Výsledky vyhledávání - "Domsgen, Erna"

Coxsackie-adenovirus receptor expression is enhanced in pancreas from patients with type 1 diabetes

Autor: Hodik, M., Anagandula, M., Fuxe, J., Krogvold, L., Dahl JÃ¸rgensen, K., Hyã¶ty, H., Sarmiento, L., Frisk, G, Atkinson, Mark, Pugliese, Alberto, Campbell Thompson, Martha, Kaddis, John, Kusmartseva, Irina, Yang, Mingder, Clare Salzler, Michael, Chapman, Nora, Tracy, Steven, Smithee, Shane, Alhazmi, Abdulaziz, Frisk, Gun, Anagandula, Mahesh, Sarimiento, Luis, Giepmans, Ben, Kalicharan, Ruby, Kuipers, Jeroen, Wolters, Anouk, Jelmer, Willems, Morgan, Noel, Richardson, Sarah, Dyahal, Shalinee, Russell, Mark, Leete, Pia, Nadler, Jerry, Morris, Margaret, Nyalwidhe, Julius, Burch, Tayna, von Herrath, Matthias, Rodriguez Calvo, Teresa, Sabouri, Somayeh, Anquetil Besnard, Florence, Plagnol, Vincent, Breuer, Judith, Depledge, Daniel, Dotta, Francesco, Sebastiani, Guido, Patti, Aurora, Nigi, Laura, Ventriglia, Giuliana, Mancarella, Francesca, Gerling, Ivan, Kakoola, Dorothy, Lenchik, Nataliya, Hyã¶ty, Heikki, Laiho, Jutta, Oikarnen, Maarit, Eizirik, Decio, Opdebeeck, Anne, Lloyd, Richard, Petrosino, Joseph, Homann, Dirk, Kent, Sally, Sarkar, Suparna, Toniolo, Antonio, Gianani, Roberto, Horwitz, Marc, Marjomaki, Varpu, FlodstrÃ¶m Tullberg, Malin, Domsgen, Erna, Larsson, Pã¤r, PIEMONTI, LORENZO

Publikováno v: BMJ Open Diabetes Research & Care

Objectives: One of the theories connecting enterovirus (EV) infection of human islets with type 1 diabetes (T1D) is the development of a fertile field in the islets. This implies induction of appropriate proteins for the viral replication such as the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7dbe42b202fe6a872f94015c99ac468a
https://pubmed.ncbi.nlm.nih.gov/27933184

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Involvement of Pattern Recognition Receptors in Coxsackievirus B Infection of Human Islets

Autor: Domsgen, Erna

T1DM is an autoimmune disease in which beta-cells are selectively destroyed by the immune system. As consequence of beta-cell destruction insulin levels decline resulting in elevated blood glucose levels. The disease is highly associated with genetic

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______139::9144d258909ae9e367fefa7753c35dd9
https://media.suub.uni-bremen.de/handle/elib/461

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