A TGFβ-ECM-integrin signaling axis drives structural reconfiguration of the bile duct to promote polycystic liver disease.

Autor: Waddell, Scott H., Yao, Yuelin, Olaizola, Paula, Walker, Alexander, Jarman, Edward J., Gournopanos, Konstantinos, Gradinaru, Andreea, Christodoulou, Ersi, Gautier, Philippe, Boerrigter, Melissa M., Cadamuro, Massimiliano, Fabris, Luca, Drenth, Joost PH, Kendall, Timothy J., Banales, Jesus M., Khamseh, Ava, Mill, Pleasantine, Boulter, Luke

Publikováno v: Science Translational Medicine; 9/13/2023, Vol. 15 Issue 713, p1-12, 12p

THU-115-YI Ursodeoxycholic acid treatment for primary sclerosing cholangitis

Autor: Mol, Bregje, van Munster, Kim, Weersma, Rinse, Inderson, Akin, Boonstra, Kirsten, Drenth, Joost PH, van Meer, Suzanne, de Vries, Elsemieke S., Spanier, B.W. Marcel, Vrij, Ton, Verlaan, Tess, van Duijn, Femke, Ponsioen, Willemijn, Beuers, Ulrich, Dijkgraaf, Marcel, Ponsioen, Cyriel

Publikováno v: In Journal of Hepatology June 2024 80 Supplement 1:S317-S317

Global multi-stakeholder endorsement of the MAFLD definition

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Publikováno v: In The Lancet Gastroenterology & Hepatology May 2022 7(5):388-390

Rationale and design of the RESOLVE trial: lanreotide as a volume reducing treatment for polycystic livers in patients with autosomal dominant polycystic kidney disease

Autor: Gevers Tom JG, Chrispijn Melissa, Wetzels Jack FM, Drenth Joost PH

Publikováno v: BMC Nephrology, Vol 13, Iss 1, p 17 (2012)

Abstract Background A large proportion of patients with autosomal dominant polycystic kidney disease (ADPKD) suffers from polycystic liver disease. Symptoms arise when liver volume increases. The somatostatin analogue lanreotide has proven to reduce

Externí odkaz: https://doaj.org/article/f41609decad248c78afaf3a2399c160e

Intra- and interfamily phenotypic diversity in pain syndromes associated with a gain-of-function variant of NaV1.7

Autor: Estacion Mark, Han Chongyang, Choi Jin-Sung, Hoeijmakers Janneke GJ, Lauria Giuseppe, Drenth Joost PH, Gerrits Monique M, Dib-Hajj Sulayman D, Faber Catharina G, Merkies Ingemar SJ, Waxman Stephen G

Publikováno v: Molecular Pain, Vol 7, Iss 1, p 92 (2011)

Abstract Background Sodium channel NaV1.7 is preferentially expressed within dorsal root ganglia (DRG), trigeminal ganglia and sympathetic ganglion neurons and their fine-diamter axons, where it acts as a threshold channel, amplifying stimuli such as

Externí odkaz: https://doaj.org/article/e177c716b7ab4c698cf34336fb513beb