Implication of the SMN complex in the biogenesis and steady state level of the Signal Recognition Particle

Autor: Piazzon, N., Schlotter, F., Lefebvre, S., Dodre, M., Mereau, A., Soret, J., Besse, A., Barkats, M., Bordonne, R., Branlant, C., Massenet, S.

Publikováno v: Nucleic Acids Research
Nucleic Acids Research, Oxford University Press, 2013, 41 (2), pp.1255-1272. ⟨10.1093/nar/gks1224⟩
Nucleic Acids Research, Oxford University Press, 2013, 41 (2), pp.1255-1272. 〈10.1093/nar/gks1224〉
Nucleic Acids Research, 2013, 41 (2), pp.1255-1272. ⟨10.1093/nar/gks1224⟩

International audience; Spinal muscular atrophy is a severe motor neuron disease caused by reduced levels of the ubiquitous Survival of MotoNeurons (SMN) protein. SMN is part of a complex that is essential for spliceosomal UsnRNP biogenesis. Signal r

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::d096426cc9cd65de4d73c840e83c6ef8
https://hal.archives-ouvertes.fr/hal-02995998