Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Douglas T. Beattie"'
Autor:
Bhavya Papudeshi, J. Matthew Haggerty, Michael Doane, Megan M. Morris, Kevin Walsh, Douglas T. Beattie, Dnyanada Pande, Parisa Zaeri, Genivaldo G. Z. Silva, Fabiano Thompson, Robert A. Edwards, Elizabeth A. Dinsdale
Publikováno v:
BMC Genomics, Vol 18, Iss 1, Pp 1-13 (2017)
Abstract Background Microbiome/host interactions describe characteristics that affect the host's health. Shotgun metagenomics includes sequencing a random subset of the microbiome to analyze its taxonomic and metabolic potential. Reconstruction of DN
Externí odkaz:
https://doaj.org/article/98402d61ac1e45ee93a9ec9e396b2aec
Publikováno v:
Frontiers in Marine Science, Vol 4 (2018)
Kelp forests provide essential habitats for organisms in temperate rocky shores. Loss of kelp forests has occurred over large areas in a number of temperate regions, including in Australia, where the dominant kelp Ecklonia radiata has been lost from
Externí odkaz:
https://doaj.org/article/37fc5eeaba254a2dba7814aed2948f93
Autor:
Dnyanada Pande, Elizabeth A. Dinsdale, Genivaldo G. Z. Silva, Parisa Zaeri, Robert Edwards, Megan M. Morris, J. Matthew Haggerty, Douglas T. Beattie, Fabiano L. Thompson, Kevin Walsh, Michael P. Doane, Bhavya Papudeshi
Publikováno v:
BMC Genomics
BMC Genomics, Vol 18, Iss 1, Pp 1-13 (2017)
BMC Genomics, Vol 18, Iss 1, Pp 1-13 (2017)
Background Microbiome/host interactions describe characteristics that affect the host's health. Shotgun metagenomics includes sequencing a random subset of the microbiome to analyze its taxonomic and metabolic potential. Reconstruction of DNA fragmen
Autor:
Douglas T. Beattie, Shirley Nakhla, Emiel P. C. van der Vorst, Kerry-Anne Rye, Philip J. Barter, Alison K. Heather, Maria L. Castro, Christina A. Bursill
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 30:1773-1778
Objective— To investigate whether high-density lipoproteins (HDLs) suppress chemokine (CCL2, CCL5, and CX 3 CL1) and chemokine receptor (CCR2 and CX 3 CR1) expression, a mechanism for the atheroprotective properties of HDLs. Methods and Results—