Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Dounia Abbadi"'
AUF1 gene transfer increases exercise performance and improves skeletal muscle deficit in adult mice
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 22, Iss , Pp 222-236 (2021)
Muscle function and mass begin declining in adults long before evidence of sarcopenia and include reduced mitochondrial function, although much remains to be characterized. We found that mRNA decay factor AU-rich mRNA binding factor 1 (AUF1), which s
Externí odkaz:
https://doaj.org/article/87f7bc3a86ac4d28bf86668fa01b2274
Autor:
Mathilde Bizou, Romain Itier, Mina Majdoubi, Dounia Abbadi, Estelle Pichery, Marianne Dutaur, Dimitri Marsal, Denis Calise, Barbara Garmy-Susini, Victorine Douin-Echinard, Jérome Roncalli, Angelo Parini, Nathalie Pizzinat
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-19 (2021)
Abstract The lymphatic network of mammalian heart is an important regulator of interstitial fluid compartment and immune cell trafficking. We observed a remodeling of the cardiac lymphatic vessels and a reduced lymphatic efficiency during heart hyper
Externí odkaz:
https://doaj.org/article/797a301585f043259cce91e10bc4e8b5
AUF1 gene transfer increases exercise performance and improves skeletal muscle deficit in adult mice
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 22, Iss, Pp 222-236 (2021)
Molecular Therapy. Methods & Clinical Development
Molecular Therapy. Methods & Clinical Development
Muscle function and mass begin declining in adults long before evidence of sarcopenia and include reduced mitochondrial function, although much remains to be characterized. We found that mRNA decay factor AU-rich mRNA binding factor 1 (AUF1), which s
Autor:
Estelle Pichery, Mathilde Bizou, Angelo Parini, Mina Majdoubi, Barbara Garmy-Susini, Jerome Roncalli, Romain Itier, Dounia Abbadi, Nathalie Pizzinat, Victorine Douin-Echinard, Dimitri Marsal, Marianne Dutaur, Denis Calise
Publikováno v:
Scientific Reports
Scientific Reports, 2021, 11 (1), ⟨10.1038/s41598-021-95723-y⟩
Scientific Reports, Nature Publishing Group, 2021, 11 (1), ⟨10.1038/s41598-021-95723-y⟩
Scientific Reports, Vol 11, Iss 1, Pp 1-19 (2021)
Scientific Reports, 2021, 11 (1), ⟨10.1038/s41598-021-95723-y⟩
Scientific Reports, Nature Publishing Group, 2021, 11 (1), ⟨10.1038/s41598-021-95723-y⟩
Scientific Reports, Vol 11, Iss 1, Pp 1-19 (2021)
The lymphatic network of mammalian heart is an important regulator of interstitial fluid compartment and immune cell trafficking. We observed a remodeling of the cardiac lymphatic vessels and a reduced lymphatic efficiency during heart hypertrophy an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2c37bf4392c2e6e14a64fdb4fbcffd5
https://hal.science/hal-03435556/document
https://hal.science/hal-03435556/document
Autor:
Anne-Catherine Prats, Dounia Abbadi, Marianne Dutaur, Florence Tortosa, Danièle Daviaud, Jerome Roncalli, Denis Calise, Edith Renaud-Gabardos, Frédérique Gaits-Iacovoni, Christine Delage, Joffrey Pozzo, Mathilde Bizou, Angelo Parini, Nathalie Pizzinat, Victorine Douin-Echinard, Fanny Laroumanie
Publikováno v:
Cardiovascular Research
Cardiovascular Research, Oxford University Press (OUP), 2018, 114 (1), pp.123-137. ⟨10.1093/cvr/cvx221⟩
Cardiovascular Research, Oxford University Press (OUP), 2018, 114 (1), pp.123-137. ⟨10.1093/cvr/cvx221⟩
Aims Tenascin-C (TNC) is an endogenous danger signal molecule strongly associated with inflammatory diseases and with poor outcome in patients with cardiomyopathies. Its function within pathological cardiac tissue during pressure overload remains poo
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 116(23)
AUF1 promotes rapid decay of mRNAs containing 3' untranslated region (3'UTR) AU-rich elements (AREs). AUF1 depletion in mice accelerates muscle loss and causes limb girdle muscular dystrophy. Here, we demonstrate that the selective, targeted degradat
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 6
Backgrounds Tenascin-C (TN-C) is an extracellular matrix glycoprotein slightly presented in adult tissue but transiently expressed upon tissue injury. It is a well-known regulator of multiple cellular functions during embryogenesis, wound healing and