Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Dorota Anna Zieba"'
Autor:
Maciej Wójcik, Dorota Anna Zieba, Joanna Bochenek, Agata Krawczyńska, Marcin Barszcz, Alina Gajewska, Hanna Antushevich, Andrzej Przemysław Herman
Publikováno v:
International Journal of Inflammation, Vol 2024 (2024)
The hypothalamic-pituitary-somatotropic (HPS) axis controls many physiological and pathophysiological processes. The phenomenon of insensitivity to growth hormone resistance (GHres) was previously reported to be due to the development of inflammation
Externí odkaz:
https://doaj.org/article/712717610f2a4571a88b3426b1ccf573
Publikováno v:
Frontiers in Neuroscience, Vol 17 (2023)
The short form of the leptin receptor (LeptRa) plays a key role in the transport of leptin to the central nervous system (CNS). Here, MTS-leptin and recombinant ovine (ro) leptin-mediated expression of LeptRa and VEGFA and VEGFR2 concentration in sel
Externí odkaz:
https://doaj.org/article/93a90a319c1f4687b61ae5ff7ae06a82
Autor:
Maciej Wójcik, Agata Krawczyńska, Dorota Anna Zieba, Hanna Antushevich, Andrzej Przemysław Herman
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 9, p 8036 (2023)
Leptin is an adipokine with a pleiotropic impact on many physiological processes, including hypothalamic-pituitary-somatotropic (HPS) axis activity, which plays a key role in regulating mammalian metabolism. Leptin insensitivity/resistance is a patho
Externí odkaz:
https://doaj.org/article/9ceb11f87f9c44ee9533c0581e175912
Autor:
Agata Krawczyńska, Andrzej Przemysław Herman, Hanna Antushevich, Joanna Bochenek, Karolina Wojtulewicz, Dorota Anna Zieba
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 15, p 8109 (2022)
As a day animal with sensitivity to inflammation similar to that of humans, the sheep may highly outperform the rodent model in inflammation studies. Additionally, seasonality makes sheep an interesting model in endocrinology research. Although there
Externí odkaz:
https://doaj.org/article/9baafb4378ca414a96fbbaded8856b29
Publikováno v:
Animals, Vol 12, Iss 3, p 403 (2022)
This study examined how leptin affects growth hormone (GH) release and investigated the effects of leptin, GH, and day length on the suppressor of cytokine signaling-3 (SOCS-3) mRNA levels in the adenohypophyses of sheep. The study consisted of two e
Externí odkaz:
https://doaj.org/article/f6278f9121974399bf0318127482738e
Publikováno v:
Animals, Vol 11, Iss 8, p 2451 (2021)
The short form of the leptin receptor (LRa) plays a key role in the transport of leptin to the central nervous system (CNS). Here, the resistin (RSTN)-mediated expression of LRa in the preoptic area (POA), ventromedial and dorsomedial nuclei (VMH/DMH
Externí odkaz:
https://doaj.org/article/3e63a969ba1544e9ac902c2e4c9a5ced
Autor:
Dorota Anna Zieba, Weronika Biernat, Malgorzata Szczesna, Katarzyna Kirsz, Justyna Barć, Tomasz Misztal
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 12, p 4238 (2020)
Both long-term undernutrition and overnutrition disturb metabolic balance, which is mediated partially by the action of two adipokines, leptin and resistin (RSTN). In this study, we manipulated the diet of ewes to produce either a thin (lean) or fat
Externí odkaz:
https://doaj.org/article/f833299c243b4f0c9702757a9f74e89c
Publikováno v:
Nutrients, Vol 11, Iss 9, p 2180 (2019)
We hypothesized that resistin is engaged in the development of leptin central insensitivity/resistance in sheep, which is a unique animal model to explore reversible leptin resistance. Thirty Polish Longwool ewes, which were ovariectomized with estro
Externí odkaz:
https://doaj.org/article/9f20dcce163b4b60b2ad33176cbdfb47
Publikováno v:
Reproductive biology. 2(1)
Folliculogenesis was studied daily in the 18 oestrous cycles in six prolific Olkuska ewes from October to December using transrectal ultrasonography to record the number and size of all ovarian folliclesor =2 mm in diameter. Blood samples were taken
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1f9c6815bef91931ea4b5b16bdcc0005
https://pubmed.ncbi.nlm.nih.gov/14666161
https://pubmed.ncbi.nlm.nih.gov/14666161