Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Doan T. Le"'
Autor:
Janine Slevinsky, Zoe Liberman, Julia W. Van de Vondervoort, Doan T. Le, J. Kiley Hamlin, Conor M. Steckler
Publikováno v:
British Journal of Developmental Psychology. 36:482-500
Recent research has shown that infants selectively approach prosocial versus antisocial characters, suggesting that foundations of sociomoral development may be present early in life. Despite this, to date, the mental processes involved in infants' p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::74f399a9747ee0b098f72d0de0f9b565
https://doi.org/10.1111/bjdp.12232
https://doi.org/10.1111/bjdp.12232
Publikováno v:
Cognition. 168
Whereas adults largely base their evaluations of others’ actions on others’ intentions, a host of research in developmental psychology suggests that younger children privilege outcome over intention, leading them to condemn accidental harm. To da
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b75e2e975e43f9f72a5b6a88650dddff
https://pubmed.ncbi.nlm.nih.gov/28688284
https://pubmed.ncbi.nlm.nih.gov/28688284
Publikováno v:
Cognition. 124:201-208
How is ownership established over non-owned things? We suggest that people may view ownership as a kind of credit given to agents responsible for making possession of a non-owned object possible. On this view, judgments about the establishment of own
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a74fe8e0ebb81efcbceaeac03fed2d89
https://doi.org/10.1016/j.cognition.2012.04.006
https://doi.org/10.1016/j.cognition.2012.04.006
Autor:
Stephen M. Wiesner, Miechaleen D. Diers, Andrew Kim, Kevin Shannon, Jennifer O. Lauchle, Kelly Morgan, Doan T. Le, Diane E. Hasz, Scott C. Kogan, Jennifer L. Geurts, Luis F. Parada, David A. Largaespada
Publikováno v:
Blood. 109:1687-1691
Neurofibromatosis type 1 (NF1) syndrome is caused by germline mutations in the NF1 tumor suppressor, which encodes neurofibromin, a GTPase activating protein for Ras. Children with NF1 are predisposed to juvenile myelomonocytic leukemia (JMML) and le
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9e1e6e090ea5271af1e8a631bcd48a6
https://doi.org/10.1182/blood-2006-05-025395
https://doi.org/10.1182/blood-2006-05-025395
Autor:
Benjamin S. Braun, Namie Kong, Luis F. Parada, Jennifer O. Lauchle, Kevin Shannon, Abigail Aiyigari, Scott C. Kogan, Michelle M. Le Beau, Endi Wang, Yuan Zhu, Doan T. Le
Publikováno v:
Blood. 103:4243-4250
The NF1 tumor suppressor gene encodes a guanosine triphosphotase (GTPase)-activating protein that negatively regulates Ras signaling and is inactivated in a subset of juvenile myelomonocytic leukemias (JMMLs). Adoptive transfer of fetal liver cells f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0848b966ebeaf721e403c940d12cce57
https://doi.org/10.1182/blood-2003-08-2650
https://doi.org/10.1182/blood-2003-08-2650
Autor:
Namie Kong, David A. Tuveson, Tyler Jacks, Michelle M. Le Beau, Scott C. Kogan, Benjamin S. Braun, Doan T. Le, Kevin Shannon, Jacob Rozmus
Publikováno v:
Proceedings of the National Academy of Sciences. 101:597-602
RAS mutations are common in myeloid malignancies; however, it is not known whether oncogenic RAS can initiate leukemia. We show that expressing mutant K-Ras G12D protein from the endogenous murine locus rapidly induces a fatal myeloproliferative diso
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::16d32efcd11c4974aff48fea4d0ba48b
https://doi.org/10.1073/pnas.0307203101
https://doi.org/10.1073/pnas.0307203101
Autor:
Kegan Warner, Ernesto Diaz-Flores, Jeannette M. Bonifas, Mary Tran, Doan T. Le, Matthew F. Gorman, Neal G. Copeland, Luis F. Parada, Linda Wolff, Keiko Akagi, Kevin Shannon, Michael Crone, Sally Przybranowski, Judith Sebolt-Leopold, Jeroen P. Roose, Jennifer O. Lauchle, Scott C. Kogan, Doris Kim, Nancy A. Jenkins, Qing Li, Kimberly Krisman, Kristen M Coakley
Publikováno v:
Nature
In a mouse model for myeloproliferative disorder (MPD) driven by loss of NF-1 (which activates the Ras–MEK–MAPK pathway), additional mutations were created by insertional mutagenesis that led to progression of the disease to acute myeloid leukaem
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99c806d2b04a09313b92fded70112160
https://pubmed.ncbi.nlm.nih.gov/19727076
https://pubmed.ncbi.nlm.nih.gov/19727076
Autor:
Keiko Akagi, Jennifer O. Lauchle, Kevin Shannon, Judith Sebolt-Leopold, Mary Tran, Neal G. Copeland, Luis F. Parada, Matthew F. Gorman, Linda Wolff, Doris Kim, Nancy A. Jenkins, Doan T. Le
Publikováno v:
Blood. 110:594-594
Hyperactive Ras is a common feature of many cancers, including myelodysplastic syndrome (MDS), myeloproliferative disease (MPD), and acute myeloid leukemia (AML). Conditional inactivation of the Nf1 tumor suppressor, which encodes a GTPase activating
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ae7b6a0eb5a773262c1f10268819280d
https://doi.org/10.1182/blood.v110.11.594.594
https://doi.org/10.1182/blood.v110.11.594.594
Publikováno v:
Journal of Drug Targeting; 1998, Vol. 5 Issue 4, p275-289, 15p
Publikováno v:
Journal of Controlled Release; 2000, Vol. 67 Issue: 2 p337-346, 10p