Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Difei Shen"'
Autor:
Yizhou Feng, Yuan Yuan, Hongxia Xia, Zhaopeng Wang, Yan Che, Zhefu Hu, Jiangyang Deng, Fangfang Li, Qingqing Wu, Zhouyan Bian, Heng Zhou, Difei Shen, Qizhu Tang
Publikováno v:
Journal of Translational Medicine, Vol 21, Iss 1, Pp 1-20 (2023)
Abstract Background Oncostatin M (OSM) is a secreted cytokine of the interleukin (IL)-6 family that induces biological effects by activating functional receptor complexes of the common signal transducing component glycoprotein 130 (gp130) and OSM rec
Externí odkaz:
https://doaj.org/article/694e59a5294c4ff5ad06033fc099e10b
Autor:
Zhouyan Bian, Haihan Liao, Yan Zhang, Qingqing Wu, Heng Zhou, Zheng Yang, Jinrong Fu, Teng Wang, Ling Yan, Difei Shen, Hongliang Li, Qizhu Tang
Publikováno v:
PLoS ONE, Vol 9, Iss 4, p e96095 (2014)
Cardiac hypertrophy appears to be a specialized form of cellular growth that involves the proliferation control and cell cycle regulation. NIMA (never in mitosis, gene A)-related kinase-6 (Nek6) is a cell cycle regulatory gene that could induce centr
Externí odkaz:
https://doaj.org/article/f5ece70f80674cd5869d363d0b42c186
Autor:
Wei Deng, Xinan Wang, Jinfeng Xiao, Kuoju Chen, Heng Zhou, Difei Shen, Hongliang Li, Qizhu Tang
Publikováno v:
PLoS ONE, Vol 7, Iss 1, p e30256 (2012)
BACKGROUND: The effect of regulator of G protein signaling 5 (RGS5) on cardiac hypertrophy, atherosclerosis and angiogenesis has been well demonstrated, but the role in the development of obesity and insulin resistance remains completely unknown. We
Externí odkaz:
https://doaj.org/article/d9bce6911938454685182c0326a92da5
Publikováno v:
Cellular signalling. 86
Cardiac hypertrophy is the uppermost risk factor for the development of heart failure, leading to irreversible cardiac structural remodeling and sudden death. As a major mediator of cardiac remodeling, oncostatin M (OSM) and its receptor, OSMR, attra
Autor:
Heng Zhou, Ning Li, Chen Liu, Wei Wang, Difei Shen, Wei Deng, Mingxia Duan, Qing-Qing Wu, Hai-Ming Wu, Qi-Zhu Tang
Publikováno v:
Free radical biologymedicine. 160
Ferroptosis is a reactive oxygen species (ROS)- and iron-dependent form of regulated cell death (RCD), playing critical roles in organ injury and targeting therapy of cancers. Previous studies have demonstrated that ferroptosis participates in the de
Autor:
Jun, Bu, Mao, Chen, Xiaoshu, Cheng, Yifei, Dong, Weiyi, Fang, Junbo, Ge, Yanjun, Gong, Ben, He, Lan, Huang, Yong, Huo, Shaobin, Jia, Jun, Jiang, Yue, Li, Zhao, Li, Chun, Liang, Xuebo, Liu, Zhenyu, Liu, Xiang, Ma, Yitong, Ma, Juying, Qian, Chengxing, Shen, Difei, Shen, Li, Shen, Ruizheng, Shi, Xi, Su, Yingxian, Sun, Yida, Tang, Jianan, Wang, Yue, Wu, Dingcheng, Xiang, Tongda, Xu, Yawei, Xu, Yuejin, Yang, Hesong, Zeng, Cheng, Zhang, Guogang, Zhang, Ruiyan, Zhang, Shuning, Zhang, Yun, Zhang, Zheng, Zhang, Bo, Zheng, Ning, Zhou
Publikováno v:
Nan fang yi ke da xue xue bao = Journal of Southern Medical University. 40(2)
The SARS-CoV-2 epidemic starting in Wuhan in December, 2019 has spread rapidly throughout the nation. The control measures to contain the epidemic also produced influences on the transport and treatment process of patients with acute myocardial infar
Publikováno v:
Journal of Cellular Biochemistry. 118:1911-1920
Cardiac fibrosis is characterized by over-deposition of extracellular matrix (ECM) proteins and over-proliferation of cardiac fibroblast, and contributes to both systolic and diastolic dysfunction in many cardiac pathophysiologic conditions. Transfor
Publikováno v:
Molecular Medicine Reports
Myocardial remodeling serves an important role in the pathophysiology of coronary heart disease. The angiotensin-converting enzyme (ACE)2-angiotensin-(1-7) [Ang (1‑7)]‑Mas receptor (MasR) axis is a key regulator in myocardial remodeling and devel
Autor:
Zhao-Peng Wang, Yuan Yuan, Heng Zhou, Yan-Yan Meng, Difei Shen, Ya-Ge Jin, Yan Che, Sha-Sha Wang, Qing-Qing Wu
Publikováno v:
Bioscience Reports
Aim: In this work, we explored the role of corosolic acid (CRA) during pressure overload-induced cardiac hypertrophy. Methods and results: Cardiac hypertrophy was induced in mice by aortic banding. Four weeks post-surgery, CRA-treated mice developed
Autor:
Qi-Zhu Tang, Yuan Yuan, Heng Zhou, Ya-Ge Jin, Ning Li, Wei Deng, Hongliang Li, Difei Shen, Qing-Qing Wu, Ling Yan, Zhou-Yan Bian
Publikováno v:
Journal of molecular medicine (Berlin, Germany). 98(5)
Cardiac hypertrophy is a complex pathological process, and the molecular mechanisms underlying hypertrophic remodeling have not been clearly elucidated. Leukocyte immunoglobulin-like receptor B4 (lilrb4) is an inhibitory transmembrane protein that is