Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Dina A. Schneider"'
Autor:
Sarah A. Woller, Soo-Ho Choi, Eun Jung An, Hann Low, Dina A. Schneider, Roshni Ramachandran, Jungsu Kim, Yun Soo Bae, Dmitri Sviridov, Maripat Corr, Tony L. Yaksh, Yury I. Miller
Publikováno v:
Cell Reports, Vol 23, Iss 9, Pp 2667-2677 (2018)
Apolipoprotein A-I binding protein (AIBP) reduces lipid raft abundance by augmenting the removal of excess cholesterol from the plasma membrane. Here, we report that AIBP prevents and reverses processes associated with neuroinflammatory-mediated spin
Externí odkaz:
https://doaj.org/article/752f1381fb2546bd8b718014a285dc72
Autor:
Dina A. Schneider, Soo-Ho Choi, Colin Agatisa-Boyle, Laurence Zhu, Jungsu Kim, Jennifer Pattison, Dorothy D. Sears, PhilipL.S.M. Gordts, Longhou Fang, Yury I. Miller
Publikováno v:
Journal of Lipid Research, Vol 59, Iss 5, Pp 854-863 (2018)
Apolipoprotein A-I binding protein (AIBP) has been shown to augment cholesterol efflux from endothelial cells and macrophages. In zebrafish and mice, AIBP-mediated regulation of cholesterol levels in the plasma membrane of endothelial cells controls
Externí odkaz:
https://doaj.org/article/4b90aadd8f674110b8eb0228f6b2d70f
Autor:
Chao Liu, Keith P. Gates, Longhou Fang, Marcelo J. Amar, Dina A. Schneider, Honglian Geng, Wei Huang, Jungsu Kim, Jennifer Pattison, Jian Zhang, Joseph L. Witztum, Alan T. Remaley, P. Duc Dong, Yury I. Miller
Publikováno v:
Disease Models & Mechanisms, Vol 8, Iss 8, Pp 989-998 (2015)
Apolipoprotein C-II (APOC2) is an obligatory activator of lipoprotein lipase. Human patients with APOC2 deficiency display severe hypertriglyceridemia while consuming a normal diet, often manifesting xanthomas, lipemia retinalis and pancreatitis. Hyp
Externí odkaz:
https://doaj.org/article/0e8f7229942e490c806f467aa4dab7cb
Autor:
Shurong Huang, Jennifer M. Rutkowsky, Ryan G. Snodgrass, Kikumi D. Ono-Moore, Dina A. Schneider, John W. Newman, Sean H. Adams, Daniel H. Hwang
Publikováno v:
Journal of Lipid Research, Vol 53, Iss 9, Pp 2002-2013 (2012)
Toll-like receptor 4 (TLR4) and TLR2 were shown to be activated by saturated fatty acids (SFAs) but inhibited by docosahexaenoic acid (DHA). However, one report suggested that SFA-induced TLR activation in cell culture systems is due to contaminants
Externí odkaz:
https://doaj.org/article/c8134a3dc6134548b026ee338ffc615d
Autor:
Eun Jung An, Yury I. Miller, Tony L. Yaksh, Jungsu Kim, Hann Low, Maripat Corr, Roshni Ramachandran, Sarah A. Woller, Yun Soo Bae, Dina A. Schneider, Dmitri Sviridov, Soo-Ho Choi
Publikováno v:
Cell Reports, Vol 23, Iss 9, Pp 2667-2677 (2018)
Cell reports, vol 23, iss 9
Cell reports, vol 23, iss 9
Apolipoprotein A-I binding protein (AIBP) reduces lipid raft abundance by augmenting the removal of excess cholesterol from the plasma membrane. Here, we report that AIBP prevents and reverses processes associated with neuroinflammatory-mediated spin
Autor:
Peter J. Havel, Dina A. Schneider, James L. Graham, Ryan G. Snodgrass, Jeong-a Kim, Ling Zhao, Michael J. Quon, Shurong Huang, Daniel H. Hwang, Kikumi D. Ono-Moore, Jennifer M. Rutkowsky
Publikováno v:
Immunity, Inflammation and Disease
