan S. Harischandra, Vellareddy Anantharam, Naveen Kondru, Dustin P. Martin, Arthi Kanthasamy, Anumantha G. Kanthasamy, Huajun Jin
Publikováno v:
Prion. 8:143-153
Prion diseases are infectious and inevitably fatal neurodegenerative diseases characterized by prion replication, widespread protein aggregation and spongiform degeneration of major brain regions controlling motor function. Oxidative stress has been
Autor:
Huajun Jin, Arthi Kanthasamy, Vellareddy Anantharam, Travis M. Witte, Robert S. Houk, Dustin P. Martin, Anumantha G. Kanthasamy
Publikováno v:
NeuroToxicology. 32:554-562
Protein misfolding and aggregation are considered key features of many neurodegenerative diseases, but biochemical mechanisms underlying protein misfolding and the propagation of protein aggregates are not well understood. Prion disease is a classica
Autor:
Ganesh Konduri, Kirkwood A. Pritchard, Xigang Jing, Ru-Jeng Teng, Dustin P. Martin, Stephen Naylor
Publikováno v:
Free Radical Biology and Medicine. 128:S32
Bronchopulmonary dysplasia (BPD) is the most common chronic pulmonary disease of premature neonates. While ventilation with supplemental O2 is required for development and survival of the premature neonate, it increases the risk of BPD. Both hyperoxi
Autor:
Kirkwood A. Pritchard, Cheryl A. Hillery, Dustin P. Martin, Ye Liang, Stephen Naylor, Guoliang Yu
Publikováno v:
Free Radical Biology and Medicine. 128:S32
Sickle cell disease (SCD) is caused by a single point mutation of the β-globin gene that increases the polymerization of hemoglobin under hypoxia and subsequent sickling of red blood cells (RBCs). Sickled RBCs occlude capillaries, inducing ischemic