PERK inhibition attenuates the abnormalities of the secretory pathway and the increased apoptotic rate induced by SIL1 knockdown in HeLa cells

Autor: Capone, Vanessa, Clemente, Emanuela, Restelli, Elena, Di Campli, Antonella, Sperduti, Samantha, Ornaghi, Francesca, Pietrangelo, Laura, Protasi, Feliciano, Chiesa, Roberto, Sallese, Michele

Publikováno v: In BBA - Molecular Basis of Disease October 2018 1864(10):3164-3180

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Autophagosome–lysosome fusion triggers a lysosomal response mediated by TLR9 and controlled by OCRL

Autor: De Leo, Maria Giovanna, Staiano, Leopoldo, Vicinanza, Mariella, Luciani, Alessandro, Carissimo, Annamaria, Mutarelli, Margherita, Di Campli, Antonella, Polishchuk, Elena, Di Tullio, Giuseppe, Morra, Valentina, Levtchenko, Elena, Oltrabella, Francesca, Starborg, Tobias, Santoro, Michele, Devuyst, Olivier, Lowe, Martin, Medina, Diego L., BALLABIO, ANDREA, DE MATTEIS, Maria Antonietta, MEDINA SANABRIA, Diego Luis, DI BERNARDO, DIEGO

Publikováno v: Nature cell biology 18 (2016). doi:10.1038/ncb3386
info:cnr-pdr/source/autori:De Leo, Maria Giovanna; Staiano, Leopoldo; Vicinanza, Mariella; Luciani, Alessandro; Carissimo, Annamaria; Mutarelli, Margherita; Di Campli, Antonella; Polishchuk, Elena; Di Tullio, Giuseppe; Morra, Valentina; Levtchenko, Elena; Oltrabella, Francesca; Starborg, Tobias; Santoro, Michele; Di Bernardo, Diego; Devuyst, Olivier; Lowe, Martin; Medina, Diego L.; Ballabio, Andrea; De Matteis, Maria Antonietta/titolo:Autophagosome-lysosome fusion triggers a lysosomal response mediated by TLR9 and controlled by OCRL/doi:10.1038%2Fncb3386/rivista:Nature cell biology/anno:2016/pagina_da:/pagina_a:/intervallo_pagine:/volume:18
De Leo, M G, Staiano, L, Vicinanza, M, Luciani, A, Carissimo, A, Mutarelli, M, Di Campli, A, Polishchuk, E, Di Tullio, G, Morra, V, Levtchenko, E, Oltrabella, F, Starborg, T, Santoro, M, di Bernardo, D, Devuyst, O, Lowe, M, Medina, D L, Ballabio, A & De Matteis, M A 2016, ' Autophagosome-lysosome fusion triggers a lysosomal response mediated by TLR9 and controlled by OCRL ', Nature Cell Biology, vol. 18, no. 8, pp. 839-50 . https://doi.org/10.1038/ncb3386
Nature Cell Biology
Nat Cell Biol
Nature cell biology 18 (2016): 839–850. doi:10.1038/ncb3386
info:cnr-pdr/source/autori:De Leo M.G.; Staiano L.; Vicinanza M.; Luciani A.; Carissimo A.; Mutarelli M.; Di Campli A.; Polishchuk E.; Di Tullio G.; Morra V.; Levtchenko E.; Oltrabella F.; Starborg T.; Santoro M.; Di Bernardo D.; Devuyst O.; Lowe M.; Medina D.L.; Ballabio A.; De Matteis M.A./titolo:Autophagosome-lysosome fusion triggers a lysosomal response mediated by TLR9 and controlled by OCRL/doi:10.1038%2Fncb3386/rivista:Nature cell biology/anno:2016/pagina_da:839/pagina_a:850/intervallo_pagine:839–850/volume:18

Phosphoinositides (PtdIns) control fundamental cell processes, and inherited defects of PtdIns kinases or phosphatases cause severe human diseases, including Lowe syndrome due to mutations in OCRL, which encodes a PtdIns(4,5)P2 5-phosphatase. Here we

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2827715c6eec0b6f41a0bf1415d2ecf1
https://doi.org/10.5167/uzh-133228

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