Zobrazeno 1 - 10
of 38
pro vyhledávání: '"Dragana Odobasic"'
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Externí odkaz:
https://doaj.org/article/fd4267aaff8f4d3897eb2ac65a6cbe3f
Publikováno v:
Frontiers in Physiology, Vol 12 (2021)
Chronic kidney disease (CKD) leads to significant morbidity and mortality worldwide. Glomerulonephritis (GN) is the second leading cause of CKD resulting in end stage renal failure. The most severe and rapidly progressive type of GN is characterized
Externí odkaz:
https://doaj.org/article/1193cecd53134da19b71f2b6fa443ab1
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Anti-myeloperoxidase vasculitis (MPO-AAV) is a life-threatening autoimmune disease which causes severe inflammation of small blood vessels, mainly in the kidney. As for many other autoimmune diseases, current treatments, which consist of general immu
Externí odkaz:
https://doaj.org/article/5ebf5d0ec0fd4f54b4b90da62fb3cddf
Publikováno v:
Journal of Immunology Research, Vol 2016 (2016)
Neutrophils are no longer seen as leukocytes with a sole function of being the essential first responders in the removal of pathogens at sites of infection. Being armed with numerous pro- and anti-inflammatory mediators, these phagocytes can also con
Externí odkaz:
https://doaj.org/article/0f80f42ece81411ea5634235ead5621e
Publikováno v:
Odobasic, D, Hawkins, C L & Radic, M 2022, ' New roles of neutrophils and granulocytic MDSC in autoimmune diseases, inflammation, and anti-microbial immunity ', Frontiers in Immunology, vol. 13, 974062 . https://doi.org/10.3389/fimmu.2022.974062
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f4fd35735a9543dd8d5ab7237237633
https://curis.ku.dk/ws/files/318440878/New_roles_of_neutrophils_and_granulocytic_MDSC_in_autoimmune_diseases_inflammation_and_anti_microbial_immunity.pdf
https://curis.ku.dk/ws/files/318440878/New_roles_of_neutrophils_and_granulocytic_MDSC_in_autoimmune_diseases_inflammation_and_anti_microbial_immunity.pdf
Publikováno v:
Frontiers in Physiology, Vol 12 (2021)
Frontiers in Physiology
Frontiers in Physiology
Chronic kidney disease (CKD) leads to significant morbidity and mortality worldwide. Glomerulonephritis (GN) is the second leading cause of CKD resulting in end stage renal failure. The most severe and rapidly progressive type of GN is characterized
Autor:
Devi Ngo, Stephen R. Holdsworth, Yuan Jia, Wenping Kao, Xuemin Wei, Yuan Hang Yang, Eric F Morand, Ran Gu, Dragana Odobasic, Huapeng Fan, A. Richard Kitching
Publikováno v:
International Immunopharmacology. 61:140-149
The effects of formyl peptide receptors (FPRs) on effector T cells and inflammation-causing tissue-resident cells are not well known. Here, we explored the effect of FPR activation on efferent T cell responses in models of rheumatoid arthritis (RA) a
Publikováno v:
Nephrology Dialysis Transplantation. 34:429-441
Background The functional relevance of OX40 ligand (OX40L) in the effector phase of crescentic glomerulonephritis (GN) is unknown. These studies defined the role of endogenous OX40L during the effector stage of murine crescentic GN. Methods GN was in
Autor:
Stephen R. Holdsworth, Anqi Li, Sharon L. Ford, Maliha A. Alikhan, Clare L. V. Westhorpe, Charles R. Mackay, A. Richard Kitching, Joshua D. Ooi, Sven H. Loosen, Jonathan Dick, Trent M. Woodruff, Dragana Odobasic, Michael J. Hickey, Poh-Yi Gan, Pam Hall
Publikováno v:
Kidney International. 93:615-625
The prospects for complement-targeted therapy in ANCA-associated vasculitis have been enhanced by a recent clinical trial in which C5a receptor 1 (C5aR1) inhibition safely replaced glucocorticoids in induction treatment. C5aR1 primes neutrophils for
Autor:
Dragana Odobasic, Stephen R. Holdsworth, Raymond Shim, Takeshi Fujita, Kim M. O’Sullivan, Jonathan Dick, Joshua D. Ooi, Arthur R Kitching, Poh-Yi Gan, Maliha A. Alikhan
Publikováno v:
The Journal of Immunology. 199:3042-3050
Myeloperoxidase (MPO) anti-neutrophil cytoplasmic Ab (ANCA)–associated vasculitis results from autoimmunity to MPO. IL-17A plays a critical role in generating this form of autoimmune injury but its cell of origin is uncertain. We addressed the hypo