Immunity, inflammation and disease, vol 5, iss 4
Ono-Moore, Kikumi D; Zhao, Ling; Huang, Shurong; Kim, Jeonga; Rutkowsky, Jennifer M; Snodgrass, Ryan G; et al.(2017). Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity.. Immunity, inflammation and disease, 5(4), 526-540. doi: 10.1002/iid3.162. UC Davis: Retrieved from: http://www.escholarship.org/uc/item/8qr484zm
Immunity, inflammation and disease, vol 5, iss 4
Ono-Moore, Kikumi D; Zhao, Ling; Huang, Shurong; Kim, Jeonga; Rutkowsky, Jennifer M; Snodgrass, Ryan G; et al.(2017). Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity.. Immunity, inflammation and disease, 5(4), 526-540. doi: 10.1002/iid3.162. UC Davis: Retrieved from: http://www.escholarship.org/uc/item/8qr484zm
Author(s): Ono-Moore, Kikumi D; Zhao, Ling; Huang, Shurong; Kim, Jeonga; Rutkowsky, Jennifer M; Snodgrass, Ryan G; Schneider, Dina A; Quon, Michael J; Graham, James L; Havel, Peter J; Hwang, Daniel H | Abstract: IntroductionChronic low-grade inflamma
Autor:
Jungsu Kim, Longhou Fang, Aaron M. Wallace, Benjamin T. Suratt, Elena Alekseeva, Elianne Burg, Yury I. Miller, Carlyne D. Cool, Dina A. Schneider, Soo-Ho Choi, Niki D.J. Ubags
Publikováno v:
JCI insight, vol 3, iss 16
Acute respiratory distress syndrome (ARDS) is characterized by an excessive pulmonary inflammatory response. Removal of excess cholesterol from the plasma membrane of inflammatory cells helps reduce their activation. The secreted apolipoprotein A-I b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a3517e89ceb6e069db8b25d54f06ccb0
https://escholarship.org/uc/item/40g7m6fg
https://escholarship.org/uc/item/40g7m6fg
Autor:
Colin Agatisa-Boyle, Soo-Ho Choi, Philip L.S.M. Gordts, Longhou Fang, Jungsu Kim, Dorothy D. Sears, Jennifer Pattison, Yury I. Miller, Laurence Zhu, Dina A. Schneider
Publikováno v:
Journal of lipid research, vol 59, iss 5
Journal of Lipid Research, Vol 59, Iss 5, Pp 854-863 (2018)
Journal of Lipid Research, Vol 59, Iss 5, Pp 854-863 (2018)
Apolipoprotein A-I binding protein (AIBP) has been shown to augment cholesterol efflux from endothelial cells and macrophages. In zebrafish and mice, AIBP-mediated regulation of cholesterol levels in the plasma membrane of endothelial cells controls
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28a3751532fcccd0d02bc30f8224473c
https://escholarship.org/uc/item/2x2048c8
https://escholarship.org/uc/item/2x2048c8
Autor:
Kikumi D. Ono-Moore, Jennifer M. Rutkowsky, Colin S. McCoin, Shurong Huang, Shamsher Singh, Daniel H. Hwang, Dina A. Schneider, Trina A. Knotts, Sean H. Adams
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 306:E1378-E1387
Incomplete β-oxidation of fatty acids in mitochondria is a feature of insulin resistance and type 2 diabetes mellitus (T2DM). Previous studies revealed that plasma concentrations of medium- and long-chain acylcarnitines (by-products of incomplete β
Publikováno v:
Cancer Immunology Research. 7:B029-B029
Recent advances in genetic engineering have resulted in exponential growth in cell therapy technologies. As an increasing number of CAR-T therapies are entering clinical trials, there is a scarcity of resources put into optimizing production strategi