Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Dilara Batan"'
Autor:
Megan E. Schroeder, Dilara Batan, Andrea Gonzalez Rodriguez, Kelly F. Speckl, Douglas K. Peters, Bruce E. Kirkpatrick, Grace K. Hach, Cierra J. Walker, Joseph C. Grim, Brian A. Aguado, Robert M. Weiss, Kristi S. Anseth
Publikováno v:
Bioengineering & Translational Medicine, Vol 8, Iss 1, Pp n/a-n/a (2023)
Abstract Patients with aortic valve stenosis (AVS) have sexually dimorphic phenotypes in their valve tissue, where male valvular tissue adopts a calcified phenotype and female tissue becomes more fibrotic. The molecular mechanisms that regulate sex
Externí odkaz:
https://doaj.org/article/4f693d1bbced4f58a85c14afab8db778
Autor:
Cierra J. Walker, Dilara Batan, Carrie T. Bishop, Daniel Ramirez, Brian A. Aguado, Megan E. Schroeder, Claudia Crocini, Jessica Schwisow, Karen Moulton, Laura Macdougall, Robert M. Weiss, Mary A. Allen, Robin Dowell, Leslie A. Leinwand, Kristi S. Anseth
Publikováno v:
Bioengineering & Translational Medicine, Vol 7, Iss 3, Pp n/a-n/a (2022)
Abstract Aortic valve stenosis (AVS) is a progressive fibrotic disease that is caused by thickening and stiffening of valve leaflets. At the cellular level, quiescent valve interstitial cells (qVICs) activate to myofibroblasts (aVICs) that persist wi
Externí odkaz:
https://doaj.org/article/7131e16927834fb5a636e98358625fa7
Autor:
Brian A. Aguado, Cierra J. Walker, Joseph C. Grim, Megan E. Schroeder, Dilara Batan, Brandon J. Vogt, Andrea Gonzalez Rodriguez, Jessica A. Schwisow, Karen S. Moulton, Robert M. Weiss, Donald D. Heistad, Leslie A. Leinwand, Kristi S. Anseth
Publikováno v:
Circulation
Background: Aortic valve stenosis is a sexually dimorphic disease, with women often presenting with sustained fibrosis and men with more extensive calcification. However, the intracellular molecular mechanisms that drive these clinically important se
Autor:
Megan E. Schroeder, Dilara Batan, Andrea Gonzalez Rodriguez, Kelly F. Speckl, Douglas K. Peters, Bruce E. Kirkpatrick, Grace K. Hach, Cierra J. Walker, Joseph C. Grim, Brian A. Aguado, Robert M. Weiss, Kristi S. Anseth
Publikováno v:
Bioengineering & Translational Medicine. 8
Autor:
null Megan E. Schroeder, null Dilara Batan, null Andrea Gonzalez Rodriguez, null Kelly F. Speckl, null Douglas K. Peters, null Bruce E. Kirkpatrick, null Grace K. Hach, null Cierra J. Walker, null Joseph C. Grim, null Brian A. Aguado, null Robert M. Weiss, null Kristi S. Anseth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8328e69e0c5fabc4af47011f829d3f5b
https://doi.org/10.1002/btm2.10358/v3/response1
https://doi.org/10.1002/btm2.10358/v3/response1
Autor:
Dilara Batan, Douglas K. Peters, Megan E. Schroeder, Brian A. Aguado, Mark W. Young, Robert M. Weiss, Kristi S. Anseth
Publikováno v:
FASEB J
As aortic valve stenosis develops, valve tissue becomes stiffer. In response to this change in environmental mechanical stiffness, valvular interstitial cells (VICs) activate into myofibroblasts. We aimed to investigate the role of mechanosensitive c
Autor:
Cierra J. Walker, Dilara Batan, Carrie T. Bishop, Daniel Ramirez, Brian A. Aguado, Megan E. Schroeder, Claudia Crocini, Jessica Schwisow, Karen Moulton, Laura Macdougall, Robert M. Weiss, Mary A. Allen, Robin Dowell, Leslie A. Leinwand, Kristi S. Anseth
Publikováno v:
Bioengineeringtranslational medicine. 7(3)
Aortic valve stenosis (AVS) is a progressive fibrotic disease that is caused by thickening and stiffening of valve leaflets. At the cellular level, quiescent valve interstitial cells (qVICs) activate to myofibroblasts (aVICs) that persist within the
Autor:
Megan E. Schroeder, Brian A. Aguado, Robert M. Weiss, Andrea Gonzalez-Rodriguez, F. Max Yavitt, Cassidy L. Andrichik, Dilara Batan, Joseph C. Grim, Brandon J. Vogt, Kristi S. Anseth
Publikováno v:
Arterioscler Thromb Vasc Biol
Objective: Resident valvular interstitial cells (VICs) activate to myofibroblasts during aortic valve stenosis progression, which further promotes fibrosis or even differentiate into osteoblast-like cells that can lead to calcification of valve tissu
Autor:
Hao Ma, Dilara Batan, Andrea Gonzalez-Rodriguez, Robert M. Weiss, Laura J. Macdougall, Megan E. Schroeder, Kristi S. Anseth
Publikováno v:
Macromol Biosci
The role viscoelasticity plays in fibrotic disease progression is an emerging area of interest. Here, a fast-relaxing hydrogel system is exploited to investigate potential crosstalk between calcium signaling and mechanotransduction. Poly(ethylene gly
A Multicolor Split-Fluorescent Protein Approach to Visualize Listeria Protein Secretion in Infection
Autor:
Michael Minson, Esther Braselmann, Dieu My T. Nguyen, Amy E. Palmer, Dilara Batan, Pascale Cossart
Publikováno v:
Biophysical Journal
Biophysical Journal, 2018, 115 (2), pp.251-262. ⟨10.1016/j.bpj.2018.03.016⟩
Biophysical Journal, Biophysical Society, 2018, 115 (2), pp.251-262. ⟨10.1016/j.bpj.2018.03.016⟩
Biophysical Journal, 2018, 115 (2), pp.251-262. ⟨10.1016/j.bpj.2018.03.016⟩
Biophysical Journal, Biophysical Society, 2018, 115 (2), pp.251-262. ⟨10.1016/j.bpj.2018.03.016⟩
International audience; Listeria monocytogenes is an intracellular food-borne pathogen that has evolved to enter mammalian host cells, survive within them, spread from cell to cell, and disseminate throughout the body. A series of secreted virulence
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::293c975532d9200dfdf8f3c8e8158d8c
https://hal-pasteur.archives-ouvertes.fr/pasteur-01857232
https://hal-pasteur.archives-ouvertes.fr/pasteur-01857232