Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Didem Dayangac-Erden"'
Publikováno v:
Cellular & Molecular Biology Letters, Vol 23, Iss 1, Pp 1-12 (2018)
Abstract Background Perineuronal nets (PNNs), which are localized around neurons during development, are specialized forms of neural extracellular matrix with neuroprotective and plasticity-regulating roles. Hyaluronan and proteoglycan link protein 1
Externí odkaz:
https://doaj.org/article/5ada53fd48e64b43809362cbdd4a484f
Autor:
Didem Dayangac-Erden, Nagehan Emiralioglu, Nural Kiper, İlksen Berfin Ekinci, Ebru Yalcin, Ugur Ozcelik, Deniz Dogru, Mina Gharibzadeh Hizal
Publikováno v:
Pediatric Pulmonology. 56:910-920
Cystic fibrosis (CF) is an autosomal recessive disease caused by CFTR gene mutations. Despite having the same mutation, CF patients may demonstrate clinical variability in severity and prognosis of the disease. In this study, we aimed to determine di
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc0b84071bc599543980fec47969d133
https://doi.org/10.1002/ppul.25237
https://doi.org/10.1002/ppul.25237
Autor:
Haluk Topaloglu, Ayse Yuzbasioglu, Evrim Aksu-Menges, Burcu Balci-Hayta, Didem Dayangac-Erden, Banu Balci-Peynircioglu, Beril Talim, Yeliz Z. Akkaya-Ulum
Publikováno v:
The American Journal of Pathology. 190:2136-2145
Secondary mitochondrial damage in skeletal muscles is a common feature of different neuromuscular disorders, which fall outside the mitochondrial cytopathies. The common cause of mitochondrial dysfunction and structural changes in skeletal muscle tis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c88947cff52d1e33cd79024e4a7df91e
https://doi.org/10.1016/j.ajpath.2020.06.011
https://doi.org/10.1016/j.ajpath.2020.06.011
Autor:
Merve Atalay, Engin Yilmaz, Nural Kiper, Ebru Yalcin, Ugur Ozcelik, Nagehan Emiralioglu, Sanem Eryilmaz Polat, Didem Dayangac-Erden, Mina Gharibzadeh Hizal, Deniz Dogru
Publikováno v:
Clinica chimica acta; international journal of clinical chemistry. 510
Background Cystic fibrosis, the most prevalent autosomal recessive genetic disease, is caused by mutations in the CFTR gene. The spectrum and frequency of CFTR mutations in Turkish patients show heterogeneity. Methods We investigated CFTR gene mutati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4f750f5888c0275676f936bcda9621c
https://pubmed.ncbi.nlm.nih.gov/32687833
https://pubmed.ncbi.nlm.nih.gov/32687833
Autor:
Burcu Balci-Hayta, Can Ebru Bekircan-Kurt, Didem Dayangac-Erden, Ersin Tan, Sevim Erdem-Ozdamar, Evrim Aksu
Publikováno v:
Journal of the Neurological Sciences. 393:100-104
Background Primary myoblast cell cultures display the phenotypic characteristics and genetic defects of the donor tissue and represent an in vitro model system reflecting the disease pathology. They have been generated only from freshly harvested tis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b66576da7e390f1a9a6d2a33468cf3e8
https://doi.org/10.1016/j.jns.2018.08.018
https://doi.org/10.1016/j.jns.2018.08.018
Publikováno v:
Pediatrics International. 59:53-56
BACKGROUND In single gene disorders, patients with the same genotype may have variations in severity. One of the main factors affecting disease severity is modifier genes. Spinal muscular atrophy (SMA) is an autosomal recessive neuromuscular disorder
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56fe01128a7ccfd0fa085e58c7d8a9f7
https://doi.org/10.1111/ped.13052
https://doi.org/10.1111/ped.13052
Publikováno v:
Cellular & Molecular Biology Letters, Vol 23, Iss 1, Pp 1-12 (2018)
Cellular & Molecular Biology Letters
Cellular & Molecular Biology Letters
Background Perineuronal nets (PNNs), which are localized around neurons during development, are specialized forms of neural extracellular matrix with neuroprotective and plasticity-regulating roles. Hyaluronan and proteoglycan link protein 1 (HAPLN1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd8a59fbf05f0687e27c0ed7597dd8ed
https://doi.org/10.1186/s11658-018-0073-5
https://doi.org/10.1186/s11658-018-0073-5
Sepsis, one of the priority diseases in health research, is a severe disease condition characterized by a systemic inflammatory response to infections, and its incidence still keeps increasing worldwide. To date, no reliable markers for prognosis, ea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::73cb6ca5410ce5b69b59500d766157cb
https://doi.org/10.1016/b978-0-12-805364-5.00013-5
https://doi.org/10.1016/b978-0-12-805364-5.00013-5
Autor:
Yasemin Oztemur-Islakoglu, Bala Gur-Dedeoglu, Didem Dayangac-Erden, Sevim Erdem-Ozdamar, Fatma Nazli Eskici
Spinal muscular atrophy (SMA) is one of the most common childhood onset neurodegenerative disorders in global health whereby novel biomarkers and therapeutic targets are sorely needed. SMA is an autosomal recessive genetic disorder resulting in degen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b8b0f19ed18d0ec3ec758505bb913b9
https://aperta.ulakbim.gov.tr/record/34181
https://aperta.ulakbim.gov.tr/record/34181
Autor:
Mandy Ahlborg, Anna Bakenecker, Chiara M. Ciniselli, Maria G. Daidone, Didem Dayangac-Erden, Christina Debbeler, Hans-Peter Deigner, Mine Durusu-Tanriover, Manuela Gariboldi, Jenny Groten, Dorothee Haas, Felicitas S. Holzer, Lisa J. Jacob, Afroz Jahan, Christian Kaethner, Burcu Kesikli, Agnes Kisser, Matthias Kohl, Mara Lecchi, Jan Lüddecke, Kerstin Lüdtke-Buzug, Yasmina A. Mansur, Ignacio D. Mastroleo, Roland Mertelsmann, Meral Ozguç, James R. Perkins, Navneet Phogat, Juan A.G. Ranea, Elena Rojano, Melanie Scharr, Sowmya Srinivasan Perumbakkam, Imran Uddin, Anusha Venkatraman, Paolo Verderio, Stephan Vilgis, Marcel Wegmann, Ayse Yuzbasioglu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::796470eca93b579b1bfa08deb1fd477e
https://doi.org/10.1016/b978-0-12-805364-5.09989-3
https://doi.org/10.1016/b978-0-12-805364-5.09989-3