Zobrazeno 1 - 10
of 84
pro vyhledávání: '"Dongchun Liang"'
Autor:
Dongchun Liang, Jeong-Im Woo, Hui Shao, Willi K Born, Rebecca L O'Brien, Henry J Kaplan, Deming Sun
Publikováno v:
PLoS ONE, Vol 13, Iss 5, p e0197189 (2018)
Whether γδ T cells inhibit or enhance the Foxp3 T cell response depends upon their activation status. The critical enhancing effector in the supernatant is adenosine. Activated γδ T cells express adenosine receptors at high levels, which enables
Externí odkaz:
https://doaj.org/article/160b4676efa74df4a6b0b6524420edf6
Publikováno v:
PLoS ONE, Vol 13, Iss 6, p e0199601 (2018)
We previously reported that activated γδ T cells greatly enhance autoimmune responses, particularly the Th17 response. To determine the mechanisms involved, we made a series of comparisons between activated and non-activated γδ T cells. Our resul
Externí odkaz:
https://doaj.org/article/0f138dd9efc742369bb0d95489fbbda9
Publikováno v:
PLoS ONE, Vol 13, Iss 11, p e0207546 (2018)
[This corrects the article DOI: 10.1371/journal.pone.0199601.].
Externí odkaz:
https://doaj.org/article/07fa61fa626a4f4d92ad8f75a4b9179d
Autor:
Dongchun Liang, Aijun Zuo, Ronglan Zhao, Hui Shao, Willi K Born, Rebecca L O'Brien, Henry J Kaplan, Deming Sun
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0150078 (2016)
γδ T cells can either enhance or inhibit an adaptive immune response, but the mechanisms involved are not fully understood. Given that CD73 is the main enzyme responsible for conversion of AMP into the immunosuppressive molecule adenosine, we inves
Externí odkaz:
https://doaj.org/article/840edddbb37849c8996a1fc7007dce9e
Publikováno v:
PLoS ONE, Vol 11, Iss 5, p e0155953 (2016)
Various pathological conditions are accompanied by ATP release from the intracellular to the extracellular compartment. Extracellular ATP (eATP) functions as a signaling molecule by activating purinergic P2 purine receptors. The key P2 receptor invol
Externí odkaz:
https://doaj.org/article/cd7c7f62b24f4508ba9e64c0b429e8db
Autor:
Dongchun Liang, Aijun Zuo, Ronglan Zhao, Hui Shao, Willi K Born, Rebecca L O'Brien, Henry J Kaplan, Deming Sun
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0164502 (2016)
[This corrects the article DOI: 10.1371/journal.pone.0150078.].
Externí odkaz:
https://doaj.org/article/49d6cc901ebd4f6da1d7836fb472b513
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0132348 (2015)
We have recently reported that, although adenosine receptor (AR) agonists have a suppressive effect on Th1 autoreactive T cells, their effect on Th17 autoreactive T cells and γδ T cells is stimulatory and this effect is mainly mediated via A2A aden
Externí odkaz:
https://doaj.org/article/e42234415c9b46fca355ebe17092409d
Publikováno v:
PLoS ONE, Vol 9, Iss 9, p e108932 (2014)
The adenosine A2A receptor (A2AR), the main functional adenosine receptor on murine T cells, plays a unique role in the attenuation of inflammation and tissue damage in vivo. Here, we showed that, of the immune cell types tested, activated γδ T cel
Externí odkaz:
https://doaj.org/article/5ce10823aad54cd9a6269d3dcdf2c29d
Autor:
liwei dong, xiaoning sun, zhichao ma, jiao fu, fujin liu, baili huang, dongchun liang, deming sun, cheng lan
Background: γδT cells play an important role in the mucosa inflammation and immunity-associated disorders. Our previous study reported that γδT cells producing IL-17 were involved in the pathogenesis of post-infectious irritable bowel syndrome (P
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::779bc9408d254d5738f1ea6869257e9a
https://doi.org/10.21203/rs.3.rs-24111/v3
https://doi.org/10.21203/rs.3.rs-24111/v3
Autor:
liwei dong, xiaoning sun, zhichao ma, jiao fu, fujin liu, baili huang, dongchun liang, deming sun, cheng lan
Background: γδT cells play an important role in the mucosa inflammation and immunity-associated disorders. Our previous study reported that γδT cells producing IL-17 were involved in the pathogenesis of post-infectious irritable bowel syndrome (P
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c454e67d93c6488b3d2b3d26ccf1f42
https://doi.org/10.21203/rs.3.rs-24111/v1
https://doi.org/10.21203/rs.3.rs-24111/v1